Pages that link to "Q40907012"
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The following pages link to Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons. (Q40907012):
Displaying 50 items.
- NaN/Nav1.9: a sodium channel with unique properties (Q28215683) (← links)
- Identification of gene expression profile of dorsal root ganglion in the rat peripheral axotomy model of neuropathic pain. (Q31076606) (← links)
- Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediators (Q33639471) (← links)
- Tetrodotoxin (TTX) as a therapeutic agent for pain. (Q34260913) (← links)
- Transcriptional channelopathies: an emerging class of disorders (Q34354397) (← links)
- Central and peripheral roles of prostaglandins in pain and their interactions with novel neuropeptides nociceptin and nocistatin (Q34469703) (← links)
- Are Voltage-Gated Sodium Channels on the Dorsal Root Ganglion Involved in the Development of Neuropathic Pain? (Q34639336) (← links)
- The roles of sodium channels in nociception: Implications for mechanisms of pain (Q34674268) (← links)
- Collagen gel three-dimensional matrices combined with adhesive proteins stimulate neuronal differentiation of mesenchymal stem cells (Q35014463) (← links)
- Neurotrophic factors as novel therapeutics for neuropathic pain (Q35140851) (← links)
- Enhanced nonpeptidergic intraepidermal fiber density and an expanded subset of chloroquine-responsive trigeminal neurons in a mouse model of dry skin itch. (Q35269209) (← links)
- Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons (Q35609134) (← links)
- Voltage-gated Na+ channels: multiplicity of expression, plasticity, functional implications and pathophysiological aspects (Q35667228) (← links)
- Cerebellar dysfunction in multiple sclerosis: evidence for an acquired channelopathy (Q36015265) (← links)
- Biophysical and Pharmacological Characterization of Nav1.9 Voltage Dependent Sodium Channels Stably Expressed in HEK-293 Cells (Q36111427) (← links)
- Animal toxins can alter the function of Nav1.8 and Nav1.9. (Q36246739) (← links)
- Tetrodotoxin-resistant voltage-dependent sodium channels in identified muscle afferent neurons (Q36532591) (← links)
- Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons (Q36678533) (← links)
- Identification of the sensory neuron specific regulatory region for the mouse gene encoding the voltage-gated sodium channel NaV1.8. (Q36925406) (← links)
- Effect of amitriptyline on tetrodotoxin-resistant Nav1.9 currents in nociceptive trigeminal neurons (Q36953269) (← links)
- Transgenic GDNF Positively Influences Proliferation, Differentiation, Maturation and Survival of Motor Neurons Produced from Mouse Embryonic Stem Cells. (Q37246814) (← links)
- Neurotrophin-3 significantly reduces sodium channel expression linked to neuropathic pain states (Q37362605) (← links)
- Understanding inflammatory pain: ion channels contributing to acute and chronic nociception (Q37693805) (← links)
- The role of slow and persistent TTX-resistant sodium currents in acute tumor necrosis factor-α-mediated increase in nociceptors excitability (Q39199255) (← links)
- Comparative study of the distribution of the alpha-subunits of voltage-gated sodium channels in normal and axotomized rat dorsal root ganglion neurons (Q39963179) (← links)
- Reversal of neurochemical changes and pain-related behavior in a model of neuropathic pain using modified lentiviral vectors expressing GDNF. (Q40310929) (← links)
- Pharmacological properties of neuronal TTX-resistant sodium channels and the role of a critical serine pore residue (Q40404449) (← links)
- Beta1 adducin gene expression in DRG is developmentally regulated and is upregulated by glial-derived neurotrophic factor and nerve growth factor (Q40798749) (← links)
- The Non-Survival Effects of Glial Cell Line-Derived Neurotrophic Factor on Neural Cells (Q41525652) (← links)
- Recovery of viscerosensory innervation from the dorsal root ganglia of the adult rat following capsaicin-induced injury. (Q42996663) (← links)
- Glycosylation alters steady-state inactivation of sodium channel Nav1.9/NaN in dorsal root ganglion neurons and is developmentally regulated. (Q43819967) (← links)
- Electrophysiological characterization of the tetrodotoxin-resistant Na+ channel, Na(v)1.9, in mouse dorsal root ganglion neurons (Q45004562) (← links)
- The role of Nav1.9 channel in the development of neuropathic orofacial pain associated with trigeminal neuralgia. (Q45979268) (← links)
- Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis (Q48177322) (← links)
- Sodium channels and pain: from toxins to therapies (Q48337085) (← links)
- The tetrodotoxin-resistant Na+ channel Na (v)1.8 reduces the potency of local anesthetics in blocking C-fiber nociceptors (Q48377936) (← links)
- Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells (Q48422070) (← links)
- Axotomy does not up-regulate expression of sodium channel Na(v)1.8 in Purkinje cells (Q48607314) (← links)
- trkA is expressed in nociceptive neurons and influences electrophysiological properties via Nav1.8 expression in rapidly conducting nociceptors. (Q50484843) (← links)
- Characteristics of late Na(+) current in adult rat small sensory neurons (Q51295135) (← links)
- Selective expression of a persistent tetrodotoxin-resistant Na+ current and NaV1.9 subunit in myenteric sensory neurons. (Q51300821) (← links)
- Developmental expression of the TTX-resistant voltage-gated sodium channels Nav1.8 (SNS) and Nav1.9 (SNS2) in primary sensory neurons. (Q51351071) (← links)
- Intense Isolectin-B4 Binding in Rat Dorsal Root Ganglion Neurons Distinguishes C-Fiber Nociceptors with Broad Action Potentials and High Nav1.9 Expression (Q60736387) (← links)
- Extracellular signal-regulated kinases mediate the enhancing effects of inflammatory mediators on resurgent currents in dorsal root ganglion neurons (Q64073242) (← links)
- Ectopic activity in cutaneous regenerating afferent nerve fibers following nerve lesion in the rat (Q79300419) (← links)
- Pathophysiology of pain (Q79806318) (← links)
- Chapter 5 Molecular mechanisms of nociception and pain (Q82028681) (← links)
- Transfection of rat or mouse neurons by biolistics or electroporation (Q84255216) (← links)
- Transcriptional Regulation of Voltage-Gated Sodium Channels Contributes to GM-CSF-Induced Pain (Q90251219) (← links)
- Sodium channels enable fast electrical signaling and regulate phagocytosis in the retinal pigment epithelium (Q92621627) (← links)