Pages that link to "Q42846777"

The following pages link to Yang Yang (Q42846777):

Displaying 28 items.

• Molecular basis and structural insight of vascular K(ATP) channel gating by S-glutathionylation. (Q30498793) (← links)
• K(ATP) channel action in vascular tone regulation: from genetics to diseases. (Q34044875) (← links)
• Oxidative Stress Inhibits Vascular KATP Channels by S-Glutathionylation (Q34352381) (← links)
• Network topology of NaV1.7 mutations in sodium channel-related painful disorders. (Q36290427) (← links)
• Prolonged exposure to methylglyoxal causes disruption of vascular KATP channel by mRNA instability (Q36382451) (← links)
• The Na(V)1.7 sodium channel: from molecule to man. (Q38066723) (← links)
• S-glutathionylation of ion channels: insights into the regulation of channel functions, thiol modification crosstalk, and mechanosensing (Q38120049) (← links)
• Rosiglitazone inhibits vascular KATP channels and coronary vasodilation produced by isoprenaline. (Q39524834) (← links)
• Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli. (Q39603773) (← links)
• Down-regulation of Stat3 induces apoptosis of human glioma cell: a potential method to treat brain cancer. (Q40090115) (← links)
• The Domain II S4-S5 Linker in Nav1.9: A Missense Mutation Enhances Activation, Impairs Fast Inactivation, and Produces Human Painful Neuropathy. (Q41190621) (← links)
• Acute exposure of methylglyoxal leads to activation of KATP channels expressed in HEK293 cells. (Q41502455) (← links)
• Structural modelling and mutant cycle analysis predict pharmacoresponsiveness of a Na(V)1.7 mutant channel. (Q42211742) (← links)
• Functional elements on SIRPalpha IgV domain mediate cell surface binding to CD47. (Q42846724) (← links)
• Multistate structural modeling and voltage-clamp analysis of epilepsy/autism mutation Kv10.2-R327H demonstrate the role of this residue in stabilizing the channel closed state. (Q43995088) (← links)
• Impairment of the Vascular KATP Channel Imposes Fatal Susceptibility to Experimental Diabetes Due to Multi-Organ Injuries. (Q46751044) (← links)
• Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons. (Q46987387) (← links)
• NaV1.7 as a Pharmacogenomic Target for Pain: Moving Toward Precision Medicine. (Q47645869) (← links)
• Reverse pharmacogenomics: carbamazepine normalizes activation and attenuates thermal hyperexcitability of sensory neurons due to Nav 1.7 mutation I234T. (Q47977892) (← links)
• Familial gain-of-function Nav1.9 mutation in a painful channelopathy. (Q48045901) (← links)
• Pharmacotherapy for Pain in a Family With Inherited Erythromelalgia Guided by Genomic Analysis and Functional Profiling. (Q48057846) (← links)
• Depolarized inactivation overcomes impaired activation to produce DRG neuron hyperexcitability in a Nav1.7 mutation in a patient with distal limb pain. (Q48215989) (← links)
• bFGF and heparin but not laminin are necessary factors in the mediums that affect NSCs differentiation into cholinergic neurons (Q48658159) (← links)
• Inhibition of AF116909 gene expression enhances the differentiation of neural stem cells (Q48834117) (← links)
• Nav1.8 expression is not restricted to nociceptors in mouse peripheral nervous system (Q84389261) (← links)
• S-Glutathionylation underscores the modulation of the heteromeric Kir4.1-Kir5.1 channel in oxidative stress (Q84829787) (← links)
• Resilience to Pain: A Peripheral Component Identified Using Induced Pluripotent Stem Cells and Dynamic Clamp (Q93249605) (← links)
• Wikidata:WikiProject Names/lists/people with a given name identical to their family name (← links | edit)