Pages that link to "Q35876959"

The following pages link to Aβ delays fibrin clot lysis by altering fibrin structure and attenuating plasminogen binding to fibrin. (Q35876959):

Displaying 46 items.

• Proteins behaving badly. Substoichiometric molecular control and amplification of the initiation and nature of amyloid fibril formation: lessons from and for blood clotting (Q26739684) (← links)
• Breaking boundaries-coagulation and fibrinolysis at the neurovascular interface (Q26781646) (← links)
• The role of iron-induced fibrin in the pathogenesis of Alzheimer's disease and the protective role of magnesium (Q26859190) (← links)
• Role of fibrinogen in cerebrovascular dysfunction after traumatic brain injury (Q26995130) (← links)
• The Fibrin Matrix Regulates Angiogenic Responses within the Hemostatic Microenvironment through Biochemical Control (Q28547610) (← links)
• A novel Aβ-fibrinogen interaction inhibitor rescues altered thrombosis and cognitive decline in Alzheimer's disease mice (Q30579707) (← links)
• Genetic association of urokinase-type plasminogen activator gene rs2227564 site polymorphism with sporadic Alzheimer's disease in the Han Chinese population (Q34720291) (← links)
• An Enzyme from Aristolochia indica Destabilizes Fibrin-β Amyloid Co-Aggregate: Implication in Cerebrovascular Diseases (Q35834455) (← links)
• The α-helix to β-sheet transition in stretched and compressed hydrated fibrin clots (Q36207917) (← links)
• Tumour imaging by the detection of fibrin clots in tumour stroma using an anti-fibrin Fab fragment. (Q36724890) (← links)
• The Alzheimer's disease peptide β-amyloid promotes thrombin generation through activation of coagulation factor XII. (Q36909451) (← links)
• The APOE ɛ4/ɛ4 genotype potentiates vascular fibrin(ogen) deposition in amyloid-laden vessels in the brains of Alzheimer's disease patients (Q37073413) (← links)
• Hyperhomocysteinemia exacerbates Alzheimer's disease pathology by way of the β-amyloid fibrinogen interaction (Q37104293) (← links)
• Neprilysin Inhibits Coagulation through Proteolytic Inactivation of Fibrinogen (Q37112216) (← links)
• Fibrinogen and altered hemostasis in Alzheimer's disease (Q38032520) (← links)
• Adjusting the compass: new insights into the role of angiogenesis in Alzheimer's disease (Q38172335) (← links)
• Diagnostic morphology: biophysical indicators for iron-driven inflammatory diseases (Q38203153) (← links)
• Localization of Fibrinogen in the Vasculo-Astrocyte Interface after Cortical Contusion Injury in Mice (Q38690287) (← links)
• A possible new role for Aβ in vascular and inflammatory dysfunction in Alzheimer's disease. (Q38840372) (← links)
• Role of gut microbiota and nutrients in amyloid formation and pathogenesis of Alzheimer disease. (Q38955990) (← links)
• Interactions of β-amyloid peptide with fibrinogen and coagulation factor XII may contribute to Alzheimer's disease (Q39404969) (← links)
• Biochemical and structural analysis of the interaction between β-amyloid and fibrinogen. (Q40953485) (← links)
• Plasma fibrinogen is a natural deterrent to amyloid beta-induced platelet activation and neuronal toxicity (Q40974661) (← links)
• Fibrin deposited in the Alzheimer's disease brain promotes neuronal degeneration (Q41916686) (← links)
• Alzheimer disease is in part a thrombohemorrhagic disorder (Q47886413) (← links)
• Aminopyrimidine Class Aggregation Inhibitor Effectively Blocks Aβ-Fibrinogen Interaction and Aβ-Induced Contact System Activation. (Q49374917) (← links)
• Blood will out: vascular contributions to Alzheimer's disease. (Q49498166) (← links)
• The role of fibrinolysis inhibition in engineered vascular networks derived from endothelial cells and adipose-derived stem cells (Q49733672) (← links)
• Fibrinogen in neurological diseases: mechanisms, imaging and therapeutics. (Q52606339) (← links)
• Both lipopolysaccharide and lipoteichoic acids potently induce anomalous fibrin amyloid formation: assessment with novel Amytracker™ stains. (Q52694985) (← links)
• No effects without causes: the Iron Dysregulation and Dormant Microbes hypothesis for chronic, inflammatory diseases. (Q53698735) (← links)
• To What Extent Are the Terminal Stages of Sepsis, Septic Shock, Systemic Inflammatory Response Syndrome, and Multiple Organ Dysfunction Syndrome Actually Driven by a Prion/Amyloid Form of Fibrin? (Q53723228) (← links)
• Coagulation factor XIIIa crosslinks amyloid b into dimers and oligomers and to blood proteins (Q58582473) (← links)
• Iron Dysregulation and Dormant Microbes as Causative Agents for Impaired Blood Rheology and Pathological Clotting in Alzheimer's Type Dementia (Q59798274) (← links)
• Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation (Q62640193) (← links)
• Abnormal clotting of the intrinsic/contact pathway in Alzheimer disease patients is related to cognitive ability (Q64045770) (← links)
• Fibrinogen αC domain: Its importance in physiopathology (Q64228855) (← links)
• Epitope Mapping Immunoassay Analysis of the Interaction between β-Amyloid and Fibrinogen (Q64276058) (← links)
• Vascular Dysfunction in Alzheimer's Disease: A Prelude to the Pathological Process or a Consequence of It? (Q64986754) (← links)
• Hemostasis biomarkers and incident cognitive impairment: the REGARDS study (Q88589021) (← links)
• Long-Term Dabigatran Treatment Delays Alzheimer's Disease Pathogenesis in the TgCRND8 Mouse Model (Q90636107) (← links)
• Analysis of β-Amyloid-induced Abnormalities on Fibrin Clot Structure by Spectroscopy and Scanning Electron Microscopy (Q90736438) (← links)
• Differentiation of Induced Pluripotent Stem Cells towards Mesenchymal Stromal Cells is Hampered by Culture in 3D Hydrogels (Q91012144) (← links)
• A Possible Role of Amyloidogenic Blood Clotting in the Evolving Haemodynamics of Female Migraine-With-Aura: Results From a Pilot Study (Q92071188) (← links)
• Fibrin(ogen) in human disease: both friend and foe (Q92710708) (← links)
• Deficiency of tPA Exacerbates White Matter Damage, Neuroinflammation, Glymphatic Dysfunction and Cognitive Dysfunction in Aging Mice (Q92801314) (← links)