Focused cerebellar laser light induced hyperthermia improves symptoms and pathology of polyglutamine disease SCA1 in a mouse model (Q35188379)

7 August 2017

title

Focused cerebellar laser light induced hyperthermia improves symptoms and pathology of polyglutamine disease SCA1 in a mouse model (English)

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7 August 2017

Scoty M Hearst

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7 August 2017

Qingmei Shao

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7 August 2017

Mariper Lopez

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7 August 2017

Drazen Raucher

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7 August 2017

Parminder J S Vig

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7 August 2017

publication date

1 October 2014

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7 August 2017

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7 August 2017

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7 August 2017

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7 August 2017

page(s)

596-606

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The pathogenesis of spinocerebellar ataxia

7 August 2017

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The design and delivery of a thermally responsive peptide to inhibit S100B-mediated neurodegeneration

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Suppression of calbindin-D28k expression exacerbates SCA1 phenotype in a disease mouse model.

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Bergmann glial S100B activates myo-inositol monophosphatase 1 and Co-localizes to purkinje cell vacuoles in SCA1 transgenic mice

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12 December 2020

Purkinje cell expression of a mutant allele of SCA1 in transgenic mice leads to disparate effects on motor behaviors, followed by a progressive cerebellar dysfunction and histological alterations.

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12 December 2020

Polyglutamine protein aggregates are dynamic

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12 December 2020

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Relationship between ataxin-1 nuclear inclusions and Purkinje cell specific proteins in SCA-1 transgenic mice

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12 December 2020

Analysis of the role of heat shock protein (Hsp) molecular chaperones in polyglutamine disease

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12 December 2020

10.1007/S12311-014-0576-1

Identifiers

DOI

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7 August 2017

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