Pages that link to "Q30573030"
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The following pages link to Rapamycin prevents epilepsy in a mouse model of tuberous sclerosis complex. (Q30573030):
Displaying 50 items.
- The emerging role of synaptic cell-adhesion pathways in the pathogenesis of autism spectrum disorders (Q22251439) (← links)
- Optimal management of seizures associated with tuberous sclerosis complex: current and emerging options (Q22305852) (← links)
- Strategies for treatment in Alexander disease (Q24596794) (← links)
- Emerging Pharmacotherapies for Neurodevelopmental Disorders (Q24619170) (← links)
- Ribosomal Protein S6 Phosphorylation in the Nervous System: From Regulation to Function (Q26770762) (← links)
- The neurology of mTOR (Q26824286) (← links)
- Recent treatment advances and novel therapeutic approaches in epilepsy (Q26824512) (← links)
- Harnessing the power of metabolism for seizure prevention: focus on dietary treatments (Q26991869) (← links)
- The role of glutamate and its receptors in autism and the use of glutamate receptor antagonists in treatment (Q26998975) (← links)
- Pediatric epileptology (Q27010355) (← links)
- Shedding new light on neurodegenerative diseases through the mammalian target of rapamycin (Q27015640) (← links)
- Potential for treatment of severe autism in tuberous sclerosis complex (Q27021739) (← links)
- Finding a better drug for epilepsy: the mTOR pathway as an antiepileptogenic target (Q27024547) (← links)
- Anti-epileptogenic clinical trial designs in epilepsy: issues and options (Q27027689) (← links)
- Inflammatory mechanisms contribute to the neurological manifestations of tuberous sclerosis complex (Q27308042) (← links)
- Pathogenesis and new candidate treatments for infantile spasms and early life epileptic encephalopathies: A view from preclinical studies (Q27308052) (← links)
- mTOR inhibition suppresses established epilepsy in a mouse model of cortical dysplasia. (Q27312412) (← links)
- Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment (Q27318635) (← links)
- Rapamycin attenuates the development of posttraumatic epilepsy in a mouse model of traumatic brain injury (Q27319659) (← links)
- Hyperactive mTOR signals in the proopiomelanocortin-expressing hippocampal neurons cause age-dependent epilepsy and premature death in mice (Q27332424) (← links)
- Diagnostic methods and treatment options for focal cortical dysplasia (Q28081144) (← links)
- mTOR signaling in tumorigenesis (Q28252695) (← links)
- Translational Mouse Models of Autism: Advancing Toward Pharmacological Therapeutics (Q28394524) (← links)
- Multicenter phase 2 trial of sirolimus for tuberous sclerosis: kidney angiomyolipomas and other tumors regress and VEGF- D levels decrease (Q28476810) (← links)
- Mapping the spatio-temporal pattern of the mammalian target of rapamycin (mTOR) activation in temporal lobe epilepsy (Q28480776) (← links)
- Vigabatrin inhibits seizures and mTOR pathway activation in a mouse model of tuberous sclerosis complex (Q28486250) (← links)
- Gene expression profiling of a hypoxic seizure model of epilepsy suggests a role for mTOR and Wnt signaling in epileptogenesis (Q28533881) (← links)
- Effect of Chronic Administration of Low Dose Rapamycin on Development and Immunity in Young Rats (Q28547057) (← links)
- Upregulation of glutamate transporter GLT-1 by mTOR-Akt-NF-кB cascade in astrocytic oxygen-glucose deprivation (Q28576235) (← links)
- The mammalian target of rapamycin signaling pathway mediates epileptogenesis in a model of temporal lobe epilepsy (Q28579751) (← links)
- Behavioural phenotyping assays for mouse models of autism (Q29618414) (← links)
- Prenatal rapamycin results in early and late behavioral abnormalities in wildtype C57BL/6 mice (Q30444988) (← links)
- Hereditary Genodermatoses with Cancer Predisposition (Q30470596) (← links)
- Impaired astrocytic gap junction coupling and potassium buffering in a mouse model of tuberous sclerosis complex (Q30491063) (← links)
- Modulation of astrocyte glutamate transporters decreases seizures in a mouse model of Tuberous Sclerosis Complex (Q30493205) (← links)
- Pharmacological inhibition of the mammalian target of rapamycin pathway suppresses acquired epilepsy (Q30496223) (← links)
- Tsc2 gene inactivation causes a more severe epilepsy phenotype than Tsc1 inactivation in a mouse model of tuberous sclerosis complex. (Q30497818) (← links)
- A pulse rapamycin therapy for infantile spasms and associated cognitive decline (Q30501073) (← links)
- The differential effects of prenatal and/or postnatal rapamycin on neurodevelopmental defects and cognition in a neuroglial mouse model of tuberous sclerosis complex. (Q30518266) (← links)
- Excessive activation of mTOR in postnatally generated granule cells is sufficient to cause epilepsy (Q30525675) (← links)
- Mammalian target of rapamycin complex 1 activation negatively regulates Polo-like kinase 2-mediated homeostatic compensation following neonatal seizures. (Q30538200) (← links)
- Rapamycin prevents seizures after depletion of STRADA in a rare neurodevelopmental disorder (Q30541746) (← links)
- Pharmacological inhibition of mTORC1 suppresses anatomical, cellular, and behavioral abnormalities in neural-specific Pten knock-out mice (Q30569745) (← links)
- Searching for the ideal antiepileptogenic agent in experimental models: single treatment versus combinatorial treatment strategies (Q30576762) (← links)
- Selective suppression of excessive GluN2C expression rescues early epilepsy in a tuberous sclerosis murine model. (Q30586288) (← links)
- Mechanism-based treatment in tuberous sclerosis complex (Q30746587) (← links)
- Diffusion tensor imaging and related techniques in tuberous sclerosis complex: review and future directions. (Q30747071) (← links)
- Rapamycin weekly maintenance dosing and the potential efficacy of combination sorafenib plus rapamycin but not atorvastatin or doxycycline in tuberous sclerosis preclinical models (Q33430534) (← links)
- Inhibition of the mammalian target of rapamycin signaling pathway suppresses dentate granule cell axon sprouting in a rodent model of temporal lobe epilepsy (Q33642375) (← links)
- Rapamycin ameliorates PKD resulting from conditional inactivation of Pkd1. (Q33702386) (← links)