Pages that link to "Q43816373"

The following pages link to Lung tissue eosinophils may be cleared through luminal entry rather than apoptosis: effects of steroid treatment (Q43816373):

Displaying 23 items.

• Anti-Fas mAb-induced apoptosis and cytolysis of airway tissue eosinophils aggravates rather than resolves established inflammation (Q24814265) (← links)
• Polarized localization of epithelial CXCL11 in chronic obstructive pulmonary disease and mechanisms of T cell egression (Q30481534) (← links)
• Occurrence of apoptosis, secondary necrosis, and cytolysis in eosinophilic nasal polyps (Q33204402) (← links)
• Metabolomic biomarkers in a model of asthma exacerbation: urine nuclear magnetic resonance (Q33377897) (← links)
• Early phase resolution of mucosal eosinophilic inflammation in allergic rhinitis (Q33866414) (← links)
• Asthma, airway biology, and nasal disorders in AJRCCM 2001. (Q34550135) (← links)
• Withdrawal of fluticasone propionate from combined salmeterol/fluticasone treatment in patients with COPD causes immediate and sustained disease deterioration: a randomised controlled trial (Q35537740) (← links)
• Antagonism of the prostaglandin D2 receptor CRTH2 attenuates asthma pathology in mouse eosinophilic airway inflammation (Q35663953) (← links)
• Epithelial Rho GTPases and the transepithelial migration of lymphocytes. (Q37029743) (← links)
• Apoptosis, airway inflammation and anti-asthma therapy: from immunobiology to clinical application (Q37052175) (← links)
• Targeting eosinophils in asthma (Q37056577) (← links)
• Eosinophil viability is increased by acidic pH in a cAMP- and GPR65-dependent manner (Q37372842) (← links)
• Resolution of cell-mediated airways diseases (Q37764375) (← links)
• Resolution of leucocyte-mediated mucosal diseases. A novel in vivo paradigm for drug development (Q37952781) (← links)
• Hypoxia causes IL-8 secretion, Charcot Leyden crystal formation, and suppression of corticosteroid-induced apoptosis in human eosinophils. (Q39078255) (← links)
• Mapracorat, a selective glucocorticoid receptor agonist, causes apoptosis of eosinophils infiltrating the conjunctiva in late-phase experimental ocular allergy (Q40816092) (← links)
• Rapid and efficient clearance of airway tissue granulocytes through transepithelial migration (Q42014635) (← links)
• Effects of steroid treatment on lung CC chemokines, apoptosis and transepithelial cell clearance during development and resolution of allergic airway inflammation. (Q42225465) (← links)
• Altered allergen-induced eosinophil trafficking and physiological dysfunction in airways with preexisting virus-induced injury (Q45413441) (← links)
• Eosinophil-epithelial cell interactions: an important facet of asthmatic inflammation (Q51714845) (← links)
• Allergic Bronchopulmonary Aspergillosis-A Luminal Hypereosinophilic Disease With Extracellular Trap Cell Death (Q58572838) (← links)
• Unbalanced research (Q74587281) (← links)
• Quantitative Assessment of Pulmonary Targeting of Inhaled Corticosteroids Using Ex Vivo Receptor Binding Studies (Q89459627) (← links)