Pages that link to "Q24672509"
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The following pages link to Transmission of cell stress from endoplasmic reticulum to mitochondria: enhanced expression of Lon protease (Q24672509):
Displaying 50 items.
- Downregulation of the human Lon protease impairs mitochondrial structure and function and causes cell death (Q24293262) (← links)
- Two proteases, trypsin domain-containing 1 (Tysnd1) and peroxisomal lon protease (PsLon), cooperatively regulate fatty acid β-oxidation in peroxisomal matrix (Q24293436) (← links)
- HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells (Q24302194) (← links)
- Thermodynamic characterization of specific interactions between the human Lon protease and G-quartet DNA (Q24305446) (← links)
- Islet amyloid in type 2 diabetes, and the toxic oligomer hypothesis (Q24655066) (← links)
- Cell death and survival through the endoplasmic reticulum-mitochondrial axis (Q26823387) (← links)
- Multitasking in the mitochondrion by the ATP-dependent Lon protease (Q27003208) (← links)
- Evolution and significance of the Lon gene family in Arabidopsis organelle biogenesis and energy metabolism (Q27003925) (← links)
- Structural properties of substrate proteins determine their proteolysis by the mitochondrial AAA+ protease Pim1. (Q27936117) (← links)
- Mitochondrial Lon protease in human disease and aging: Including an etiologic classification of Lon-related diseases and disorders (Q28071866) (← links)
- Inflammation and cellular stress: a mechanistic link between immune-mediated and metabolically driven pathologies (Q28236897) (← links)
- Mitochondrial proteases and cancer (Q28302068) (← links)
- Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling (Q28394742) (← links)
- Evidence that two ATP-dependent (Lon) proteases in Borrelia burgdorferi serve different functions (Q28472063) (← links)
- The ATP-dependent Lon protease of Mus musculus is a DNA-binding protein that is functionally conserved between yeast and mammals (Q28507579) (← links)
- Keap1-Independent Regulation of Nrf2 Activity by Protein Acetylation and a BET Bromodomain Protein (Q28552496) (← links)
- Lon peptidase 1, mitochondrial (Q28556336) (← links)
- Role of Herp in the endoplasmic reticulum stress response (Q28566759) (← links)
- Differential transport and local translation of cytoskeletal, injury-response, and neurodegeneration protein mRNAs in axons (Q28581651) (← links)
- Tetramethylpyrazine blocks TFAM degradation and up-regulates mitochondrial DNA copy number by interacting with TFAM. (Q33698136) (← links)
- TRPC1 inhibits apoptotic cell degeneration induced by dopaminergic neurotoxin MPTP/MPP(+). (Q33797760) (← links)
- Protein modulation in mouse heart under acute and chronic hypoxia. (Q34030929) (← links)
- Mitochondrial Protein Quality Control Systems in Aging and Disease (Q34141153) (← links)
- 4-O-carboxymethyl ascochlorin causes ER stress and induced autophagy in human hepatocellular carcinoma cells (Q34204002) (← links)
- Mitochondrial Lon protease regulates mitochondrial DNA copy number and transcription by selective degradation of mitochondrial transcription factor A (TFAM) (Q34276613) (← links)
- Biological roles of the Podospora anserina mitochondrial Lon protease and the importance of its N-domain (Q34302373) (← links)
- Phosphorylation of human TFAM in mitochondria impairs DNA binding and promotes degradation by the AAA+ Lon protease (Q34314791) (← links)
- Upregulation of the mitochondrial Lon Protease allows adaptation to acute oxidative stress but dysregulation is associated with chronic stress, disease, and aging. (Q34370238) (← links)
- Mitochondrial Lon protease at the crossroads of oxidative stress, ageing and cancer. (Q34493828) (← links)
- CODAS syndrome is associated with mutations of LONP1, encoding mitochondrial AAA+ Lon protease. (Q34902747) (← links)
- Inhibition of Lon blocks cell proliferation, enhances chemosensitivity by promoting apoptosis and decreases cellular bioenergetics of bladder cancer: potential roles of Lon as a prognostic marker and therapeutic target in baldder cancer (Q34957287) (← links)
- Down-regulating overexpressed human Lon in cervical cancer suppresses cell proliferation and bioenergetics (Q35049430) (← links)
- Active-site-directed chemical tools for profiling mitochondrial Lon protease (Q35172759) (← links)
- Endoplasmic reticulum: a primary target in various acute disorders and degenerative diseases of the brain (Q35196245) (← links)
- Mitochondrial (dys)function - a factor underlying the variability of efavirenz-induced hepatotoxicity? (Q35223924) (← links)
- The mitochondrial ATP-dependent Lon protease: a novel target in lymphoma death mediated by the synthetic triterpenoid CDDO and its derivatives (Q35876952) (← links)
- Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation (Q35888498) (← links)
- Matrix proteases in mitochondrial DNA function (Q36059010) (← links)
- Cellular functions of endoplasmic reticulum chaperones calreticulin, calnexin, and ERp57. (Q36242107) (← links)
- Mitochondrial Lon regulates apoptosis through the association with Hsp60-mtHsp70 complex. (Q36347232) (← links)
- Integration of cellular bioenergetics with mitochondrial quality control and autophagy (Q36675381) (← links)
- Functional mechanics of the ATP-dependent Lon protease- lessons from endogenous protein and synthetic peptide substrates (Q36743161) (← links)
- Overexpression of Lon contributes to survival and aggressive phenotype of cancer cells through mitochondrial complex I-mediated generation of reactive oxygen species. (Q36985078) (← links)
- ATP-dependent proteases differ substantially in their ability to unfold globular proteins (Q37254059) (← links)
- The mechanisms of brain ischemic insult and potential protective interventions (Q37485957) (← links)
- The peroxisomal Lon protease LonP2 in aging and disease: functions and comparisons with mitochondrial Lon protease LonP1 (Q37507163) (← links)
- Mitochondrial Lon is over-expressed in high-grade gliomas, and mediates hypoxic adaptation: potential role of Lon as a therapeutic target in glioma. (Q37684592) (← links)
- Mitochondrial stress: a bridge between mitochondrial dysfunction and metabolic diseases? (Q37880356) (← links)
- The multifaceted role of Lon proteolysis in seedling establishment and maintenance of plant organelle function: living from protein destruction. (Q37949008) (← links)
- Mitochondria at the interface between danger signaling and metabolism: role of unfolded protein responses in chronic inflammation (Q37970025) (← links)