Pages that link to "Q43976165"
Jump to navigation
Jump to search
The following pages link to Endogenous endothelium-derived nitric oxide inhibits myocardial caspase activity: implications for treatment of end-stage heart failure (Q43976165):
Displaying 12 items.
- Apoptosis: biochemical aspects and clinical implications (Q33185094) (← links)
- Non-nuclear actions of estrogen: new targets for prevention and treatment of cardiovascular disease (Q35679460) (← links)
- Genetic maneuvers to ameliorate ventricular function in heart failure: therapeutic potential and future implications (Q36050486) (← links)
- Intervention for apoptosis in cardiomyopathy (Q37031992) (← links)
- Simvastatin preserves cardiac function in genetically determined cardiomyopathy (Q37098893) (← links)
- Endothelial nitric oxide synthase (NOS) deficiency affects energy metabolism pattern in murine oxidative skeletal muscle. (Q41954913) (← links)
- Increased B-type-natriuretic peptide promotes myocardial cell apoptosis via the B-type-natriuretic peptide/long non-coding RNA LSINCT5/caspase-1/interleukin 1β signaling pathway (Q43196441) (← links)
- Apoptosis and necrosis induced by cyclic mechanical stretching in alveolar type II cells (Q44575987) (← links)
- Voluntary physical activity alterations in endothelial nitric oxide synthase knockout mice. (Q44995349) (← links)
- Endothelial hypoxic preconditioning in rat hypoxic isolated aortic segments. (Q46381206) (← links)
- Ovariectomy augments pressure overload-induced hypertrophy associated with changes in Akt and nitric oxide synthase signaling pathways in female rats (Q51736896) (← links)
- Role of oxidative-nitrosative stress and downstream pathways in various forms of cardiomyopathy and heart failure. (Q55663138) (← links)