Pages that link to "Q73487705"
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The following pages link to Activation of a pro-apoptotic amplification loop through inhibition of NF-kappaB-dependent survival signals by caspase-mediated inactivation of RIP (Q73487705):
Displaying 50 items.
- The mitochondrial serine protease HtrA2/Omi cleaves RIP1 during apoptosis of Ba/F3 cells induced by growth factor withdrawal (Q24298215) (← links)
- The subunit CSN6 of the COP9 signalosome is cleaved during apoptosis (Q24299081) (← links)
- Caspase-8 prevents sustained activation of NF-kappaB in monocytes undergoing macrophagic differentiation (Q24307493) (← links)
- Autoproteolysis of PIDD marks the bifurcation between pro-death caspase-2 and pro-survival NF-kappaB pathway (Q24328745) (← links)
- RIP4 (DIK/PKK), a novel member of the RIP kinase family, activates NF-kappa B and is processed during apoptosis. (Q24538893) (← links)
- Ripped to death (Q24632980) (← links)
- Elevated NF-kappaB responses and FLIP levels in leukemic but not normal lymphocytes: reduction by salicylate allows TNF-induced apoptosis (Q24682686) (← links)
- Necroptosis: molecular signalling and translational implications (Q27015896) (← links)
- IG20, in contrast to DENN-SV, (MADD splice variants) suppresses tumor cell survival, and enhances their susceptibility to apoptosis and cancer drugs (Q28238134) (← links)
- N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-kappaB signaling pathway (Q28629323) (← links)
- Ubiquitination of RIP1 regulates an NF-kappaB-independent cell-death switch in TNF signaling (Q28757756) (← links)
- Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment (Q33589801) (← links)
- Receptor-interacting protein shuttles between cell death and survival signaling pathways (Q33623823) (← links)
- RIP3 overexpression sensitizes human breast cancer cells to parthenolide in vitro via intracellular ROS accumulation (Q33869714) (← links)
- Suppression of MMP-2 Attenuates TNF-α Induced NF-κB Activation and Leads to JNK Mediated Cell Death in Glioma (Q33900925) (← links)
- The most unkindest cut of all: on the multiple roles of mammalian caspases (Q34002897) (← links)
- NF-κB inhibition significantly upregulates the norepinephrine transporter system, causes apoptosis in pheochromocytoma cell lines and prevents metastasis in an animal model (Q34038994) (← links)
- Tumor necrosis factor signaling. (Q34185821) (← links)
- TRAIL in cancer therapy: present and future challenges (Q34291312) (← links)
- Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells. (Q34299368) (← links)
- Nuclear factor kappa-B and the heart (Q34333586) (← links)
- Unique and overlapping substrate specificities of caspase-8 and caspase-10. (Q34454210) (← links)
- Signaling to gene activation and cell death by tumor necrosis factor receptors and Fas. (Q34563012) (← links)
- Disassembly of the JIP1/JNK molecular scaffold by caspase-3-mediated cleavage of JIP1 during apoptosis (Q34713655) (← links)
- RIP kinases: key decision makers in cell death and innate immunity (Q34924104) (← links)
- Apoptosis-independent functions of killer caspases. (Q35021220) (← links)
- Non-apoptotic Fas signaling. (Q35027104) (← links)
- Proteolytic signaling by TNFalpha: caspase activation and IkappaB degradation (Q35161589) (← links)
- The ribonucleotide reductase R1 subunits of herpes simplex virus 1 and 2 protect cells against poly(I · C)-induced apoptosis (Q35193001) (← links)
- Diversity in the mechanisms of neuronal cell death. (Q35558449) (← links)
- Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β. (Q35671251) (← links)
- TRAIL and NFkappaB signaling--a complex relationship (Q35756866) (← links)
- Effector CD4+ T cells generate intermediate caspase activity and cleavage of caspase-8 substrates (Q35910540) (← links)
- The N-end rule pathway counteracts cell death by destroying proapoptotic protein fragments (Q36079454) (← links)
- Death receptors and caspases: role in lymphocyte proliferation, cell death, and autoimmunity (Q36290306) (← links)
- The CASBAH: a searchable database of caspase substrates (Q36726238) (← links)
- Transcriptional profiling of swine lung tissue after experimental infection with Actinobacillus pleuropneumoniae (Q36913786) (← links)
- Antigen-mediated T cell expansion regulated by parallel pathways of death (Q36970979) (← links)
- CYLD Proteolysis Protects Macrophages from TNF-Mediated Auto-necroptosis Induced by LPS and Licensed by Type I IFN. (Q37008848) (← links)
- Chronicles of a death foretold: dual sequential cell death checkpoints in TNF signaling (Q37710949) (← links)
- Focal adhesion kinase suppresses apoptosis by binding to the death domain of receptor-interacting protein (Q37732113) (← links)
- Proliferative versus apoptotic functions of caspase-8 Hetero or homo: the caspase-8 dimer controls cell fate (Q37894323) (← links)
- RIP1-mediated regulation of lymphocyte survival and death responses (Q37950926) (← links)
- Regulating the balance between necroptosis, apoptosis and inflammation by inhibitors of apoptosis proteins. (Q39025072) (← links)
- Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1. (Q39143432) (← links)
- Activation of TNFR2 sensitizes macrophages for TNFR1-mediated necroptosis (Q39144288) (← links)
- Cellular FLICE-inhibitory protein (cFLIP) isoforms block CD95- and TRAIL death receptor-induced gene induction irrespective of processing of caspase-8 or cFLIP in the death-inducing signaling complex (Q39567110) (← links)
- The caspase-generated fragments of PKR cooperate to activate full-length PKR and inhibit translation (Q40167543) (← links)
- Granulocyte macrophage-colony stimulating factor and interleukin-3 increase expression of type II tumour necrosis factor receptor, increasing susceptibility to tumour necrosis factor-induced apoptosis. Control of leukaemia cell life/death switching (Q40508783) (← links)
- Drug-mediated sensitization to TRAIL-induced apoptosis in caspase-8-complemented neuroblastoma cells proceeds via activation of intrinsic and extrinsic pathways and caspase-dependent cleavage of XIAP, Bcl-xL and RIP. (Q40565103) (← links)