Calmodulin mediates Fas-induced FADD-independent survival signaling in pancreatic cancer cells via activation of Src-extracellular signal-regulated kinase (ERK). (Q39536531)

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scientific article published on 25 May 2011

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English	Calmodulin mediates Fas-induced FADD-independent survival signaling in pancreatic cancer cells via activation of Src-extracellular signal-regulated kinase (ERK).	scientific article published on 25 May 2011

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scholarly article

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Europe PubMed Central

PMC publication ID

12 September 2017

Calmodulin mediates Fas-induced FADD-independent survival signaling in pancreatic cancer cells via activation of Src-extracellular signal-regulated kinase (ERK). (English)

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Europe PubMed Central

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12 September 2017

pancreatic cancer

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Yabing Chen

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12 September 2017

author name string

Kaiyu Yuan

1

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12 September 2017

Gu Jing

2

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12 September 2017

Jianfeng Chen

3

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12 September 2017

Hui Liu

4

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12 September 2017

Kui Zhang

5

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12 September 2017

Yuebin Li

6

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12 September 2017

Hui Wu

7

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12 September 2017

Jay M McDonald

8

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12 September 2017

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publication date

25 May 2011

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12 September 2017

Journal of Biological Chemistry

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12 September 2017

286

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12 September 2017

28

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12 September 2017

24776-24784

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Europe PubMed Central

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12 September 2017

CD95 promotes tumour growth

1 reference

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Src kinases as therapeutic targets for cancer

1 reference

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The role of Src in solid tumors

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CD95, BIM and T cell homeostasis

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Sorafenib and vorinostat kill colon cancer cells by CD95-dependent and -independent mechanisms

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How ERK1/2 activation controls cell proliferation and cell death: Is subcellular localization the answer?

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The many roles of FAS receptor signaling in the immune system

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Calcium signaling

1 reference

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Pancreatic cancer.

1 reference

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The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells

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Src kinase phosphorylates Caspase-8 on Tyr380: a novel mechanism of apoptosis suppression

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Expression of kinase-defective mutants of c-Src in human metastatic colon cancer cells decreases Bcl-xL and increases oxaliplatin- and Fas-induced apoptosis

1 reference

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N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-kappaB signaling pathway

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Regulation of cell cycle progression by calcium/calmodulin-dependent pathways

1 reference

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13 September 2017

Calmodulin binding to the Fas death domain. Regulation by Fas activation

1 reference

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CD95's deadly mission in the immune system

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Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations

1 reference

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The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

1 reference

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Caspase activation is required for T cell proliferation

1 reference

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Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity

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Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

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A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

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Sequence motifs for calmodulin recognition

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Apoptosis by death factor

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Calmodulin binding to cellular FLICE-like inhibitory protein modulates Fas-induced signalling

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Calmodulin binding to the Fas-mediated death-inducing signaling complex in cholangiocarcinoma cells.

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10.1074/JBC.M110.202804

1 reference

Europe PubMed Central

PMC publication ID

12 September 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 September 2017

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