Pages that link to "Q40607491"
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The following pages link to O-GlcNAc modification is an endogenous inhibitor of the proteasome (Q40607491):
Displaying 50 items.
- Autoubiquitination of the 26S proteasome on Rpn13 regulates breakdown of ubiquitin conjugates (Q24297402) (← links)
- Proteasome function is regulated by cyclic AMP-dependent protein kinase through phosphorylation of Rpt6 (Q24310109) (← links)
- Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance (Q24315875) (← links)
- O-GlcNAcylation regulates EZH2 protein stability and function (Q24324113) (← links)
- O-GlcNAc modification of Sp1 inhibits the functional interaction between Sp1 and Oct1 (Q24324116) (← links)
- Crosstalk between O-GlcNAcylation and proteolytic cleavage regulates the host cell factor-1 maturation pathway (Q24338891) (← links)
- Exploring the O-GlcNAc proteome: direct identification of O-GlcNAc-modified proteins from the brain (Q24561943) (← links)
- The utility of ETD mass spectrometry in proteomic analysis (Q24682151) (← links)
- The life cycle of the 26S proteasome: from birth, through regulation and function, and onto its death (Q26738549) (← links)
- The role of posttranslational modifications in the assembly of stress granules (Q26825332) (← links)
- Three Decades of Research on O-GlcNAcylation - A Major Nutrient Sensor That Regulates Signaling, Transcription and Cellular Metabolism (Q26853027) (← links)
- Regulation of cardiac proteasomes by ubiquitination, SUMOylation, and beyond (Q26859368) (← links)
- The ubiquitin-proteasome system and cardiovascular disease (Q27021582) (← links)
- Chemical Arsenal for the Study of O-GlcNAc (Q27687493) (← links)
- Priming the proteasome by protein kinase G: a novel cardioprotective mechanism of sildenafil (Q28084015) (← links)
- Impact of Type 2 diabetes and aging on cardiomyocyte function and O-linked N-acetylglucosamine levels in the heart (Q28276371) (← links)
- The hexosamine signaling pathway: deciphering the "O-GlcNAc code" (Q28284365) (← links)
- Cycling of O-linked beta-N-acetylglucosamine on nucleocytoplasmic proteins (Q28299550) (← links)
- A mechanism of ubiquitin-independent proteasomal degradation of the tumor suppressors p53 and p73 (Q28305429) (← links)
- Regulation of proteasome activity in health and disease (Q28392708) (← links)
- Structure and mechanism of a bacterial beta-glucosaminidase having O-GlcNAcase activity (Q28492135) (← links)
- Distinct OGT-Binding Sites Promote HCF-1 Cleavage (Q28547471) (← links)
- Insulin dynamically regulates calmodulin gene expression by sequential o-glycosylation and phosphorylation of sp1 and its subcellular compartmentalization in liver cells (Q28566805) (← links)
- Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis (Q28568343) (← links)
- Ataxin-10 interacts with O-linked beta-N-acetylglucosamine transferase in the brain (Q28572487) (← links)
- Regulation of AMPA receptor trafficking by O-glycosylation (Q28578244) (← links)
- Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling (Q28581505) (← links)
- O-GlcNAcylation of master growth repressor DELLA by SECRET AGENT modulates multiple signaling pathways in Arabidopsis (Q30277743) (← links)
- Aberrant O-GlcNAcylation disrupts GNE enzyme activity in GNE myopathy (Q30315345) (← links)
- Glutathione depletion and acute exercise increase O-GlcNAc protein modification in rat skeletal muscle. (Q30368871) (← links)
- O-GlcNAc regulates FoxO activation in response to glucose (Q30439420) (← links)
- O-GlcNAc cycling mutants modulate proteotoxicity in Caenorhabditis elegans models of human neurodegenerative diseases (Q30528027) (← links)
- Prioritizing genes for X-linked diseases using population exome data (Q30850994) (← links)
- O-Glycosylation Regulates Ubiquitination and Degradation of the Anti-Inflammatory Protein A20 to Accelerate Atherosclerosis in Diabetic ApoE-Null Mice (Q30993051) (← links)
- Modification of p53 with O-linked N-acetylglucosamine regulates p53 activity and stability (Q33256999) (← links)
- Galectin-4 is involved in p27-mediated activation of the myelin basic protein promoter (Q33280834) (← links)
- O-GlcNAc modification of proteins affects volume regulation in Jurkat cells. (Q33521462) (← links)
- The hexosamine signaling pathway: O-GlcNAc cycling in feast or famine (Q33625053) (← links)
- O-linked beta-N-acetylglucosamine (O-GlcNAc): Extensive crosstalk with phosphorylation to regulate signaling and transcription in response to nutrients and stress (Q33625265) (← links)
- Identification of nitric oxide as an endogenous inhibitor of 26S proteasomes in vascular endothelial cells (Q33650608) (← links)
- Decreased O-linked GlcNAcylation protects from cytotoxicity mediated by huntingtin exon1 protein fragment (Q33676752) (← links)
- Hyperglycemia impairs proteasome function by methylglyoxal (Q33688582) (← links)
- Phosphatase UBLCP1 controls proteasome assembly (Q33747496) (← links)
- Glycopeptide-specific monoclonal antibodies suggest new roles for O-GlcNAc (Q33802435) (← links)
- Enrichment of O-GlcNAc modified proteins by the periodate oxidation-hydrazide resin capture approach (Q33835452) (← links)
- Phosphorylation of the 19S regulatory particle ATPase subunit, Rpt6, modifies susceptibility to proteotoxic stress and protein aggregation. (Q33853086) (← links)
- O-GlcNAcylation/phosphorylation cycling at Ser10 controls both transcriptional activity and stability of delta-lactoferrin (Q33911287) (← links)
- Proteasomal degradation of O-GlcNAc transferase elevates hypoxia-induced vascular endothelial inflammatory response†. (Q34048268) (← links)
- O-GlcNAc signaling in the cardiovascular system (Q34056858) (← links)
- Tyrosine nitration of PA700 links proteasome activation to endothelial dysfunction in mouse models with cardiovascular risk factors (Q34137316) (← links)