Glucagon receptor gene deletion in insulin knockout mice modestly reduces blood glucose and ketones but does not promote survival (Q28830385)

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English	Glucagon receptor gene deletion in insulin knockout mice modestly reduces blood glucose and ketones but does not promote survival	scientific article

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instance of

scholarly article

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title

Glucagon receptor gene deletion in insulin knockout mice modestly reduces blood glucose and ketones but does not promote survival (English)

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main subject

knockout mouse

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glucagon

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based on heuristic

inferred from title

author

Timothy J Kieffer

series ordinal

6

object named as

Timothy J Kieffer

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Maureen J Charron

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5

object named as

Maureen J Charron

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author name string

Ursula H Neumann

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1

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Jessica S S Ho

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2

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Majid Mojibian

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3

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Scott D Covey

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4

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language of work or name

English

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publication date

August 2016

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published in

Molecular Metabolism

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stated in

Crossref

volume

5

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issue

8

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page(s)

731-6

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cites work

Lower blood glucose, hyperglucagonemia, and pancreatic alpha cell hyperplasia in glucagon receptor knockout mice

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

16 March 2017

Blockade of glucagon signaling prevents or reverses diabetes onset only if residual β-cells persist.

1 reference

stated in

PubMed Central

reference URL

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retrieved

3 June 2018

Glucagon receptor antibody completely suppresses type 1 diabetes phenotype without insulin by disrupting a novel diabetogenic pathway

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Evidence That in Uncontrolled Diabetes, Hyperglucagonemia Is Required for Ketosis but Not for Increased Hepatic Glucose Production or Hyperglycemia.

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

New insights into adipocyte-specific leptin gene expression

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Metabolic manifestations of insulin deficiency do not occur without glucagon action

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Glucagonocentric restructuring of diabetes: a pathophysiologic and therapeutic makeover

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

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3 June 2018

Pancreatic neuroendocrine tumors in glucagon receptor-deficient mice

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Glucagon receptor knockout prevents insulin-deficient type 1 diabetes in mice

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Disruption of Hepatic Leptin Signaling Protects Mice From Age- and Diet-Related Glucose Intolerance

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

3 June 2018

Phenotypic alterations in insulin-deficient mutant mice.

1 reference

stated in

PubMed Central

reference URL

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retrieved

3 June 2018

Insulin Knockout Mice Have Extended Survival but Volatile Blood Glucose Levels on Leptin Therapy.

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

26 November 2018

Acute disruption of glucagon secretion or action does not improve glucose tolerance in an insulin-deficient mouse model of diabetes.

1 reference

stated in

PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5021664

retrieved

26 November 2018

Absence of glucagon and insulin action reveals a role for the GLP-1 receptor in endogenous glucose production

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/27656411

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Insulin–a growth hormone

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/27656411

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Hyperglucagonemia in diabetic ketoacidosis. Its prevalence and significance

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/27656411

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Evidence for a Major Role for Glucagon in Regulation of Plasma Glucose in Conscious, Nondiabetic, and Alloxan-Induced Diabetic Rabbits

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/27656411

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Documentation of hyperglucagonemia throughout the day in nonobese and obese patients with noninsulin-dependent diabetes mellitus

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/27656411

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

DOI

10.1016/J.MOLMET.2016.05.014

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4811397

OpenCitations bibliographic resource ID

4811397

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4811397

PMC publication ID

5021664

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4811397

PubMed publication ID

27656411

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4811397

 

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