Altered expression of DJ-1 and PINK1 in sporadic ALS and in the SOD1(G93A) ALS mouse model (Q48932124)

16 February 2018

title

Altered expression of DJ-1 and PINK1 in sporadic ALS and in the SOD1(G93A) ALS mouse model (English)

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amyotrophic lateral sclerosis

main subject

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Reinhard Dengler

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Susanne Petri

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Sonja Körner

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Sarah Knippenberg

object named as

Sarah Knippenberg

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Nadine Thau-Habermann

object named as

Nadine Thau-Habermann

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Klaus Jan Rath

object named as

Klaus Jan Rath

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author name string

Julia Sipos

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16 February 2018

publication date

1 November 2013

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Journal of Neuropathology & Experimental Neurology

published in

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volume

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issue

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page(s)

1052-1061

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Hereditary early-onset Parkinson's disease caused by mutations in PINK1

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The kinase domain of mitochondrial PINK1 faces the cytoplasm

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Pink1 regulates the oxidative phosphorylation machinery via mitochondrial fission

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Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

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The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations

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PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

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PINK1-dependent recruitment of Parkin to mitochondria in mitophagy

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Dopamine induced neurodegeneration in a PINK1 model of Parkinson's disease

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Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants

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Parkin negatively regulates JNK pathway in the dopaminergic neurons of Drosophila

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Increased glutathione S-transferase activity rescues dopaminergic neuron loss in a Drosophila model of Parkinson's disease

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El Escorial revisited: revised criteria for the diagnosis of amyotrophic lateral sclerosis

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

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16 February 2018

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16 February 2018