Pages that link to "Q48151820"

The following pages link to The p21-activated kinase 3 implicated in mental retardation regulates spine morphogenesis through a Cdc42-dependent pathway (Q48151820):

Displaying 47 items.

• PAK signalling in neuronal physiology (Q24318480) (← links)
• Local, persistent activation of Rho GTPases during plasticity of single dendritic spines (Q24629206) (← links)
• Kalirin regulates cortical spine morphogenesis and disease-related behavioral phenotypes (Q24655969) (← links)
• Dendritic spine pathology in schizophrenia (Q26991935) (← links)
• p21-Activated kinase 3 is overexpressed in thymic neuroendocrine tumors (carcinoids) with ectopic ACTH syndrome and participates in cell migration (Q28296152) (← links)
• Distinct, but compensatory roles of PAK1 and PAK3 in spine morphogenesis (Q28574760) (← links)
• p21-Activated kinases 1 and 3 control brain size through coordinating neuronal complexity and synaptic properties (Q28590676) (← links)
• The RhoGEF DOCK10 is essential for dendritic spine morphogenesis (Q28771510) (← links)
• Identification of a Protein Network Driving Neuritogenesis of MGE-Derived GABAergic Interneurons. (Q30354530) (← links)
• Impaired fear response in mice lacking GIT1. (Q30480461) (← links)
• Interaction of Cupidin/Homer2 with two actin cytoskeletal regulators, Cdc42 small GTPase and Drebrin, in dendritic spines (Q33421135) (← links)
• Identification of neuronal substrates implicates Pak5 in synaptic vesicle trafficking (Q34257319) (← links)
• Intellectual disability and autism spectrum disorders: causal genes and molecular mechanisms (Q34289505) (← links)
• Structural plasticity: mechanisms and contribution to developmental psychiatric disorders (Q34442643) (← links)
• PAK1 protein expression in the auditory cortex of schizophrenia subjects (Q34683597) (← links)
• Parental binge alcohol abuse alters F1 generation hypothalamic gene expression in the absence of direct fetal alcohol exposure (Q35107054) (← links)
• Neuronal apoptosis induced by selective inhibition of Rac GTPase versus global suppression of Rho family GTPases is mediated by alterations in distinct mitogen-activated protein kinase signaling cascades (Q35351477) (← links)
• p21-activated Kinase 3 (PAK3) Protein Regulates Synaptic Transmission through Its Interaction with the Nck2/Grb4 Protein Adaptor (Q35562644) (← links)
• The p21-activated kinase PAK3 forms heterodimers with PAK1 in brain implementing trans-regulation of PAK3 activity (Q36216292) (← links)
• Characterization of a Cdc42 protein inhibitor and its use as a molecular probe (Q36708621) (← links)
• Neural palmitoyl-proteomics reveals dynamic synaptic palmitoylation (Q37030648) (← links)
• Sequence analysis of P21-activated kinase 3 (PAK3) in chronic schizophrenia with cognitive impairment (Q37074452) (← links)
• Regulation of synaptic Rac1 activity, long-term potentiation maintenance, and learning and memory by BCR and ABR Rac GTPase-activating proteins. (Q37361588) (← links)
• Rpph1 Upregulates CDC42 Expression and Promotes Hippocampal Neuron Dendritic Spine Formation by Competing with miR-330-5p (Q37627142) (← links)
• Dendritic spine abnormalities in mental retardation (Q37809762) (← links)
• Emerging major synaptic signaling pathways involved in intellectual disability (Q37949205) (← links)
• Deconstructing signal transduction pathways that regulate the actin cytoskeleton in dendritic spines (Q37981628) (← links)
• The role of small GTPases in neuronal morphogenesis and polarity (Q38011133) (← links)
• Mouse models of PAK function (Q38060731) (← links)
• The role of human-specific gene duplications during brain development and evolution. (Q38115583) (← links)
• The neurobiology of X-linked intellectual disability. (Q38118810) (← links)
• Dendritic spines: the locus of structural and functional plasticity (Q38175135) (← links)
• Overexpressed Down Syndrome Cell Adhesion Molecule (DSCAM) Deregulates P21-Activated Kinase (PAK) Activity in an In Vitro Neuronal Model of Down Syndrome: Consequences on Cell Process Formation and Extension (Q38787153) (← links)
• Alterations in Prefrontal Cortical Circuitry and Cognitive Dysfunction in Schizophrenia. (Q38954270) (← links)
• Expression of p21-activated kinases 1 and 3 is altered in the brain of subjects with depression (Q41576968) (← links)
• Next-Generation Sequencing Reveals Novel Mutations in X-linked Intellectual Disability (Q41919817) (← links)
• A mutation in PAK3 with a dual molecular effect deregulates the RAS/MAPK pathway and drives an X-linked syndromic phenotype. (Q41930624) (← links)
• Activating Transcription Factor 4 (ATF4) modulates Rho GTPase levels and function via regulation of RhoGDIα. (Q42369182) (← links)
• A delta-catenin signaling pathway leading to dendritic protrusions (Q46352528) (← links)
• Improvement of Self-Injury With Dopamine and Serotonin Replacement Therapy in a Patient With a Hemizygous PAK3 Mutation: A New Therapeutic Strategy for Neuropsychiatric Features of an Intellectual Disability Syndrome (Q47274476) (← links)
• Functional analysis of rare variants found in schizophrenia implicates a critical role for GIT1-PAK3 signaling in neuroplasticity (Q48143208) (← links)
• Nigral dopaminergic PAK4 prevents neurodegeneration in rat models of Parkinson's disease (Q48411251) (← links)
• Alteration of synaptic network dynamics by the intellectual disability protein PAK3. (Q48716662) (← links)
• Activating Mutations in PAK1, Encoding p21-Activated Kinase 1, Cause a Neurodevelopmental Disorder (Q57023907) (← links)
• Transcriptional Downregulation of miR-4306 serves as a New Therapeutic Target for Triple Negative Breast Cancer (Q64238313) (← links)
• Cdc42 Regulates Neuronal Polarity during Cerebellar Axon Formation and Glial-Guided Migration (Q89059659) (← links)
• Novel PAK3 gene missense variant associated with two Chinese siblings with intellectual disability: a case report (Q89684195) (← links)