Endonuclease G is an apoptotic DNase when released from mitochondria (Q28206238)

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scientific article (publication date: 5 July 2001)

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Language	Label	Description	Also known as
English	Endonuclease G is an apoptotic DNase when released from mitochondria	scientific article (publication date: 5 July 2001)

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instance of

scholarly article

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title

Endonuclease G is an apoptotic DNase when released from mitochondria (English)

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main subject

apoptotic process

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mitochondrion

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based on heuristic

inferred from title

author name string

L Y Li

series ordinal

1

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X Luo

series ordinal

2

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X Wang

series ordinal

3

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language of work or name

English

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publication date

5 July 2001

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published in

Nature

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volume

412

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issue

6842

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page(s)

95-9

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cites work

Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

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7 January 2021

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DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

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7 January 2021

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The 40-kDa subunit of DNA fragmentation factor induces DNA fragmentation and chromatin condensation during apoptosis

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7 January 2021

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A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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7 January 2021

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Resistance to DNA fragmentation and chromatin condensation in mice lacking the DNA fragmentation factor 45

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DNA fragmentation in apoptosis

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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stated in

Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

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7 January 2021

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Chromosomal localization of mitochondrial transcription factor A (TCF6), single-stranded DNA-binding protein (SSBP), and endonuclease G (ENDOG), three human housekeeping genes involved in mitochondrial biogenesis

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Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Primers for mitochondrial DNA replication generated by endonuclease G.

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7 January 2021

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CD95's deadly mission in the immune system

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7 January 2021

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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7 January 2021

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The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

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7 January 2021

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Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

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Action of mitochondrial endonuclease G on DNA damaged by L-ascorbic acid, peplomycin, and cis-diamminedichloroplatinum (II)

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BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

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Crossref

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7 January 2021

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An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Molecular characterization of mitochondrial apoptosis-inducing factor

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues

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Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Differential requirement for caspase 9 in apoptotic pathways in vivo.

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Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

based on heuristic

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Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

based on heuristic

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Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Adult Apaf-1-deficient mice exhibit male infertility

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Crossref

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7 January 2021

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Interdigital cell death can occur through a necrotic and caspase-independent pathway.

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Crossref

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https://api.crossref.org/works/10.1038%2F35083620

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7 January 2021

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Identifiers

DOI

10.1038/35083620

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

530201

Dimensions Publication ID

1009664464

0 references

OpenCitations bibliographic resource ID

530201

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

530201

PubMed publication ID

11452314

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

530201

 

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