Pages that link to "Q24621785"
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The following pages link to The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells (Q24621785):
Displaying 46 items.
- Ripped to death (Q24632980) (← links)
- FADD and caspase-8 control the outcome of autophagic signaling in proliferating T cells (Q24646372) (← links)
- Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways (Q27010127) (← links)
- Necroptosis: molecular signalling and translational implications (Q27015896) (← links)
- Role of Fas-associated death domain-containing protein (FADD) phosphorylation in regulating glucose homeostasis: from proteomic discovery to physiological validation (Q28293997) (← links)
- Mechanisms of necroptosis in T cells (Q28592588) (← links)
- Toll-like receptor signaling in cell proliferation and survival (Q33598805) (← links)
- Expression of serine 194-phosphorylated Fas-associated death domain protein correlates with proliferation in B-cell non-Hodgkin lymphomas (Q33873985) (← links)
- Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages (Q33946622) (← links)
- The many roles of FAS receptor signaling in the immune system (Q34205697) (← links)
- The complex interplay between autophagy, apoptosis, and necrotic signals promotes T-cell homeostasis (Q34252212) (← links)
- Fas-associated death domain (FADD) and the E3 ubiquitin-protein ligase TRIM21 interact to negatively regulate virus-induced interferon production (Q34675854) (← links)
- Functional complementation between FADD and RIP1 in embryos and lymphocytes (Q34768810) (← links)
- Inhibition of Fas-associated death domain-containing protein (FADD) protects against myocardial ischemia/reperfusion injury in a heart failure mouse model (Q34994373) (← links)
- Intertwined pathways of programmed cell death in immunity (Q35108580) (← links)
- The Fas/CD95 Receptor Regulates the Death of Autoreactive B Cells and the Selection of Antigen-Specific B Cells (Q36114138) (← links)
- Daxx plays a novel role in T cell survival but is dispensable in Fas-induced apoptosis (Q36310860) (← links)
- Sensing of viral infection and activation of innate immunity by toll-like receptor 3 (Q36422519) (← links)
- Beyond tumor necrosis factor receptor: TRADD signaling in toll-like receptors. (Q36843563) (← links)
- Antigen-mediated T cell expansion regulated by parallel pathways of death (Q36970979) (← links)
- FADD is a key regulator of lipid metabolism (Q37141375) (← links)
- A role for cFLIP in B cell proliferation and stress MAPK regulation (Q37287191) (← links)
- Toll-like receptors and their role in carcinogenesis and anti-tumor treatment. (Q37354939) (← links)
- Death receptor signal transducers: nodes of coordination in immune signaling networks (Q37417486) (← links)
- Critical function of the necroptosis adaptor RIPK3 in protecting from intestinal tumorigenesis (Q37564748) (← links)
- It cuts both ways: reconciling the dual roles of caspase 8 in cell death and survival (Q37947994) (← links)
- TNF and ubiquitin at the crossroads of gene activation, cell death, inflammation, and cancer (Q37948115) (← links)
- RIP1-mediated regulation of lymphocyte survival and death responses (Q37950926) (← links)
- Programmed necrosis: backup to and competitor with apoptosis in the immune system (Q37956879) (← links)
- Fas-associated death domain protein and adenosine partnership: fad in RA. (Q37976645) (← links)
- Functions of caspase 8: the identified and the mysterious (Q38214149) (← links)
- Cell Death Signaling (Q38655649) (← links)
- Modulation of innate immune-related pathways in nicotine-treated SH-SY5Y cells (Q39424731) (← links)
- Calmodulin mediates Fas-induced FADD-independent survival signaling in pancreatic cancer cells via activation of Src-extracellular signal-regulated kinase (ERK). (Q39536531) (← links)
- Pax2 regulates a fadd-dependent molecular switch that drives tissue fusion during eye development (Q39649368) (← links)
- A novel function of RIP1 in postnatal development and immune homeostasis by protecting against RIP3-dependent necroptosis and FADD-mediated apoptosis (Q41230653) (← links)
- Effects of celecoxib on cell apoptosis and Fas, FasL and Bcl-2 expression in a BGC-823 human gastric cancer cell line (Q41672403) (← links)
- The death domain of FADD is essential for embryogenesis, lymphocyte development, and proliferation (Q42067854) (← links)
- FADD deficiency impairs early hematopoiesis in the bone marrow (Q42224191) (← links)
- FADD negatively regulates lipopolysaccharide signaling by impairing interleukin-1 receptor-associated kinase 1-MyD88 interaction (Q42910629) (← links)
- Constitutive phosphorylation mutation in Fas-associated death domain (FADD) results in early cell cycle defects (Q43552428) (← links)
- Kinome analysis of Toll-like receptor signaling in bovine monocytes (Q45722415) (← links)
- Structural and functional modeling of viral protein 5 of Infectious Bursal Disease Virus (Q50087143) (← links)
- Soluble TNF-like weak inducer of apoptosis (TWEAK) enhances poly(I:C)-induced RIPK1-mediated necroptosis (Q57801740) (← links)
- Redundant and receptor-specific activities of TRADD, RIPK1 and FADD in death receptor signaling (Q61796844) (← links)
- FADD and the NF-kappaB family member Bcl-3 regulate complementary pathways to control T-cell survival and proliferation (Q62817691) (← links)