Pages that link to "Q58376306"
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The following pages link to Unique Preservation of Neural Cells in Hutchinson- Gilford Progeria Syndrome Is Due to the Expression of the Neural-Specific miR-9 MicroRNA (Q58376306):
Displaying 50 items.
- Targeting protein prenylation in progeria (Q24594564) (← links)
- Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cells (Q26740026) (← links)
- Neuronal dark matter: the emerging role of microRNAs in neurodegeneration (Q26859224) (← links)
- miR-9: the sentinel of neurons in progeria (Q27024586) (← links)
- Potential therapeutic effects of the MTOR inhibitors for preventing ageing and progeria-related disorders (Q28067037) (← links)
- Recent advances in animal and human pluripotent stem cell modeling of cardiac laminopathy (Q28077171) (← links)
- Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior (Q30619979) (← links)
- Drug screening on Hutchinson Gilford progeria pluripotent stem cells reveals aminopyrimidines as new modulators of farnesylation. (Q30846580) (← links)
- Metformin decreases progerin expression and alleviates pathological defects of Hutchinson-Gilford progeria syndrome cells. (Q33914338) (← links)
- Nuclear lamins and neurobiology (Q34056472) (← links)
- Accelerating neuronal aging in in vitro model brain disorders: a focus on reactive oxygen species (Q34406924) (← links)
- Strand-specific RNA-seq reveals widespread occurrence of novel cis-natural antisense transcripts in rice (Q34519975) (← links)
- Glomerular input patterns in the mouse olfactory bulb evoked by retronasal odor stimuli (Q34659037) (← links)
- Progerin reduces LAP2α-telomere association in Hutchinson-Gilford progeria. (Q36048936) (← links)
- Reprogramming progeria fibroblasts re-establishes a normal epigenetic landscape. (Q36396535) (← links)
- Nuclear lamins in the brain - new insights into function and regulation. (Q36514069) (← links)
- The contrasting roles of lamin B1 in cellular aging and human disease (Q37260945) (← links)
- Alzheimer's disease: presence and role of microRNAs (Q37280174) (← links)
- Antisense-Based Progerin Downregulation in HGPS-Like Patients' Cells (Q37293792) (← links)
- A High Throughput Phenotypic Screening reveals compounds that counteract premature osteogenic differentiation of HGPS iPS-derived mesenchymal stem cells (Q37339956) (← links)
- New Lmna knock-in mice provide a molecular mechanism for the 'segmental aging' in Hutchinson-Gilford progeria syndrome (Q37593037) (← links)
- Induced pluripotent stem cells reveal functional differences between drugs currently investigated in patients with hutchinson-gilford progeria syndrome (Q37681010) (← links)
- miR-9: a versatile regulator of neurogenesis (Q38169147) (← links)
- Normal and aberrant splicing of LMNA. (Q38181458) (← links)
- Do lamin A and lamin C have unique roles? (Q38256841) (← links)
- Nuclear lamina remodelling and its implications for human disease (Q38297406) (← links)
- A ceRNA analysis on LMNA gene focusing on the Hutchinson-Gilford progeria syndrome (Q38492694) (← links)
- Current status of pluripotent stem cells: moving the first therapies to the clinic. (Q38590664) (← links)
- Back and forth in time: Directing age in iPSC-derived lineages. (Q38642565) (← links)
- Speeding up the clock: The past, present and future of progeria (Q38675558) (← links)
- Alzheimer's disease: An acquired neurodegenerative laminopathy (Q38831862) (← links)
- NF-κB activation impairs somatic cell reprogramming in ageing (Q38848975) (← links)
- Lamin Mutations Accelerate Aging via Defective Export of Mitochondrial mRNAs through Nuclear Envelope Budding (Q41107546) (← links)
- Low lamin A expression in lung adenocarcinoma cells from pleural effusions is a pejorative factor associated with high number of metastatic sites and poor Performance status. (Q41383757) (← links)
- MG132-induced progerin clearance is mediated by autophagy activation and splicing regulation. (Q41601030) (← links)
- Human iPSC-based modeling of late-onset disease via progerin-induced aging (Q41868474) (← links)
- Oleanolic acid has similar effects as retinoic acid in inducing mouse embryonic stem cell 1B10 to differentiate towards germ cells (Q42810178) (← links)
- iPS cells in the study of PD molecular pathogenesis (Q46579323) (← links)
- The Potential of iPSCs for the Treatment of Premature Aging Disorders (Q47105441) (← links)
- Emerging candidate treatment strategies for Hutchinson-Gilford progeria syndrome (Q47414799) (← links)
- Aging as an Epigenetic Phenomenon (Q47590157) (← links)
- Progeria: a paradigm for translational medicine. (Q47917390) (← links)
- miR‑342‑5p promotes Zmpste24‑deficient mouse embryonic fibroblasts proliferation by suppressing GAS2. (Q49229670) (← links)
- Cellular senescence in brain aging and neurodegenerative diseases: evidence and perspectives (Q49851084) (← links)
- Biomechanical Strain Exacerbates Inflammation on a Progeria-on-a-Chip Model. (Q50455443) (← links)
- Pharmacological modulation of LMNA SRSF1-dependent splicing abrogates diet-induced obesity in mice. (Q51285904) (← links)
- MicroRNAs in hereditary and sporadic premature aging syndromes and other laminopathies. (Q52562166) (← links)
- Pathological modelling of pigmentation disorders associated with Hutchinson-Gilford Progeria Syndrome (HGPS) revealed an impaired melanogenesis pathway in iPS-derived melanocytes. (Q55341009) (← links)
- Hippocampal Gene Expression is Increased in Late-Stage Alzheimer's Disease (Q64259971) (← links)
- Are There Common Mechanisms Between the Hutchinson-Gilford Progeria Syndrome and Natural Aging? (Q64955369) (← links)