Pages that link to "Q24313068"
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The following pages link to ITCH K63-ubiquitinates the NOD2 binding protein, RIP2, to influence inflammatory signaling pathways (Q24313068):
Displaying 50 items.
- Itchy E3 ubiquitin protein ligase (Q21109507) (← links)
- The innate immune sensor NLRC3 attenuates Toll-like receptor signaling via modification of the signaling adaptor TRAF6 and transcription factor NF-κB (Q24294960) (← links)
- Activation of interferon regulatory factor 5 by site specific phosphorylation (Q24307504) (← links)
- A novel motif in the Crohn's disease susceptibility protein, NOD2, allows TRAF4 to down-regulate innate immune responses (Q24594581) (← links)
- Ubiquitin signaling in immune responses (Q26751376) (← links)
- Ubiquitin in the activation and attenuation of innate antiviral immunity (Q26771789) (← links)
- NOD-Like Receptors: Master Regulators of Inflammation and Cancer (Q26865199) (← links)
- RIP2 activity in inflammatory disease and implications for novel therapeutics (Q27025050) (← links)
- Regulation of inflammatory and antiviral signaling by A20. (Q27690854) (← links)
- The ubiquitin system: a critical regulator of innate immunity and pathogen-host interactions (Q28067104) (← links)
- International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease (Q28081406) (← links)
- CYLD Limits Lys63- and Met1-Linked Ubiquitin at Receptor Complexes to Regulate Innate Immune Signaling (Q28117541) (← links)
- Pellino3 ubiquitinates RIP2 and mediates Nod2-induced signaling and protective effects in colitis (Q28513622) (← links)
- Late domain-independent rescue of a release-deficient Moloney murine leukemia virus by the ubiquitin ligase itch (Q33558373) (← links)
- Human ITCH E3 ubiquitin ligase deficiency causes syndromic multisystem autoimmune disease. (Q33708589) (← links)
- E3 Ubiquitin ligase ZNRF4 negatively regulates NOD2 signalling and induces tolerance to MDP. (Q33861394) (← links)
- Receptor-interacting protein (RIP) kinase family. (Q34109567) (← links)
- NOD2 downregulates colonic inflammation by IRF4-mediated inhibition of K63-linked polyubiquitination of RICK and TRAF6. (Q34253078) (← links)
- A genome-wide small interfering RNA (siRNA) screen reveals nuclear factor-κB (NF-κB)-independent regulators of NOD2-induced interleukin-8 (IL-8) secretion (Q34317315) (← links)
- Inhibition of RIP2's tyrosine kinase activity limits NOD2-driven cytokine responses. (Q34362422) (← links)
- NOD1 and NOD2: signaling, host defense, and inflammatory disease (Q34745603) (← links)
- RIP kinases: key decision makers in cell death and innate immunity (Q34924104) (← links)
- Emerging significance of NLRs in inflammatory bowel disease (Q35414220) (← links)
- Inflammatory Signaling by NOD-RIPK2 Is Inhibited by Clinically Relevant Type II Kinase Inhibitors. (Q36084130) (← links)
- p63 threonine phosphorylation signals the interaction with the WW domain of the E3 ligase Itch (Q36183728) (← links)
- Receptor-Interacting Protein Kinase-2 Inhibition by CYLD Impairs Antibacterial Immune Responses in Macrophages (Q36474924) (← links)
- Blau syndrome-associated Nod2 mutation alters expression of full-length NOD2 and limits responses to muramyl dipeptide in knock-in mice (Q36490486) (← links)
- A discrete ubiquitin-mediated network regulates the strength of NOD2 signaling (Q36506610) (← links)
- Ubiquitin regulates caspase recruitment domain-mediated signaling by nucleotide-binding oligomerization domain-containing proteins NOD1 and NOD2. (Q36666117) (← links)
- Monoubiquitination of survival motor neuron regulates its cellular localization and Cajal body integrity (Q36676833) (← links)
- Ubiquitin e3 ligase itch negatively regulates osteoblast differentiation from mesenchymal progenitor cells (Q37135209) (← links)
- NOD2, an intracellular innate immune sensor involved in host defense and Crohn's disease (Q37172148) (← links)
- Functions of NOD-Like Receptors in Human Diseases (Q37231612) (← links)
- Ubiquitin E3 ligase Itch negatively regulates osteoclast formation by promoting deubiquitination of tumor necrosis factor (TNF) receptor-associated factor 6. (Q37311866) (← links)
- Clomipramine causes osteoporosis by promoting osteoclastogenesis via E3 ligase Itch, which is prevented by Zoledronic acid (Q37618061) (← links)
- Ubiquitin E3 ligase Itch negatively regulates osteoblast function by promoting proteasome degradation of osteogenic proteins (Q37733838) (← links)
- Deubiquitinases in the regulation of NF-κB signaling (Q37814068) (← links)
- The role of NOD1 and NOD2 in host defense against chlamydial infection (Q37835976) (← links)
- Peptidoglycan: a critical activator of the mammalian immune system during infection and homeostasis (Q37925062) (← links)
- Regulation of NF-κB by deubiquitinases (Q37995682) (← links)
- Ubiquitination and phosphorylation in the regulation of NOD2 signaling and NOD2-mediated disease (Q38004338) (← links)
- Recognition of bacterial infection by innate immune sensors (Q38032312) (← links)
- NOD proteins: regulators of inflammation in health and disease (Q38171221) (← links)
- Met1-linked ubiquitination in immune signalling (Q38233678) (← links)
- Insights into the molecular basis of the NOD2 signalling pathway (Q38292456) (← links)
- Multifaceted role of the ubiquitin ligase Itch in immune regulation (Q38315883) (← links)
- The E3 ligase Itch in immune regulation and beyond. (Q38530613) (← links)
- Molecular and physiological roles of Pellino E3 ubiquitin ligases in immunity (Q38530624) (← links)
- Aberrant Th2 inflammation drives dysfunction of alveolar macrophages and susceptibility to bacterial pneumonia (Q38928757) (← links)
- Itch, an E3 ligase of Oct4, is required for embryonic stem cell self-renewal and pluripotency induction. (Q39223300) (← links)