Pages that link to "Q34181813"

The following pages link to Overexpression of the ped/pea-15 gene causes diabetes by impairing glucose-stimulated insulin secretion in addition to insulin action (Q34181813):

Displaying 33 items.

• On the Quest of Cellular Functions of PEA-15 and the Therapeutic Opportunities (Q26799216) (← links)
• Negative regulators of insulin signaling revealed in a genome-wide functional screen (Q28476047) (← links)
• Tyrosine phosphorylation of phosphoinositide-dependent kinase 1 by the insulin receptor is necessary for insulin metabolic signaling (Q28564173) (← links)
• Phosphoprotein enriched in astrocytes (PEA)-15: a potential therapeutic target in multiple disease states (Q34024181) (← links)
• Age-related impairment in insulin release: the essential role of β(2)-adrenergic receptor (Q34252924) (← links)
• PED/PEA-15 inhibits hydrogen peroxide-induced apoptosis in Ins-1E pancreatic beta-cells via PLD-1 (Q34663501) (← links)
• Adenoviral gene transfer of PLD1-D4 enhances insulin sensitivity in mice by disrupting phospholipase D1 interaction with PED/PEA-15. (Q34667784) (← links)
• Characterization of a MAPK scaffolding protein logic gate in gonadotropes. (Q35001275) (← links)
• Phosphoprotein enriched in astrocytes-15 kDa expression inhibits astrocyte migration by a protein kinase C delta-dependent mechanism (Q35190978) (← links)
• Clozapine impairs insulin action by up-regulating Akt phosphorylation and Ped/Pea-15 protein abundance (Q35837406) (← links)
• PED/PEA-15 induces autophagy and mediates TGF-beta1 effect on muscle cell differentiation (Q36028460) (← links)
• Peroxisome proliferator-activated receptor-γ activation enhances insulin-stimulated glucose disposal by reducing ped/pea-15 gene expression in skeletal muscle cells: evidence for involvement of activator protein-1 (Q36466778) (← links)
• Prep1 deficiency induces protection from diabetes and increased insulin sensitivity through a p160-mediated mechanism. (Q36898758) (← links)
• The roles of MAPKs in disease (Q37112520) (← links)
• Understanding type 2 diabetes: from genetics to epigenetics (Q38405367) (← links)
• Targetting PED/PEA-15 for diabetes treatment. (Q39232367) (← links)
• Comparison between fibroblast wound healing and cell random migration assays in vitro (Q39541782) (← links)
• Prep1 Controls Insulin Glucoregulatory Function in Liver by Transcriptional Targeting of SHP1 Tyrosine Phosphatase (Q39652158) (← links)
• The PEA-15/PED protein protects glioblastoma cells from glucose deprivation-induced apoptosis via the ERK/MAP kinase pathway. (Q40093567) (← links)
• Raised expression of the antiapoptotic protein ped/pea-15 increases susceptibility to chemically induced skin tumor development. (Q40393314) (← links)
• PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms (Q42272320) (← links)
• Parkinson-like phenotype in insulin-resistant PED/PEA-15 transgenic mice (Q42416060) (← links)
• A peptide antagonist of Prep1-p160 interaction improves ceramide-induced insulin resistance in skeletal muscle cells. (Q42700870) (← links)
• NMR backbone dynamics studies of human PED/PEA-15 outline protein functional sites (Q42913727) (← links)
• Hepatocyte nuclear factor (HNF)-4alpha-driven epigenetic silencing of the human PED gene. (Q43097133) (← links)
• Molecular cloning and characterization of the human PED/PEA-15 gene promoter reveal antagonistic regulation by hepatocyte nuclear factor 4alpha and chicken ovalbumin upstream promoter transcription factor II. (Q43154745) (← links)
• Overproduction of phosphoprotein enriched in diabetes (PED) induces mesangial expansion and upregulates protein kinase C-beta activity and TGF-beta1 expression. (Q43269412) (← links)
• Biomechanical insult switches PEA-15 activity to uncouple its anti-apoptotic function and promote erk mediated tissue remodeling (Q43565613) (← links)
• Targeting of PED/PEA-15 molecular interaction with phospholipase D1 enhances insulin sensitivity in skeletal muscle cells. (Q46549512) (← links)
• Expression of phosphoprotein enriched in astrocytes 15 kDa (PEA-15) in astrocytic tumors: a novel approach of correlating malignancy grade and prognosis (Q48206002) (← links)
• The PEA15 gene is overexpressed and related to insulin resistance in healthy first-degree relatives of patients with type 2 diabetes. (Q51488380) (← links)
• Phorbol esters induce intracellular accumulation of the anti-apoptotic protein PED/PEA-15 by preventing ubiquitinylation and proteasomal degradation. (Q53337896) (← links)
• PREP1 deficiency downregulates hepatic lipogenesis and attenuates steatohepatitis in mice. (Q54408005) (← links)