BCR-ABL stimulates mutagenic homologous DNA double-strand break repair via the DNA-end-processing factor CtIP. (Q33727572)

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14 January 2020

title

BCR-ABL stimulates mutagenic homologous DNA double-strand break repair via the DNA-end-processing factor CtIP (English)

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14 January 2020

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14 January 2020

Daniela Salles

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14 January 2020

Ivanildce C Ireno

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14 January 2020

Lisa Wiesmüller

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14 January 2020

Eliana Abdelhay

series ordinal

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14 January 2020

language of work or name

English

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publication date

25 October 2010

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14 January 2020

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14 January 2020

volume

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issue

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14 January 2020

page(s)

27-34

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Induction of chronic myelogenous leukemia in mice by the P210bcr/abl gene of the Philadelphia chromosome

14 January 2020

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Human CtIP promotes DNA end resection

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The ERCC1/XPF endonuclease is required for efficient single-strand annealing and gene conversion in mammalian cells

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BCR-ABL promotes the frequency of mutagenic single-strand annealing DNA repair

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BCR/ABL and other kinases from chronic myeloproliferative disorders stimulate single-strand annealing, an unfaithful DNA double-strand break repair

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BCR/ABL regulates mammalian RecA homologs, resulting in drug resistance

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Fusion tyrosine kinases induce drug resistance by stimulation of homology-dependent recombination repair, prolongation of G(2)/M phase, and protection from apoptosis

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BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells

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Down-regulation of BRCA1 in BCR-ABL-expressing hematopoietic cells

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DNA substrate dependence of p53-mediated regulation of double-strand break repair

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Distinct roles of chromatin-associated proteins MDC1 and 53BP1 in mammalian double-strand break repair

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p53 binding protein 1 (53BP1) is an early participant in the cellular response to DNA double-strand breaks

21 January 2018

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CtIP links DNA double-strand break sensing to resection

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Ctp1/CtIP and the MRN complex collaborate in the initial steps of homologous recombination

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21 January 2018

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BRCA1 ubiquitinates its phosphorylation-dependent binding partner CtIP

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