Attenuation of the norepinephrine transporter activity and trafficking via interactions with alpha-synuclein (Q24323344)

European Journal of Neuroscience

GOA release 2020-03-11

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Christophe Wersinger

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Alexis Jeannotte

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Anita Sidhu

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language of work or name

English

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publication date

December 2006

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published in

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volume

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issue

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page(s)

3141-52

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Functional interaction between monoamine plasma membrane transporters and the synaptic PDZ domain-containing protein PICK1

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Regulated internalization and phosphorylation of the native norepinephrine transporter in response to phorbol esters. Evidence for localization in lipid rafts and lipid raft-mediated internalization

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Impaired dopamine storage resulting from alpha-synuclein mutations may contribute to the pathogenesis of Parkinson's disease

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Modulation of dopamine transporter function by alpha-synuclein is altered by impairment of cell adhesion and by induction of oxidative stress

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Modulation of the trafficking of the human serotonin transporter by human alpha-synuclein

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Lipid rafts mediate the synaptic localization of alpha-synuclein.

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Role of phosphatidylinositol 3-kinase in angiotensin II regulation of norepinephrine neuromodulation in brain neurons of the spontaneously hypertensive rat.

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Cocaine and antidepressant-sensitive biogenic amine transporters exist in regulated complexes with protein phosphatase 2A.

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Dopamine uptake through the norepinephrine transporter in brain regions with low levels of the dopamine transporter: evidence from knock-out mouse lines

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The multiple LIM domain-containing adaptor protein Hic-5 synaptically colocalizes and interacts with the dopamine transporter

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Synaptic vesicle depletion correlates with attenuated synaptic responses to prolonged repetitive stimulation in mice lacking alpha-synuclein

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A regulated interaction of syntaxin 1A with the antidepressant-sensitive norepinephrine transporter establishes catecholamine clearance capacity

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Cocaine abusers have an overexpression of alpha-synuclein in dopamine neurons.

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Treatment of attention-deficit/hyperactivity disorder: overview of the evidence

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Dopamine reuptake by norepinephrine neurons: exception or rule?

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