Pages that link to "Q40555067"
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The following pages link to Receptor interacting protein is ubiquitinated by cellular inhibitor of apoptosis proteins (c-IAP1 and c-IAP2) in vitro (Q40555067):
Displaying 50 items.
- Recruitment of the linear ubiquitin chain assembly complex stabilizes the TNF-R1 signaling complex and is required for TNF-mediated gene induction (Q22001513) (← links)
- Distinct BIR domains of cIAP1 mediate binding to and ubiquitination of tumor necrosis factor receptor-associated factor 2 and second mitochondrial activator of caspases (Q24294565) (← links)
- cIAP2 is a ubiquitin protein ligase for BCL10 and is dysregulated in mucosa-associated lymphoid tissue lymphomas (Q24300394) (← links)
- TWEAK induces apoptosis through a death-signaling complex comprising receptor-interacting protein 1 (RIP1), Fas-associated death domain (FADD), and caspase-8 (Q24300508) (← links)
- Cellular inhibitor of apoptosis protein-1 (cIAP1) can regulate E2F1 transcription factor-mediated control of cyclin transcription (Q24306110) (← links)
- cIAP1/2 are direct E3 ligases conjugating diverse types of ubiquitin chains to receptor interacting proteins kinases 1 to 4 (RIP1-4) (Q24338276) (← links)
- c-IAP1 and c-IAP2 are critical mediators of tumor necrosis factor alpha (TNFalpha)-induced NF-kappaB activation (Q24607621) (← links)
- Poly-ubiquitination in TNFR1-mediated necroptosis (Q26749315) (← links)
- Targeting Non-proteolytic Protein Ubiquitination for the Treatment of Diffuse Large B Cell Lymphoma (Q28115962) (← links)
- RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition (Q28295435) (← links)
- cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production (Q28591258) (← links)
- p42MAPK-mediated phosphorylation of xEIAP/XLX in Xenopus cytostatic factor-arrested egg extracts (Q33281715) (← links)
- Auto-ubiquitination-induced degradation of MALT1-API2 prevents BCL10 destabilization in t(11;18)(q21;q21)-positive MALT lymphoma (Q33417259) (← links)
- Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment (Q33589801) (← links)
- Necroptosis as an alternative form of programmed cell death (Q33788116) (← links)
- cIAP1-dependent TRAF2 degradation regulates the differentiation of monocytes into macrophages and their response to CD40 ligand (Q34184605) (← links)
- Alternative cell death mechanisms in development and beyond (Q34362382) (← links)
- LCL161 increases paclitaxel-induced apoptosis by degrading cIAP1 and cIAP2 in NSCLC. (Q34542571) (← links)
- RIP kinases: key decision makers in cell death and innate immunity (Q34924104) (← links)
- Modulation of pancreatic cancer chemoresistance by inhibition of TAK1 (Q35143975) (← links)
- Simultaneous induction of apoptotic and survival signaling pathways in macrophage-like THP-1 cells by Shiga toxin 1 (Q35689450) (← links)
- A novel role for RIP1 kinase in mediating TNFα production (Q36071998) (← links)
- TAK1-regulated expression of BIRC3 predicts resistance to preoperative chemoradiotherapy in oesophageal adenocarcinoma patients (Q36081375) (← links)
- IAPs, RINGs and ubiquitylation (Q36085601) (← links)
- Toll pathway modulates TNF-induced JNK-dependent cell death in Drosophila (Q36246981) (← links)
- The presence of HIV-1 Tat protein second exon delays fas protein-mediated apoptosis in CD4+ T lymphocytes: a potential mechanism for persistent viral production (Q36685165) (← links)
- TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha. (Q36749651) (← links)
- Genomic profiling reveals the potential role of TCL1A and MDR1 deficiency in chemotherapy-induced cardiotoxicity (Q36800903) (← links)
- Both cIAP1 and cIAP2 regulate TNFalpha-mediated NF-kappaB activation (Q36954601) (← links)
- IAP proteins as targets for drug development in oncology (Q37209606) (← links)
- Assembling the building blocks: structure and function of inhibitor of apoptosis proteins (Q37448134) (← links)
- The Inhibitor of Apoptosis (IAPs) in Adaptive Response to Cellular Stress (Q37512362) (← links)
- Regulation of TNFRSF and innate immune signalling complexes by TRAFs and cIAPs (Q37579634) (← links)
- Regulation of Apoptosis by Inhibitors of Apoptosis (IAPs) (Q37679203) (← links)
- Human enteroendocrine cell responses to infection with Chlamydia trachomatis: a microarray study (Q37844108) (← links)
- IAPS and ubiquitylation (Q37987881) (← links)
- NFκB and ubiquitination: partners in disarming RIPK1-mediated cell death (Q38000159) (← links)
- Striking a balance: autophagy, apoptosis, and necrosis in a normal and failing heart. (Q38045835) (← links)
- Stable incorporation of ATPase subunits into 19 S regulatory particle of human proteasome requires nucleotide binding and C-terminal tails (Q38466156) (← links)
- Cloaked in ubiquitin, a killer hides in plain sight: the molecular regulation of RIPK1. (Q38530635) (← links)
- Cell cycle-dependent expression of cIAP2 at G2/M phase contributes to survival during mitotic cell cycle arrest (Q38663042) (← links)
- Formation and removal of poly-ubiquitin chains in the regulation of tumor necrosis factor-induced gene activation and cell death (Q38691490) (← links)
- More to Life than NF-κB in TNFR1 Signaling (Q38899361) (← links)
- The Drosophila inhibitor of apoptosis protein DIAP2 functions in innate immunity and is essential to resist gram-negative bacterial infection (Q39125509) (← links)
- Negative Regulators of JAK/STAT Signaling in Rheumatoid Arthritis and Osteoarthritis (Q39154718) (← links)
- Modulation of RIP1 ubiquitylation and distribution by MeBS to sensitize cancer cells to tumor necrosis factor α‐induced apoptosis (Q39658109) (← links)
- Ubiquitin ligase MKRN1 modulates telomere length homeostasis through a proteolysis of hTERT. (Q42046068) (← links)
- Lymphotoxin α induces apoptosis, necroptosis and inflammatory signals with the same potency as tumour necrosis factor. (Q42152414) (← links)
- Translation of cIAP2 mRNA is mediated exclusively by a stress-modulated ribosome shunt (Q43077403) (← links)
- Inhibitor of apoptosis protein cIAP2 is essential for lipopolysaccharide-induced macrophage survival (Q43181903) (← links)