p73 regulates DRAM-independent autophagy that does not contribute to programmed cell death (Q40170381)

18 September 2017

title

p73 regulates DRAM-independent autophagy that does not contribute to programmed cell death (English)

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18 September 2017

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18 September 2017

D Crighton

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18 September 2017

J O'Prey

series ordinal

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18 September 2017

H S Bell

series ordinal

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18 September 2017

publication date

16 February 2007

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Cell Death & Differentiation

18 September 2017

published in

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18 September 2017

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18 September 2017

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18 September 2017

page(s)

1071-1079

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18 September 2017

cites work

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p53 mutation heterogeneity in cancer

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p53 has a direct apoptogenic role at the mitochondria

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Mitochondrial p53 activates Bak and causes disruption of a Bak-Mcl1 complex.

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Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis

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Transcriptional regulation by p53: one protein, many possibilities

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Death squads enlisted by the tumour suppressor p53.

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The coordinate regulation of the p53 and mTOR pathways in cells

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DRAM, a p53-induced modulator of autophagy, is critical for apoptosis

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Autophagy as a regulated pathway of cellular degradation

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Maturation of autophagic vacuoles in Mammalian cells

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Autophagy: dual roles in life and death?

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The role of autophagy during the early neonatal starvation period

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Growth factor regulation of autophagy and cell survival in the absence of apoptosis

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Inhibition of macroautophagy triggers apoptosis

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Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes

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Regulation of an ATG7-beclin 1 program of autophagic cell death by caspase-8

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Monoallelically Expressed Gene Related to p53 at 1p36, a Region Frequently Deleted in Neuroblastoma and Other Human Cancers

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p73 is a simian [correction of human] p53-related protein that can induce apoptosis

21 January 2018

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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

21 January 2018

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p73-deficient mice have neurological, pheromonal and inflammatory defects but lack spontaneous tumours

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p73: Friend or foe in tumorigenesis

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The common and distinct target genes of the p53 family transcription factors

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A ribonucleotide reductase gene is a transcriptional target of p53 and p73

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The dynamics of autophagy visualized in live cells: from autophagosome formation to fusion with endo/lysosomes

21 January 2018

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LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

21 January 2018

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The ubiquitin-protein ligase Itch regulates p73 stability

21 January 2018

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Relief of p53-mediated telomerase suppression by p73.

21 January 2018

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A modified oestrogen receptor ligand-binding domain as an improved switch for the regulation of heterologous proteins

21 January 2018

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A system for stable expression of short interfering RNAs in mammalian cells

21 January 2018

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A simplified system for generating recombinant adenoviruses

21 January 2018

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Two new p73 splice variants, gamma and delta, with different transcriptional activity.

21 January 2018

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Role of NF-κB in p53-mediated programmed cell death

21 January 2018

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Loss of nuclear factor-kappaB is tumor promoting but does not substitute for loss of p53.

21 January 2018

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DNA-binding independent cell death from a minimal proapoptotic region of E2F-1.

21 January 2018

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Characterization of structural p53 mutants which show selective defects in apoptosis but not cell cycle arrest

21 January 2018

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Evolution of functions within the p53/p63/p73 family.

21 January 2018

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.CDD.4402108

1 reference

Dimensions Publication ID

18 September 2017

1011061920

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1 reference