GABAergic dysfunction in schizophrenia: new treatment strategies on the horizon. (Q36112064)

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11 January 2020

review article

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title

GABAergic dysfunction in schizophrenia: new treatment strategies on the horizon (English)

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11 January 2020

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11 January 2020

Alessandro Guidotti

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11 January 2020

James Auta

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11 January 2020

John M Davis

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11 January 2020

Erbo Dong

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11 January 2020

Dennis R Grayson

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11 January 2020

Xianquan Zhang

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11 January 2020

Erminio Costa

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11 January 2020

publication date

28 April 2005

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11 January 2020

number of pages

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inferred from page(s)

published in

Psychopharmacology

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11 January 2020

volume

180

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11 January 2020

issue

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11 January 2020

page(s)

191-205

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Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics.

11 January 2020

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Interstitial white matter neurons express less reelin and are abnormally distributed in schizophrenia: towards an integration of molecular and morphologic aspects of the neurodevelopmental hypothesis

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Up-regulation of GABAA receptor binding on neurons of the prefrontal cortex in schizophrenic subjects

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Neural synchrony indexes disordered perception and cognition in schizophrenia

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A GABAergic cortical deficit dominates schizophrenia pathophysiology

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Synapse-specific localization of NMDA and GABA(A) receptor subunits revealed by antigen-retrieval immunohistochemistry

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GABA and Schizophrenia: A Review of Basic Science and Clinical Studies

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Diazepam treatment of early signs of exacerbation in schizophrenia

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Dendritic spine density in schizophrenia and depression

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Delayed response deficits produced by local injection of bicuculline into the dorsolateral prefrontal cortex in Japanese macaque monkeys

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Pathology of layer V pyramidal neurons in the prefrontal cortex of patients with schizophrenia.

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Decrease in reelin and glutamic acid decarboxylase67 (GAD67) expression in schizophrenia and bipolar disorder: a postmortem brain study

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Differential hippocampal expression of glutamic acid decarboxylase 65 and 67 messenger RNA in bipolar disorder and schizophrenia

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Density of glutamic acid decarboxylase 67 messenger RNA-containing neurons that express the N-methyl-D-aspartate receptor subunit NR2A in the anterior cingulate cortex in schizophrenia and bipolar disorder

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GABAergic interneurons: implications for understanding schizophrenia and bipolar disorder

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Emerging principles of altered neural circuitry in schizophrenia

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Isodirectional tuning of adjacent interneurons and pyramidal cells during working memory: evidence for microcolumnar organization in PFC.

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Catching up on schizophrenia: natural history and neurobiology

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Valproate corrects the schizophrenia-like epigenetic behavioral modifications induced by methionine in mice

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Dendritic spine hypoplasticity and downregulation of reelin and GABAergic tone in schizophrenia vulnerability.

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Differential synaptic localization of two major gamma-aminobutyric acid type A receptor alpha subunits on hippocampal pyramidal cells

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In Patas monkey, glutamic acid decarboxylase-67 and reelin mRNA coexpression varies in a manner dependent on layers and cortical areas

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Increased GABAA receptor binding in superficial layers of cingulate cortex in schizophrenics

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[3H]muscimol binding sites increased in autopsied brains of chronic schizophrenics

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Reduction in Reelin immunoreactivity in hippocampus of subjects with schizophrenia, bipolar disorder and major depression

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12 December 2020

Imidazenil prevention of alprazolam-induced acquisition deficit in patas monkeys is devoid of tolerance

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12 December 2020

Immunohistochemical and immunoblot study of GABA(A) alpha1 and beta2/3 subunits in the prefrontal cortex of subjects with schizophrenia and bipolar disorder

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Decreased glutamic acid decarboxylase67 messenger RNA expression in a subset of prefrontal cortical gamma-aminobutyric acid neurons in subjects with schizophrenia

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A subclass of prefrontal gamma-aminobutyric acid axon terminals are selectively altered in schizophrenia

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DNA-methyltransferase 1 mRNA is selectively overexpressed in telencephalic GABAergic interneurons of schizophrenia brains

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The reduced neuropil hypothesis: a circuit based model of schizophrenia

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