A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes (Q36594860)

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scientific article published on 7 February 2013

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English	A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes	scientific article published on 7 February 2013

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scholarly article

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Europe PubMed Central

PMC publication ID

15 August 2017

A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes (English)

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Europe PubMed Central

PMC publication ID

15 August 2017

West Nile virus

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systems biology

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based on heuristic

inferred from title

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Michael Gale

9

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Michael G Katze

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Europe PubMed Central

PMC publication ID

15 August 2017

Mehul S. Suthar

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Mehul S Suthar

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Europe PubMed Central

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15 August 2017

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Margaret M Brassil

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Europe PubMed Central

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15 August 2017

Gabriele Blahnik

3

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Europe PubMed Central

PMC publication ID

15 August 2017

Aimee McMillan

4

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Europe PubMed Central

PMC publication ID

15 August 2017

Hilario J Ramos

5

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Europe PubMed Central

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15 August 2017

Sean C Proll

6

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Europe PubMed Central

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15 August 2017

Sarah E Belisle

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Europe PubMed Central

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15 August 2017

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7 February 2013

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Europe PubMed Central

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15 August 2017

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Europe PubMed Central

PMC publication ID

15 August 2017

9

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Europe PubMed Central

PMC publication ID

15 August 2017

2

1 reference

Europe PubMed Central

PMC publication ID

15 August 2017

e1003168

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Europe PubMed Central

PMC publication ID

15 August 2017

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Creative Commons Attribution 4.0 International

7 February 2013

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April 2022 Public Data File from Crossref

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cytoscape

Cytoscape

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CZ Software Mentions: A large dataset of software mentions in the biomedical literature

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The RIG-I-like receptor LGP2 controls CD8(+) T cell survival and fitness

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2'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivo

1 reference

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West Nile virus encephalitis acquired via liver transplantation and clinical response to intravenous immunoglobulin: case report and review of the literature

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2'-O methylation of the viral mRNA cap evades host restriction by IFIT family members

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Modulation of natural killer cell activity by viruses.

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Identification of five interferon-induced cellular proteins that inhibit west nile virus and dengue virus infections

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IPS-1 Is Essential for the Control of West Nile Virus Infection and Immunity

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Anti-West Nile virus activity of in vitro expanded human primary natural killer cells

1 reference

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The IFITM proteins mediate cellular resistance to influenza A H1N1 virus, West Nile virus, and dengue virus

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Distinct and complementary functions of MDA5 and TLR3 in poly(I:C)-mediated activation of mouse NK cells

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IL-10 Signaling Blockade Controls Murine West Nile Virus Infection

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Induction of IFN-β and the Innate Antiviral Response in Myeloid Cells Occurs through an IPS-1-Dependent Signal That Does Not Require IRF-3 and IRF-7

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NKp44 Receptor Mediates Interaction of the Envelope Glycoproteins from the West Nile and Dengue Viruses with NK Cells

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Cardif-Mediated Signaling Controls the Initial Innate Response to Dengue Virus In Vivo

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Dendritic cell interactions with NK cells from different tissues

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Toll-like Receptor 7 Mitigates Lethal West Nile Encephalitis via Interleukin 23-Dependent Immune Cell Infiltration and Homing

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Tissue tropism and neuroinvasion of West Nile virus do not differ for two mouse strains with different survival rates

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Coordinated Regulation and Widespread Cellular Expression of Interferon-Stimulated Genes (ISG) ISG-49, ISG-54, and ISG-56 in the Central Nervous System after Infection with Distinct Viruses

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1 reference

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CD107a as a functional marker for the identification of natural killer cell activity.

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Interaction between conventional dendritic cells and natural killer cells is integral to the activation of effective antiviral immunity.

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Early type I interferon-mediated signals on B cells specifically enhance antiviral humoral responses

1 reference

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12 December 2020

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10.1371/JOURNAL.PPAT.1003168

1 reference

Europe PubMed Central

PMC publication ID

15 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

15 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

15 August 2017

ResearchGate publication ID

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