Depletion of the programmed death-1 receptor completely reverses established clonal anergy in CD4(+) T lymphocytes via an interleukin-2-dependent mechanism (Q33877649)

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English	Depletion of the programmed death-1 receptor completely reverses established clonal anergy in CD4(+) T lymphocytes via an interleukin-2-dependent mechanism	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

29 July 2017

Depletion of the programmed death-1 receptor completely reverses established clonal anergy in CD4(+) T lymphocytes via an interleukin-2-dependent mechanism (English)

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

author name string

Kenneth D Bishop

1

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Europe PubMed Central

PubMed publication ID

29 July 2017

John E Harris

2

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Europe PubMed Central

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29 July 2017

John P Mordes

3

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Europe PubMed Central

PubMed publication ID

29 July 2017

Dale L Greiner

4

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Europe PubMed Central

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29 July 2017

Aldo A Rossini

5

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Europe PubMed Central

PubMed publication ID

29 July 2017

Michael P Czech

6

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Europe PubMed Central

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29 July 2017

Nancy E Phillips

7

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PubMed publication ID

29 July 2017

publication date

23 February 2009

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Europe PubMed Central

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29 July 2017

Cellular Immunology

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Europe PubMed Central

PubMed publication ID

29 July 2017

256

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Europe PubMed Central

PubMed publication ID

29 July 2017

1-2

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Europe PubMed Central

PubMed publication ID

29 July 2017

86-91

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Europe PubMed Central

PubMed publication ID

29 July 2017

Control of peripheral T-cell tolerance and autoimmunity via the CTLA-4 and PD-1 pathways.

1 reference

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PD-1 and its ligands in tolerance and immunity

1 reference

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Antibody-mediated signaling through PD-1 costimulates T cells and enhances CD28-dependent proliferation.

1 reference

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PD-L2:PD-1 involvement in T cell proliferation, cytokine production, and integrin-mediated adhesion

1 reference

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Mechanisms of action of mycophenolate mofetil in preventing acute and chronic allograft rejection

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CTLA-4 and PD-1 receptors inhibit T-cell activation by distinct mechanisms

1 reference

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Induction of transplantation tolerance-the potential of regulatory T cells

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Egr-2 and Egr-3 are negative regulators of T cell activation.

1 reference

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Resting dendritic cells induce peripheral CD8+ T cell tolerance through PD-1 and CTLA-4.

1 reference

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Early growth response gene-2, a zinc-finger transcription factor, is required for full induction of clonal anergy in CD4+ T cells

1 reference

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SHP-1 and SHP-2 associate with immunoreceptor tyrosine-based switch motif of programmed death 1 upon primary human T cell stimulation, but only receptor ligation prevents T cell activation

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CD40/CD154 interactions at the interface of tolerance and immunity

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Calcineurin imposes T cell unresponsiveness through targeted proteolysis of signaling proteins

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CD4+ PD-1+ T cells accumulate as unique anergic cells in rheumatoid arthritis synovial fluid.

1 reference

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GRAIL: an E3 ubiquitin ligase that inhibits cytokine gene transcription is expressed in anergic CD4+ T cells

1 reference

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B7-H1 (programmed death-1 ligand) on dendritic cells is involved in the induction and maintenance of T cell anergy.

1 reference

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5 July 2018

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Mechanisms of transplant tolerance induction using costimulatory blockade

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The enemy within: keeping self-reactive T cells at bay in the periphery

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Islet cell transplantation tolerance

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PD-1:PD-L inhibitory pathway affects both CD4(+) and CD8(+) T cells and is overcome by IL-2

1 reference

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The role of CD4+ T-cell subsets in determining transplantation rejection or tolerance

1 reference

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Immunologic tolerance maintained by CD25+ CD4+ regulatory T cells: their common role in controlling autoimmunity, tumor immunity, and transplantation tolerance

1 reference

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Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

1 reference

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Induction of immunologic tolerance for transplantation

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Essential role of the E3 ubiquitin ligase Cbl-b in T cell anergy induction.

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Acquisition of anergic and suppressive activities in transforming growth factor-beta-costimulated CD4+CD25- T cells

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The gene related to anergy in lymphocytes, an E3 ubiquitin ligase, is necessary for anergy induction in CD4 T cells

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1 reference

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Inhibition of cell cycle progression by rapamycin induces T cell clonal anergy even in the presence of costimulation

1 reference

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The common gammac-cytokines and transplantation tolerance

1 reference

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Identifiers

10.1016/J.CELLIMM.2009.01.008

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Europe PubMed Central

PubMed publication ID

29 July 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

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