Pages that link to "Q43680987"
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The following pages link to Effect of overexpression of wild-type and mutant Cu/Zn-superoxide dismutases on oxidative stress and cell death induced by hydrogen peroxide, 4-hydroxynonenal or serum deprivation: potentiation of injury by ALS-related mutant superoxide dismutases a (Q43680987):
Displaying 30 items.
- Mitochondrial damage due to SOD1 deficiency in SH-SY5Y neuroblastoma cells: a rationale for the redundancy of SOD1 (Q24294416) (← links)
- An RNA polymerase II construct synthesizes short-hairpin RNA with a quantitative indicator and mediates highly efficient RNAi (Q24806293) (← links)
- Oxidative stress in neurodegeneration: cause or consequence? (Q28275946) (← links)
- X-Linked inhibitor of apoptosis protein is involved in mutant SOD1-mediated neuronal degeneration (Q28506929) (← links)
- A drastic superoxide-dependent oxidative stress is prerequisite for the down-regulation of IRP1: Insights from studies on SOD1-deficient mice and macrophages treated with paraquat. (Q33707880) (← links)
- Role of free radicals in the neurodegenerative diseases: therapeutic implications for antioxidant treatment (Q34400646) (← links)
- Modification of brain aging and neurodegenerative disorders by genes, diet, and behavior (Q34711201) (← links)
- Mitochondrial function in human neuroblastoma cells is up-regulated and protected by NQO1, a plasma membrane redox enzyme (Q34852496) (← links)
- Activation of a glycine transporter on spinal cord neurons causes enhanced glutamate release in a mouse model of amyotrophic lateral sclerosis. (Q35045105) (← links)
- Measuring reactive species and oxidative damage in vivo and in cell culture: how should you do it and what do the results mean? (Q35047134) (← links)
- Prospects for antiapoptotic drug therapy of neurodegenerative diseases (Q35092261) (← links)
- Potentiation of cellular antioxidant capacity by Bcl-2: implications for its antiapoptotic function (Q35557570) (← links)
- Relevance of oxidative injury in the pathogenesis of motor neuron diseases (Q35644080) (← links)
- Proteolytic dysfunction in neurodegenerative disorders (Q35941669) (← links)
- Advances in cellular models to explore the pathophysiology of amyotrophic lateral sclerosis. (Q38160415) (← links)
- Ordering of neuronal apoptosis signaling: a superoxide burst precedes mitochondrial cytochrome c release in a growth factor deprivation model. (Q39381069) (← links)
- Sonic hedgehog is cytoprotective against oxidative challenge in a cellular model of amyotrophic lateral sclerosis (Q39462008) (← links)
- Site-specific effects of peptide lipidation on beta-amyloid aggregation and cytotoxicity (Q40095201) (← links)
- Bcl-2 induces pro-oxidant state by engaging mitochondrial respiration in tumor cells (Q40131176) (← links)
- Enzyme-linked immunosorbent assay for 4-hydroxynonenal-histidine conjugates (Q40224964) (← links)
- Intrathecal upregulation of granulocyte colony stimulating factor and its neuroprotective actions on motor neurons in amyotrophic lateral sclerosis (Q40245798) (← links)
- Bcl-2 attenuation of oxidative cell death is associated with up-regulation of gamma-glutamylcysteine ligase via constitutive NF-kappaB activation (Q40543251) (← links)
- Proteasomal dysfunction induced by 4-hydroxy-2,3-trans-nonenal, an end-product of lipid peroxidation: a mechanism contributing to neurodegeneration? (Q42527701) (← links)
- Oxidative inactivation of calcineurin by Cu,Zn superoxide dismutase G93A, a mutant typical of familial amyotrophic lateral sclerosis. (Q43794664) (← links)
- High molecular weight neurofilament proteins are physiological substrates of adduction by the lipid peroxidation product hydroxynonenal (Q43815833) (← links)
- Effect of wild-type or mutant Parkin on oxidative damage, nitric oxide, antioxidant defenses, and the proteasome (Q44009269) (← links)
- Selected contribution: Lung hypoxia: antioxidant and antiapoptotic effects of liposomal alpha-tocopherol (Q44138667) (← links)
- Increased oxidative damage to DNA in an animal model of amyotrophic lateral sclerosis (Q46607725) (← links)
- An RNAi strategy for treatment of amyotrophic lateral sclerosis caused by mutant Cu,Zn superoxide dismutase (Q50779982) (← links)
- Proteostasis Failure in Neurodegenerative Diseases: Focus on Oxidative Stress (Q92063917) (← links)