Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities (Q30416844)

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English	Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities	scientific article

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13 October 2017

Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities (English)

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13 October 2017

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Serge N. Schiffmann

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5 June 2020

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D Orduz

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P Gregory

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H Moreno

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U Khan

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K J Vörckel

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D P Wolfer

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H Welzl

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D Gall

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10 March 2015

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13 October 2017

Translational Psychiatry

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5

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5 June 2020

e525

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1 March 2015

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Clustering autism: using neuroanatomical differences in 26 mouse models to gain insight into the heterogeneity

1 reference

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20 June 2018

The cerebellum, sensitive periods, and autism.

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20 June 2018

Patches of disorganization in the neocortex of children with autism

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20 June 2018

Altered neural connectivity in excitatory and inhibitory cortical circuits in autism.

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Affective communication in rodents: ultrasonic vocalizations as a tool for research on emotion and motivation.

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20 June 2018

Exploring the multifactorial nature of autism through computational systems biology: calcium and the Rho GTPase RAC1 under the spotlight

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Activity-dependent neuronal signalling and autism spectrum disorder

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Dysregulation of neural calcium signaling in Alzheimer disease, bipolar disorder and schizophrenia

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20 June 2018

Developmental delays and reduced pup ultrasonic vocalizations but normal sociability in mice lacking the postsynaptic cell adhesion protein neuroligin2.

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20 June 2018

Autistic-like behaviour and cerebellar dysfunction in Purkinje cell Tsc1 mutant mice

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Reduced excitatory neurotransmission and mild autism-relevant phenotypes in adolescent Shank3 null mutant mice

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Networks of neuronal genes affected by common and rare variants in autism spectrum disorders

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20 June 2018

Consensus paper: pathological role of the cerebellum in autism

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Interneuron dysfunction in psychiatric disorders

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20 June 2018

How the 'slow' Ca(2+) buffer parvalbumin affects transmitter release in nanodomain-coupling regimes

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Homeostatic synaptic plasticity: local and global mechanisms for stabilizing neuronal function

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20 June 2018

Homeostatic synaptic plasticity: from single synapses to neural circuits

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20 June 2018

Absence of CNTNAP2 leads to epilepsy, neuronal migration abnormalities, and core autism-related deficits

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20 June 2018

Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function

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20 June 2018

Communication impairments in mice lacking Shank1: reduced levels of ultrasonic vocalizations and scent marking behavior

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The absence of the calcium-buffering protein calbindin is associated with faster age-related decline in hippocampal metabolism.

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20 June 2018

Transcriptomic analysis of autistic brain reveals convergent molecular pathology

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20 June 2018

Behavioral profiles of mouse models for autism spectrum disorders

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Desynchronization of neocortical networks by asynchronous release of GABA at autaptic and synaptic contacts from fast-spiking interneurons.

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20 June 2018

Common circuit defect of excitatory-inhibitory balance in mouse models of autism

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Key role for gene dosage and synaptic homeostasis in autism spectrum disorders

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Role of the calcium-binding protein parvalbumin in short-term synaptic plasticity.

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Major defects in neocortical GABAergic inhibitory circuits in mice lacking the fragile X mental retardation protein

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Parvalbumin-deficiency facilitates repetitive IPSCs and gamma oscillations in the hippocampus

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Parvalbumin deficiency affects network properties resulting in increased susceptibility to epileptic seizures.

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Developmental changes in parvalbumin regulate presynaptic Ca2+ signaling.

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Parvalbumin tunes spike-timing and efferent short-term plasticity in striatal fast spiking interneurons

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The calcium-binding protein parvalbumin modulates the firing 1 properties of the reticular thalamic nucleus bursting neurons.

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