Identification of Tcf-4 as a transcriptional target of p53 signalling (Q42831096)

8 November 2017

title

Identification of Tcf-4 as a transcriptional target of p53 signalling (English)

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8 November 2017

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8 November 2017

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8 November 2017

Tarik Möröy

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8 November 2017

Ludger Klein-Hitpass

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8 November 2017

Cindy Johne

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8 November 2017

Katja Spiesbach

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8 November 2017

Ulrike Haugwitz

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8 November 2017

Katrin Tschöp

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8 November 2017

Mark Wasner

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8 November 2017

Joachim Mössner

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8 November 2017

language of work or name

English

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publication date

1 April 2004

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8 November 2017

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8 November 2017

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8 November 2017

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8 November 2017

page(s)

3376-3384

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Maintenance of genomic integrity by p53: complementary roles for activated and non-activated p53.

8 November 2017

cites work

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A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

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Suppression of human colorectal carcinoma cell growth by wild-type p53

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Functional interaction of beta-catenin with the transcription factor LEF-1

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c-Myc target gene specificity is determined by a post-DNAbinding mechanism

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Radiation-induced cell cycle arrest compromised by p21 deficiency

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p53 suppresses the activation of the Bcl-2 promoter by the Brn-3a POU family transcription factor

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Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control

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Regulation of LEF-1/TCF transcription factors by Wnt and other signals

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WAF1, a potential mediator of p53 tumor suppression

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A genetic model for colorectal tumorigenesis

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Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

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Expression of bbc3, a pro-apoptotic BH3-only gene, is regulated by diverse cell death and survival signals

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7 January 2021

The Hallmarks of Cancer

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Apoptosis or growth arrest: modulation of the cellular response to p53 by proliferative signals

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7 January 2021

p53 regulates a G2 checkpoint through cyclin B1

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Transcriptional repression by p53 through direct binding to a novel DNA element

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Lessons from hereditary colorectal cancer

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Expression of the cell cycle phosphatase cdc25C is down-regulated by the tumor suppressor protein p53 but not by p73

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7 January 2021

p21/CDKN1A mediates negative regulation of transcription by p53.

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7 January 2021

Target genes of beta-catenin-T cell-factor/lymphoid-enhancer-factor signaling in human colorectal carcinomas

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NF-Y mediates the transcriptional inhibition of the cyclin B1, cyclin B2, and cdc25C promoters upon induced G2 arrest

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7 January 2021

Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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7 January 2021

XTcf-3 transcription factor mediates beta-catenin-induced axis formation in Xenopus embryos

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7 January 2021

p53 Protein exhibits 3'-to-5' exonuclease activity

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7 January 2021

The APC tumour suppressor has a nuclear export function.

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7 January 2021

The Hedgehog and Wnt signalling pathways in cancer

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7 January 2021

Regulation and possible function of beta-catenin in human monocytes

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7 January 2021

The beta-catenin/TCF-4 complex imposes a crypt progenitor phenotype on colorectal cancer cells

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7 January 2021

Surfing the p53 network

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7 January 2021

Live or let die: the cell's response to p53

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7 January 2021

Dickkopf-1, an inhibitor of the Wnt signaling pathway, is induced by p53.

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7 January 2021

GADD45 induction of a G2/M cell cycle checkpoint

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7 January 2021

Three CCAAT-boxes and a single cell cycle genes homology region (CHR) are the major regulating sites for transcription from the human cyclin B2 promoter

1 reference

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7 January 2021

Cyclin B1 transcription is enhanced by the p300 coactivator and regulated during the cell cycle by a CHR-dependent repression mechanism

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7 January 2021

Identification and classification of p53-regulated genes

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7 January 2021

p53 negatively regulates cdc2 transcription via the CCAAT-binding NF-Y transcription factor

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1207464

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Dimensions Publication ID

8 November 2017

1003859808

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