Apoptotic cells, at all stages of the death process, trigger characteristic signaling events that are divergent from and dominant over those triggered by necrotic cells: Implications for the delayed clearance model of autoimmunity (Q36418402)

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English	Apoptotic cells, at all stages of the death process, trigger characteristic signaling events that are divergent from and dominant over those triggered by necrotic cells: Implications for the delayed clearance model of autoimmunity	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Apoptotic cells, at all stages of the death process, trigger characteristic signaling events that are divergent from and dominant over those triggered by necrotic cells: Implications for the delayed clearance model of autoimmunity (English)

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

apoptotic process

1 reference

based on heuristic

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author name string

Vimal A Patel

1

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Europe PubMed Central

PMC publication ID

14 August 2017

Angelika Longacre

2

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Europe PubMed Central

PMC publication ID

14 August 2017

Kevin Hsiao

3

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Europe PubMed Central

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14 August 2017

Hanli Fan

4

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Europe PubMed Central

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14 August 2017

Fanyong Meng

5

1 reference

Europe PubMed Central

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14 August 2017

Justin E Mitchell

6

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Joyce Rauch

7

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

David S Ucker

8

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14 August 2017

Jerrold S Levine

9

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14 August 2017

language of work or name

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publication date

22 December 2005

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14 August 2017

number of pages

8

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Journal of Biological Chemistry

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14 August 2017

281

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Europe PubMed Central

PMC publication ID

14 August 2017

8

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Europe PubMed Central

PMC publication ID

14 August 2017

4663-4670

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Impaired clearance of dying cells in systemic lupus erythematosus.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Apoptotic cells at the crossroads of tolerance and immunity

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Mannose-binding lectin-deficient mice display defective apoptotic cell clearance but no autoimmune phenotype

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Persistence of apoptotic cells without autoimmune disease or inflammation in CD14-/- mice.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Lithium activates the Wnt and phosphatidylinositol 3-kinase Akt signaling pathways to promote cell survival in the absence of soluble survival factors

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Death-defying immunity: do apoptotic cells influence antigen processing and presentation?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Innate immune discrimination of apoptotic cells: repression of proinflammatory macrophage transcription is coupled directly to specific recognition

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

A blast from the past: clearance of apoptotic cells regulates immune responses.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Delayed apoptotic cell clearance and lupus-like autoimmunity in mice lacking the c-mer membrane tyrosine kinase

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Distinct modes of macrophage recognition for apoptotic and necrotic cells are not specified exclusively by phosphatidylserine exposure

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Corpse clearance defines the meaning of cell death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Role and regulation of 90 kDa ribosomal S6 kinase (RSK) in signal transduction.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Apoptosis, oncosis, and necrosis. An overview of cell death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

30 September 2017

Glucocorticoid augmentation of macrophage capacity for phagocytosis of apoptotic cells is associated with reduced p130Cas expression, loss of paxillin/pyk2 phosphorylation, and high levels of active Rac.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

28 June 2018

Impaired phagocytosis of apoptotic cell material by monocyte-derived macrophages from patients with systemic lupus erythematosus

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

28 June 2018

Macrophages from lupus-prone MRL mice are characterized by abnormalities in Rho activity, cytoskeletal organization, and adhesiveness to extracellular matrix proteins

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Autoimmune disease and impaired uptake of apoptotic cells in MFG-E8-deficient mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Cytokine dysregulation induced by apoptotic cells is a shared characteristic of macrophages from nonobese diabetic and systemic lupus erythematosus-prone mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Tolerance to islet antigens and prevention from diabetes induced by limited apoptosis of pancreatic beta cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

C1q deficiency and autoimmunity: the effects of genetic background on disease expression.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Secondary necrosis is a source of proteolytically modified forms of specific intracellular autoantigens: implications for systemic autoimmunity

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Differential effects of apoptotic versus lysed cells on macrophage production of cytokines: role of proteases

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3504611

21 September 2018

Interleukin-1 dysregulation is an intrinsic defect in macrophages from MRL autoimmune-prone mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Immunosuppressive effects of apoptotic cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Recognizing death: the phagocytosis of apoptotic cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Abnormal regulation of the cytoskeletal regulator Rho typifies macrophages of the major murine models of spontaneous autoimmunity

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cytokine dysregulation induced by apoptotic cells is a shared characteristic of murine lupus

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Impaired uptake of apoptotic cells into tingible body macrophages in germinal centers of patients with systemic lupus erythematosus

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Antigen presentation by macrophages is enhanced by the uptake of necrotic, but not apoptotic, cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Phagocytosis of apoptotic cells by macrophages induces novel signaling events leading to cytokine-independent survival and inhibition of proliferation: activation of Akt and inhibition of extracellular signal-regulated kinases 1 and 2

1 reference

https://pubmed.ncbi.nlm.nih.gov/16377620

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1074/JBC.M508342200

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

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