Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain (Q28548202)

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English	Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain	scientific article

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scholarly article

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Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain (English)

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Jeff S McDermott

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Jeff McDermott

9

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author name string

David J Matson

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Darryl T Hamamoto

2

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Howard Bregman

3

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Melanie Cooke

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Erin F DiMauro

5

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Liyue Huang

6

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Danielle Johnson

7

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Xingwen Li

8

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Carrie Morgan

10

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Ben Wilenkin

11

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Annika B Malmberg

12

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Stefan I McDonough

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Donald A Simone

14

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publication date

2015

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10

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9

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e0138140

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copyright license

Creative Commons Attribution 4.0 International

17 September 2015

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April 2022 Public Data File from Crossref

copyright status

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Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain

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Recent progress in sodium channel modulators for pain

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An SCN9A channelopathy causes congenital inability to experience pain

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Advances in targeting voltage-gated sodium channels with small molecules

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Intra- and interfamily phenotypic diversity in pain syndromes associated with a gain-of-function variant of NaV1.7.

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Treatment of Na(v)1.7-mediated pain in inherited erythromelalgia using a novel sodium channel blocker

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Recent developments regarding voltage-gated sodium channel blockers for the treatment of inherited and acquired neuropathic pain syndromes

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Identification of a potent, state-dependent inhibitor of Nav1.7 with oral efficacy in the formalin model of persistent pain.

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Inhibition of neuronal voltage-gated sodium channels by brilliant blue G

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Loss-of-function mutations in sodium channel Nav1.7 cause anosmia.

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Nociceptors: the sensors of the pain pathway

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29 September 2017

Future potential and status of selective sodium channel blockers for the treatment of pain.

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29 September 2017

A peripherally acting Na(v)1.7 sodium channel blocker reverses hyperalgesia and allodynia on rat models of inflammatory and neuropathic pain

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29 September 2017

ProTx-II, a selective inhibitor of NaV1.7 sodium channels, blocks action potential propagation in nociceptors.

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29 September 2017

Nav1.7 expression is increased in painful human dental pulp

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29 September 2017

Distribution of the voltage-gated sodium channel Na(v)1.7 in the rat: expression in the autonomic and endocrine systems.

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The cerebral signature for pain perception and its modulation

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29 September 2017

Nerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.

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The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways

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Transfer of twelve charges is needed to open skeletal muscle Na+ channels

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29 September 2017

Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy

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An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat

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29 September 2017

Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn

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Loss-of-function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations

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Efficient analysis of experimental observations.

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The voltage-gated sodium channel Na(v)1.9 is an effector of peripheral inflammatory pain hypersensitivity.

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Exploring the role of nociceptor-specific sodium channels in pain transmission using Nav1.8 and Nav1.9 knockout mice

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21 January 2018

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

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Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

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3-Oxoisoindoline-1-carboxamides: potent, state-dependent blockers of voltage-gated sodium channel Na(V)1.7 with efficacy in rat pain models

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21 January 2018

Deletion mutation of sodium channel Na(V)1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability.

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21 January 2018

Receptor occupancy and brain free fraction

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21 January 2018

Spontaneous pain, both neuropathic and inflammatory, is related to frequency of spontaneous firing in intact C-fiber nociceptors.

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21 January 2018

Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury

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21 January 2018

Differential activation and classification of cutaneous afferents in the rat

1 reference

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Repetitive activity slows axonal conduction velocity and concomitantly increases mechanical activation threshold in single axons of the rat cranial dura

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Activity-dependent slowing of conduction velocity provides a method for identifying different functional classes of C-fibre in the rat saphenous nerve

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Mechanical and heat sensitization of cutaneous nociceptors after peripheral inflammation in the rat

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Sodium channel slow inactivation and adaptation in C-fibres

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Subcutaneous formalin-induced activity of dorsal horn neurones in the rat: differential response to an intrathecal opiate administered pre or post formalin

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Activity-dependent slowing of conduction differentiates functional subtypes of C fibres innervating human skin

1 reference

https://pubmed.ncbi.nlm.nih.gov/26379236

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0138140

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Consolidated OpenCitations Corpus – April 2017

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2015PLoSO..1038140M

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Consolidated OpenCitations Corpus – April 2017

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PMC publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

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Consolidated OpenCitations Corpus – April 2017

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