Pages that link to "Q34199383"

The following pages link to The atypical cannabinoid O-1602 protects against experimental colitis and inhibits neutrophil recruitment (Q34199383):

Displaying 46 items.

• Alternative targets within the endocannabinoid system for future treatment of gastrointestinal diseases (Q26824588) (← links)
• CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation (Q28072822) (← links)
• Minireview: recent developments in the physiology and pathology of the lysophosphatidylinositol-sensitive receptor GPR55. (Q33636763) (← links)
• Inflammatory and Neuropathic Nociception is Preserved in GPR55 Knockout Mice (Q33681317) (← links)
• Acute Δ(9)-tetrahydrocannabinol blocks gastric hemorrhages induced by the nonsteroidal anti-inflammatory drug diclofenac sodium in mice (Q33739530) (← links)
• The Myc 3' Wnt responsive element regulates neutrophil recruitment after acute colonic injury in mice (Q33923735) (← links)
• Opposing roles of prostaglandin D2 receptors in ulcerative colitis (Q34001819) (← links)
• The atypical cannabinoid O-1602 stimulates food intake and adiposity in rats. (Q34222264) (← links)
• O-1602, an atypical cannabinoid, inhibits tumor growth in colitis-associated colon cancer through multiple mechanisms (Q34298903) (← links)
• Abnormal cannabidiol attenuates experimental colitis in mice, promotes wound healing and inhibits neutrophil recruitment (Q34534223) (← links)
• Palmitoylethanolamide, a naturally occurring lipid, is an orally effective intestinal anti-inflammatory agent (Q34795735) (← links)
• The therapeutic potential of orphan GPCRs, GPR35 and GPR55. (Q35425244) (← links)
• Role of GPR55 during Axon Growth and Target Innervation. (Q36426297) (← links)
• Evidence for the putative cannabinoid receptor (GPR55)-mediated inhibitory effects on intestinal contractility in mice. (Q36544859) (← links)
• A potential role for GPR55 in gastrointestinal functions (Q36864469) (← links)
• Topical and systemic cannabidiol improves trinitrobenzene sulfonic acid colitis in mice (Q36890194) (← links)
• Orphan G protein receptor GPR55 as an emerging target in cancer therapy and management (Q36996563) (← links)
• New blood brothers: the GPR55 and CB2 partnership (Q37871338) (← links)
• Off-target cannabinoid effects mediated by GPR55. (Q37995375) (← links)
• Cannabinoids in pain management: CB1, CB2 and non-classic receptor ligands (Q38212376) (← links)
• Role of G protein-coupled orphan receptors in intestinal inflammation: novel targets in inflammatory bowel diseases (Q38280772) (← links)
• Genetic Manipulation of the Endocannabinoid System (Q38593120) (← links)
• Cannabinoids for treating inflammatory bowel diseases: where are we and where do we go? (Q38918638) (← links)
• The gastrointestinal tract - a central organ of cannabinoid signaling in health and disease (Q38937293) (← links)
• Manipulation of the Endocannabinoid System in Colitis: A Comprehensive Review (Q39025067) (← links)
• Cannabinoid effects on β amyloid fibril and aggregate formation, neuronal and microglial-activated neurotoxicity in vitro. (Q39096624) (← links)
• A role for O-1602 and G protein-coupled receptor GPR55 in the control of colonic motility in mice (Q39432376) (← links)
• Adoptive transfer of helminth antigen-pulsed dendritic cells protects against the development of experimental colitis in mice. (Q41019894) (← links)
• The GPR55 antagonist CID16020046 protects against intestinal inflammation. (Q41638096) (← links)
• Group III phospholipase A2 promotes colitis and colorectal cancer (Q42363727) (← links)
• Eosinophils Contribute to Intestinal Inflammation via Chemoattractant Receptor-homologous Molecule Expressed on Th2 Cells, CRTH2, in Experimental Crohn's Disease. (Q42742502) (← links)
• G protein-coupled receptor 55 (GPR55) expresses differently in patients with Crohn's disease and ulcerative colitis (Q43496370) (← links)
• Functional characterization of CXCR4 in mediating the expression of protein C system in experimental ulcerative colitis (Q47151148) (← links)
• Bladder function in a cannabinoid receptor type 1 knockout mouse (Q47969058) (← links)
• Analgesia and mouse strain influence neuromuscular plasticity in inflamed intestine. (Q48201261) (← links)
• The differential characterization of GPR55 receptor in human peripheral blood reveals a distinctive expression in monocytes and NK cells and a proinflammatory role in these innate cells (Q48475306) (← links)
• GPR55-dependent stimulation of insulin secretion from isolated mouse and human islets of Langerhans (Q51550177) (← links)
• Deletion of G-protein-coupled receptor 55 promotes obesity by reducing physical activity. (Q52148259) (← links)
• Some Prospective Alternatives for Treating Pain: The Endocannabinoid System and Its Putative Receptors GPR18 and GPR55 (Q60916595) (← links)
• Atypical cannabinoid ligands O-1602 and O-1918 administered chronically in diet-induced obesity (Q64230383) (← links)
• The orphan receptor GPR55 drives skin carcinogenesis and is upregulated in human squamous cell carcinomas. (Q64908003) (← links)
• Cytokine Regulation in Human CD4 T Cells by the Aryl Hydrocarbon Receptor and Gq-Coupled Receptors. (Q64943300) (← links)
• Activation of GPR55 increases neural stem cell proliferation and promotes early adult hippocampal neurogenesis (Q89059724) (← links)
• Roles of G protein-coupled receptors in inflammatory bowel disease (Q91632108) (← links)
• Cannabis, Cannabinoids, and the Endocannabinoid System-Is there Therapeutic Potential for Inflammatory Bowel Disease? (Q93002580) (← links)
• O-1602, an Agonist of Atypical Cannabinoid Receptors GPR55, Reverses the Symptoms of Depression and Detrusor Overactivity in Rats Subjected to Corticosterone Treatment (Q98177425) (← links)