Augmentation of natural immunity to a pro-inflammatory cytokine (TNF-alpha) by targeted DNA vaccine confers long-lasting resistance to experimental autoimmune encephalomyelitis (Q45861565)

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scientific article published in June 1999

Language	Label	Description	Also known as
English	Augmentation of natural immunity to a pro-inflammatory cytokine (TNF-alpha) by targeted DNA vaccine confers long-lasting resistance to experimental autoimmune encephalomyelitis	scientific article published in June 1999

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

Augmentation of natural immunity to a pro-inflammatory cytokine (TNF-alpha) by targeted DNA vaccine confers long-lasting resistance to experimental autoimmune encephalomyelitis (English)

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

author name string

Wildbaum G

1

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

Karin N

2

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

publication date

1 June 1999

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

6

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

6

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

1128-1138

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

Multiple sclerosis (first of two parts).

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Reversal of experimental autoimmune encephalomyelitis by a soluble peptide variant of a myelin basic protein epitope: T cell receptor antagonism and reduction of interferon gamma and tumor necrosis factor alpha production

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Treatment of experimental encephalomyelitis with a peptide analogue of myelin basic protein

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Role of cytokines in determining T-lymphocyte function

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Functional diversity of helper T lymphocytes

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Cytokine transcriptional events during helper T cell subset differentiation

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Interleukin 12 acts directly on CD4+ T cells to enhance priming for interferon gamma production and diminishes interleukin 4 inhibition of such priming

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The presence of interleukin 4 during in vitro priming determines the lymphokine-producing potential of CD4+ T cells from T cell receptor transgenic mice

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TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties

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The role of IL-10 in crossregulation of TH1 and TH2 responses

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Cytokine-induced immune deviation as a therapy for inflammatory autoimmune disease

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B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: application to autoimmune disease therapy

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Prevention of experimental autoimmune encephalomyelitis by antibodies against interleukin 12.

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Encephalitogenic T cells in the B10.PL model of experimental allergic encephalomyelitis (EAE) are of the Th-1 lymphokine subtype

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Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseases.

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Interleukin 4 reverses T cell proliferative unresponsiveness and prevents the onset of diabetes in nonobese diabetic mice

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Interleukin-10 prevents experimental allergic encephalomyelitis in rats.

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Interleukin-12 and its role in the generation of TH1 cells

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TNF is a potent anti-inflammatory cytokine in autoimmune-mediated demyelination

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Human tumor necrosis factor-alpha augments experimental allergic encephalomyelitis in rats

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An antibody to lymphotoxin and tumor necrosis factor prevents transfer of experimental allergic encephalomyelitis

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Anti?tumor necrosis factor therapy abrogates autoimmune demyelination

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Critical Points of Tumor Necrosis Factor Action in Central Nervous System Autoimmune Inflammation Defined by Gene Targeting

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Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice

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A Critical Role for Lymphotoxin in Experimental Allergic Encephalomyelitis

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Tumor necrosis factor alpha and lymphotoxin alpha are not required for induction of acute experimental autoimmune encephalomyelitis.

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Soluble tumor necrosis factor receptor inhibits interleukin 12 production by stimulated human adult microglial cells in vitro

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Genetic immunization is a simple method for eliciting an immune response

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T cell determinants of myelin basic protein include a unique encephalitogenic I-E-restricted epitope for Lewis rats.

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Myelin basic protein‐specific T lymphocyte lines from MS patients and healthy individuals

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Regulatory T cell clones induced by oral tolerance: suppression of autoimmune encephalomyelitis

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Induction of anergy or active suppression following oral tolerance is determined by antigen dosage.

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An interleukin (IL)-10/IL-12 immunoregulatory circuit controls susceptibility to autoimmune disease.

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Escape from "horror autotoxicus": pathogenesis and treatment of autoimmune disease

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Tolerance, danger, and the extended family

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The immune system evolved to discriminate infectious nonself from noninfectious self

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Competition for follicular niches excludes self-reactive cells from the recirculating B-cell repertoire.

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Identifiers

10.1038/SJ.GT.3300915

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

17 December 2017

http://europepmc.org/abstract/MED/10455416

ResearchGate publication ID

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