Overexpression of peptidyl-prolyl isomerase Pin1 attenuates hepatocytes apoptosis and secondary necrosis following carbon tetrachloride-induced acute liver injury in mice (Q39423783)

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English	Overexpression of peptidyl-prolyl isomerase Pin1 attenuates hepatocytes apoptosis and secondary necrosis following carbon tetrachloride-induced acute liver injury in mice	scientific article

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Europe PubMed Central

PubMed publication ID

11 September 2017

Overexpression of peptidyl-prolyl isomerase Pin1 attenuates hepatocytes apoptosis and secondary necrosis following carbon tetrachloride-induced acute liver injury in mice (English)

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Europe PubMed Central

PubMed publication ID

11 September 2017

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Prabodh Risal

1

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Europe PubMed Central

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11 September 2017

Byung-Hyun Park

2

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11 September 2017

Baik Hwan Cho

3

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11 September 2017

Jae-Chun Kim

4

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11 September 2017

Yeon Jun Jeong

5

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11 September 2017

publication date

3 November 2011

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11 September 2017

Pathology International

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11 September 2017

62

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11 September 2017

1

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11 September 2017

8-15

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11 September 2017

Hepatotoxicity and Mechanism of Action of Haloalkanes: Carbon Tetrachloride as a Toxicological Model

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor (beta) in fetal hepatocytes.

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The Caspase Inhibitor IDN-6556 Attenuates Hepatic Injury and Fibrosis in the Bile Duct Ligated Mouse

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Prolyl isomerase, Pin1: new findings of post-translational modifications and physiological substrates in cancer, asthma and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Pinning down proline-directed phosphorylation signaling

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Structural and functional analysis of the mitotic rotamase Pin1 suggests substrate recognition is phosphorylation dependent

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The prolyl isomerase PIN1: a pivotal new twist in phosphorylation signalling and disease

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Pin1 in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

A suppressive role of the prolyl isomerase Pin1 in cellular apoptosis mediated by the death-associated protein Daxx

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Phosphorylation-specific prolyl isomerization: is there an underlying theme?

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Fatal liaisons of p53 with Bax and Bak

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The peptidyl-prolyl isomerase Pin1 facilitates cytokine-induced survival of eosinophils by suppressing Bax activation

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The peptidyl-prolyl isomerase, Pin1, facilitates NF-kappaB binding in hepatocytes and protects against hepatic ischemia/reperfusion injury

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Oxidative modification and down-regulation of Pin1 in Alzheimer's disease hippocampus: A redox proteomics analysis

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Evidence of hepatocyte apoptosis in rat liver after the administration of carbon tetrachloride

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Mechanisms of carbon tetrachloride toxicity

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Apoptosis: molecular control point in toxicity.

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Striking LD50 variation associated with fluctuations of CYP2E1-positive cells in hepatic lobule during chronic CCl4 exposure in mice

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The Cdk inhibitor p21 is required for necrosis, but it inhibits apoptosis following toxin-induced liver injury

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Regulation of tumor necrosis factor alpha transcription in macrophages: involvement of four kappa B-like motifs and of constitutive and inducible forms of NF-kappa B

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

The role of tumor necrosis factor-alpha in liver toxicity, inflammation, and fibrosis induced by carbon tetrachloride

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

Increased toxin-induced liver injury and fibrosis in interleukin-6-deficient mice

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1827.2011.02744.X

21 January 2018

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10.1111/J.1440-1827.2011.02744.X

1 reference

Europe PubMed Central

PubMed publication ID

11 September 2017

release_pd5g7rlb75gn7ea3zbrj3vxvh4

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https://api.fatcat.wiki/v0/release/pd5g7rlb75gn7ea3zbrj3vxvh4

24 November 2022

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11 September 2017

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