Dopamine D1R-neuron cacna1c deficiency: a new model of extinction therapy-resistant post-traumatic stress (Q93217457)

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4 May 2020

title

Dopamine D1R-neuron cacna1c deficiency: a new model of extinction therapy-resistant post-traumatic stress (English)

1 reference

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4 May 2020

Charlotte C Bavley

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4 May 2020

Zeeba D Kabir

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4 May 2020

Alexander P Walsh

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4 May 2020

Maria Kosovsky

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4 May 2020

Jonathan Hackett

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4 May 2020

Herie Sun

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4 May 2020

Edwin Vázquez-Rosa

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4 May 2020

Coral J Cintrón-Pérez

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4 May 2020

Emiko Miller

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4 May 2020

Yeojung Koh

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4 May 2020

Andrew A Pieper

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4 May 2020

Anjali M Rajadhyaksha

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4 May 2020

publication date

24 April 2020

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4 May 2020

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describes a project that uses

4 May 2020

ImageJ

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC8214244/fullTextXML

inferred from PubMed Central ID database lookup

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Behavioral and neural analysis of extinction

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Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood.

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Forebrain elimination of cacna1c mediates anxiety-like behavior in mice

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Extinction of Contextual Cocaine Memories Requires Cav1.2 within D1R-Expressing Cells and Recruits Hippocampal Cav1.2-Dependent Signaling Mechanisms.

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Rescue of impaired sociability and anxiety-like behavior in adult cacna1c-deficient mice by pharmacologically targeting eIF2α.

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Fear extinction in traumatized civilians with posttraumatic stress disorder: relation to symptom severity

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Hippocampus function predicts severity of post-traumatic stress disorder.

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Hippocampal neurogenesis enhancers promote forgetting of remote fear memory after hippocampal reactivation by retrieval.

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The Neuropsychiatric Disease-Associated Gene cacna1c Mediates Survival of Young Hippocampal Neurons

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Deletion of the Mouse Homolog of CACNA1C Disrupts Discrete Forms of Hippocampal-Dependent Memory and Neurogenesis within the Dentate Gyrus

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