Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cells (Q40615490)

22 September 2017

title

Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cells (English)

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22 September 2017

main subject

reactive oxygen species

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Leonard J Buccellato

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22 September 2017

May Tso

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22 September 2017

Ozkan I Akinci

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22 September 2017

Navdeep S Chandel

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22 September 2017

G R Scott Budinger

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22 September 2017

language of work or name

English

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publication date

18 November 2003

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Journal of Biological Chemistry

22 September 2017

published in

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22 September 2017

volume

279

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22 September 2017

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22 September 2017

page(s)

6753-6760

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The cellular production of hydrogen peroxide

22 September 2017

cites work

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Hyperoxic sheep pulmonary microvascular endothelial cells generate free radicals via mitochondrial electron transport

21 January 2018

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Keeping killers on a tight leash: transcriptional and post-translational control of the pro-apoptotic activity of BH3-only proteins

21 January 2018

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Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

21 January 2018

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Hyperoxia-induced apoptosis does not require mitochondrial reactive oxygen species and is regulated by Bcl-2 proteins

21 January 2018

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21 January 2018

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Hyperoxia in cell culture. A non-apoptotic programmed cell death

21 January 2018

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The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.

21 January 2018

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Doxorubicin-induced apoptosis is associated with increased transcription of endothelial nitric-oxide synthase. Effect of antiapoptotic antioxidants and calcium

21 January 2018

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Bcl-x(L) prevents the initial decrease in mitochondrial membrane potential and subsequent reactive oxygen species production during tumor necrosis factor alpha-induced apoptosis

21 January 2018

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Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis

21 January 2018

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Bcl-2 family members and functional electron transport chain regulate oxygen deprivation-induced cell death

21 January 2018

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Akt/Protein kinase B inhibits cell death by preventing the release of cytochrome c from mitochondria

21 January 2018

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Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis

21 January 2018

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Hyperoxia-induced cell death in the lung--the correlation of apoptosis, necrosis, and inflammation

21 January 2018

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Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane

21 January 2018

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

21 January 2018

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BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

21 January 2018

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Interleukin-6-induced protection in hyperoxic acute lung injury

21 January 2018

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Overexpression of manganese superoxide dismutase protects lung epithelial cells against oxidant injury

21 January 2018

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Extracellular superoxide dismutase in the airways of transgenic mice reduces inflammation and attenuates lung toxicity following hyperoxia

21 January 2018

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Transgenic mice with expression of elevated levels of copper-zinc superoxide dismutase in the lungs are resistant to pulmonary oxygen toxicity

21 January 2018

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

21 January 2018

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JNK-mediated BIM phosphorylation potentiates BAX-dependent apoptosis

21 January 2018

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MAPK pathways mediate hyperoxia-induced oncotic cell death in lung epithelial cells

21 January 2018

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Inhibition of c-Jun N-terminal kinase pathway improves cell viability in response to oxidant injury

21 January 2018

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M310145200

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22 September 2017

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