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The following pages link to Dynamic O-glycosylation of nuclear and cytosolic proteins: cloning and characterization of a neutral, cytosolic beta-N-acetylglucosaminidase from human brain (Q24290716):
Displaying 250 items.
- O-GlcNAcase (Q22676717) (← links)
- Requirement for O-linked N-acetylglucosaminyltransferase in lymphocytes activation (Q24294511) (← links)
- Quantitative regulation of nuclear pore complex proteins by O-GlcNAcylation (Q24295446) (← links)
- Tet proteins connect the O-linked N-acetylglucosamine transferase Ogt to chromatin in embryonic stem cells (Q24311695) (← links)
- Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance (Q24315875) (← links)
- Hexosamines, insulin resistance, and the complications of diabetes: current status (Q24538581) (← links)
- Mammalian cells contain a second nucleocytoplasmic hexosaminidase (Q24603531) (← links)
- O-GlcNAcylation and the Metabolic Shift in High-Proliferating Cells: All the Evidence Suggests that Sugars Dictate the Flux of Lipid Biogenesis in Tumor Processes (Q26770270) (← links)
- Three Decades of Research on O-GlcNAcylation - A Major Nutrient Sensor That Regulates Signaling, Transcription and Cellular Metabolism (Q26853027) (← links)
- Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology (Q27009399) (← links)
- A potent mechanism-inspired O-GlcNAcase inhibitor that blocks phosphorylation of tau in vivo (Q27650997) (← links)
- Chemical Dissection of the Link between Streptozotocin, O-GlcNAc, and Pancreatic Cell Death (Q27651749) (← links)
- Structural insights into mechanism and specificity of O-GlcNAc transferase (Q27652304) (← links)
- Elevation of Global O-GlcNAc Levels in 3T3-L1 Adipocytes by Selective Inhibition of O-GlcNAcase Does Not Induce Insulin Resistance (Q27652457) (← links)
- Insight into a strategy for attenuating AmpC-mediated β-lactam resistance: Structural basis for selective inhibition of the glycoside hydrolase NagZ (Q27655751) (← links)
- Screening-based discovery of drug-likeO-GlcNAcase inhibitor scaffolds (Q27658729) (← links)
- Substrate and product analogues as human O-GlcNAc transferase inhibitors (Q27663568) (← links)
- Inhibition of a bacterial O-GlcNAcase homologue by lactone and lactam derivatives: structural, kinetic and thermodynamic analyses (Q27663888) (← links)
- Inhibition of O-GlcNAcase Using a Potent and Cell-Permeable Inhibitor Does Not Induce Insulin Resistance in 3T3-L1 Adipocytes (Q27664569) (← links)
- Human OGA binds substrates in a conserved peptide recognition groove (Q27664652) (← links)
- Cell-Penetrant, Nanomolar O-GlcNAcase Inhibitors Selective against Lysosomal Hexosaminidases (Q27666021) (← links)
- Synergy of Peptide and Sugar in O-GlcNAcase Substrate Recognition (Q27677479) (← links)
- Structure of a bacterial putative acetyltransferase defines the fold of the human O-GlcNAcase C-terminal domain (Q27680132) (← links)
- Selective trihydroxyazepane NagZ inhibitors increase sensitivity of Pseudomonas aeruginosa to β-lactams (Q27687260) (← links)
- Chemical arsenal for the study of O-GlcNAc. (Q27687493) (← links)
- A critical perspective of the diverse roles of O-GlcNAc transferase in chromatin (Q28080440) (← links)
- Cell-specific phosphorylation of Zfhep transcription factor (Q28216933) (← links)
- The hexosamine signaling pathway: deciphering the "O-GlcNAc code" (Q28284365) (← links)
- Cycling of O-linked beta-N-acetylglucosamine on nucleocytoplasmic proteins (Q28299550) (← links)
- Structure and mechanism of a bacterial beta-glucosaminidase having O-GlcNAcase activity (Q28492135) (← links)
- Transcription factor Nrf1 is negatively regulated by its O-GlcNAcylation status (Q28507099) (← links)
- Developmental regulation of protein O-GlcNAcylation, O-GlcNAc transferase, and O-GlcNAcase in mammalian brain (Q28568251) (← links)
- Aging leads to increased levels of protein O-linked N-acetylglucosamine in heart, aorta, brain and skeletal muscle in Brown-Norway rats (Q28568308) (← links)
- Heat shock protein 60 modified with O-linked N-acetylglucosamine is involved in pancreatic beta-cell death under hyperglycemic conditions (Q28568544) (← links)
- Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc (Q28571555) (← links)
- Increased enzymatic O-GlcNAcylation of mitochondrial proteins impairs mitochondrial function in cardiac myocytes exposed to high glucose (Q28573520) (← links)
- Alloxan is an inhibitor of the enzyme O-linked N-acetylglucosamine transferase (Q28576836) (← links)
- Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition (Q28579197) (← links)
- Streptozotocin, an O-GlcNAcase inhibitor, blunts insulin and growth hormone secretion (Q28581656) (← links)
- Global identification and characterization of both O-GlcNAcylation and phosphorylation at the murine synapse (Q28594444) (← links)
- RNA Polymerase II C-Terminal Domain: Tethering Transcription to Transcript and Template (Q29540749) (← links)
- O-GlcNAcase: promiscuous hexosaminidase or key regulator of O-GlcNAc signaling? (Q30368002) (← links)
- Evidence for a Functional O-Linked N-Acetylglucosamine (O-GlcNAc) System in the Thermophilic Bacterium Thermobaculum terrenum (Q30380552) (← links)
- Regulation of calcium/calmodulin-dependent kinase IV by O-GlcNAc modification. (Q30490721) (← links)
- Metabolic cross-talk allows labeling of O-linked beta-N-acetylglucosamine-modified proteins via the N-acetylgalactosamine salvage pathway (Q30498243) (← links)
- O-GlcNAc modifications regulate cell survival and epiboly during zebrafish development (Q33433251) (← links)
- The hexosamine signaling pathway: O-GlcNAc cycling in feast or famine (Q33625053) (← links)
- O-linked beta-N-acetylglucosamine (O-GlcNAc): Extensive crosstalk with phosphorylation to regulate signaling and transcription in response to nutrients and stress (Q33625265) (← links)
- O-linked N-acetylglucosamine modification of insulin receptor substrate-1 occurs in close proximity to multiple SH2 domain binding motifs (Q33628891) (← links)
- Interaction of Nectarin 4 with a fungal protein triggers a microbial surveillance and defense mechanism in nectar (Q33726525) (← links)
- Synaptosome proteomics (Q33786664) (← links)
- Identification of O-GlcNAc sites within peptides of the Tau protein and their impact on phosphorylation (Q33822354) (← links)
- O-GlcNAc cycling enzymes associate with the translational machinery and modify core ribosomal proteins (Q33907013) (← links)
- A Caenorhabditis elegans model of insulin resistance: altered macronutrient storage and dauer formation in an OGT-1 knockout (Q33913856) (← links)
- O-GlcNAcylation regulates cancer metabolism and survival stress signaling via regulation of the HIF-1 pathway. (Q33923934) (← links)
- Increased expression of beta-N-acetylglucosaminidase in erythrocytes from individuals with pre-diabetes and diabetes (Q33930349) (← links)
- Elevated nucleocytoplasmic glycosylation by O-GlcNAc results in insulin resistance associated with defects in Akt activation in 3T3-L1 adipocytes (Q34024499) (← links)
- O-GlcNAc signaling in the cardiovascular system (Q34056858) (← links)
- Levels of human erythrocyte membrane-bound and cytosolic glycohydrolases are associated with oxidative stress in erectile dysfunction patients (Q34064803) (← links)
- Increased O-GlcNAc levels correlate with decreased O-GlcNAcase levels in Alzheimer disease brain (Q34073596) (← links)
- Altered glycan-dependent signaling induces insulin resistance and hyperleptinemia (Q34075585) (← links)
- Cytoplasmic O-glycosylation prevents cell surface transport of E-cadherin during apoptosis (Q34083597) (← links)
- Protein glycosylation: nature, distribution, enzymatic formation, and disease implications of glycopeptide bonds (Q34131204) (← links)
- Analysis of MGEA5 on 10q24.1-q24.3 encoding the beta-O-linked N-acetylglucosaminidase as a candidate gene for type 2 diabetes mellitus in Pima Indians (Q34152654) (← links)
- A deeply divergent phosphoglucomutase (PGM) of Giardia lamblia has both PGM and phosphomannomutase activities. (Q34169536) (← links)
- Roles of the Tetratricopeptide Repeat Domain in O-GlcNAc Transferase Targeting and Protein Substrate Specificity (Q34193954) (← links)
- O-GlcNAcylation determines the solubility, filament organization, and stability of keratins 8 and 18 (Q34232124) (← links)
- O-GlcNAcylation regulates phosphorylation of tau: a mechanism involved in Alzheimer's disease (Q34333004) (← links)
- Nucleocytoplasmic O-glycosylation: O-GlcNAc and functional proteomics (Q34346450) (← links)
- O-GlcNAcylation, contractile protein modifications and calcium affinity in skeletal muscle (Q34426366) (← links)
- Versatile O-GlcNAc transferase assay for high-throughput identification of enzyme variants, substrates, and inhibitors (Q34434068) (← links)
- Aberrant O-GlcNAcylated Proteins: New Perspectives in Breast and Colorectal Cancer (Q34488445) (← links)
- Disrupting the enzyme complex regulating O-GlcNAcylation blocks signaling and development (Q34498017) (← links)
- Structural insights into the mechanism and inhibition of eukaryotic O-GlcNAc hydrolysis (Q34502637) (← links)
- Pharmacological inhibition of O-GlcNAcase (OGA) prevents cognitive decline and amyloid plaque formation in bigenic tau/APP mutant mice. (Q34515333) (← links)
- Two O-linked N-acetylglucosamine transferase genes of Arabidopsis thaliana L. Heynh. have overlapping functions necessary for gamete and seed development (Q34615508) (← links)
- Multiple tissue-specific roles for the O-GlcNAc post-translational modification in the induction of and complications arising from type II diabetes (Q34683091) (← links)
- The emerging link between O-GlcNAc and Alzheimer disease. (Q34683108) (← links)
- Molecular characterization of nucleocytosolic O-GlcNAc transferases of Giardia lamblia and Cryptosporidium parvum (Q34864447) (← links)
- Intracellular glycosylation and development (Q34989577) (← links)
- Diverse regulation of protein function by O-GlcNAc: a nuclear and cytoplasmic carbohydrate post-translational modification (Q35019401) (← links)
- Regulation of mitochondrial morphology and function by O-GlcNAcylation in neonatal cardiac myocytes (Q35057168) (← links)
- O-GlcNAc: a regulatory post-translational modification. (Q35077018) (← links)
- Pleiotropic and age-dependent effects of decreased protein modification by O-linked N-acetylglucosamine on pancreatic β-cell function and vascularization. (Q35111129) (← links)
- A lipid-droplet-targeted O-GlcNAcase isoform is a key regulator of the proteasome (Q35140884) (← links)
- O-GlcNAc turns twenty: functional implications for post-translational modification of nuclear and cytosolic proteins with a sugar (Q35163991) (← links)
- Conditional knock-out reveals a requirement for O-linked N-Acetylglucosaminase (O-GlcNAcase) in metabolic homeostasis (Q35172669) (← links)
- Dynamic interplay between O-glycosylation and O-phosphorylation of nucleocytoplasmic proteins: a new paradigm for metabolic control of signal transduction and transcription (Q35185681) (← links)
- A chemical approach for identifying O-GlcNAc-modified proteins in cells (Q35242785) (← links)
- A little sugar goes a long way: the cell biology of O-GlcNAc (Q35261648) (← links)
- O-linked β-N-acetylglucosamine transferase directs cell proliferation in idiopathic pulmonary arterial hypertension. (Q35328516) (← links)
- Diabetes-associated dysregulation of O-GlcNAcylation in rat cardiac mitochondria (Q35616023) (← links)
- Chemical approaches to understanding O-GlcNAc glycosylation in the brain (Q35648339) (← links)
- The active site of O-GlcNAc transferase imposes constraints on substrate sequence (Q35735257) (← links)
- Cross talk between O-GlcNAcylation and phosphorylation: roles in signaling, transcription, and chronic disease (Q35804446) (← links)
- Pharmacological Inhibition of O-GlcNAcase Does Not Increase Sensitivity of Glucocorticoid Receptor-Mediated Transrepression (Q35869321) (← links)
- β-N-Acetylglucosamine (O-GlcNAc) is a novel regulator of mitosis-specific phosphorylations on histone H3 (Q35874479) (← links)
- NOS1AP O-GlcNAc Modification Involved in Neuron Apoptosis Induced by Excitotoxicity (Q35902679) (← links)
- Insights into O-linked N-acetylglucosamine ([0-9]O-GlcNAc) processing and dynamics through kinetic analysis of O-GlcNAc transferase and O-GlcNAcase activity on protein substrates. (Q35939748) (← links)
- O-GlcNAc modification of transcription factor Sp1 mediates hyperglycemia-induced VEGF-A upregulation in retinal cells (Q35970441) (← links)
- The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease (Q35994096) (← links)
- Quantitative phosphoproteomics reveals crosstalk between phosphorylation and O-GlcNAc in the DNA damage response pathway (Q36044755) (← links)
- Enhanced transfer of a photocross-linking N-acetylglucosamine (GlcNAc) analog by an O-GlcNAc transferase mutant with converted substrate specificity (Q36049976) (← links)
- Evidence of the involvement of O-GlcNAc-modified human RNA polymerase II CTD in transcription in vitro and in vivo (Q36078777) (← links)
- O-GlcNAc transferase and O-GlcNAcase: achieving target substrate specificity (Q36096719) (← links)
- Does O-GlcNAc play a role in neurodegenerative diseases? (Q36126486) (← links)
- Modulation of dynamin-related protein 1 (DRP1) function by increased O-linked-β-N-acetylglucosamine modification (O-GlcNAc) in cardiac myocytes (Q36215833) (← links)
- Ogt-dependent X-chromosome-linked protein glycosylation is a requisite modification in somatic cell function and embryo viability (Q36233025) (← links)
- Post-translational O-GlcNAcylation is essential for nuclear pore integrity and maintenance of the pore selectivity filter (Q36455332) (← links)
- Transcriptome responses in alfalfa associated with tolerance to intensive animal grazing. (Q36499623) (← links)
- Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system (Q36592808) (← links)
- Reduction of O-GlcNAc protein modification does not prevent insulin resistance in 3T3-L1 adipocytes (Q36631327) (← links)
- High glucose and diabetes modulate cellular proteasome function: Implications in the pathogenesis of diabetes complications (Q36677830) (← links)
- O-linked N-acetylglucosamine transferase (OGT) is overexpressed and promotes O-linked protein glycosylation in esophageal squamous cell carcinoma (Q36682124) (← links)
- Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury (Q36807962) (← links)
- Downregulation of O-linked N-acetylglucosamine transferase by RNA interference decreases MMP9 expression in human esophageal cancer cells. (Q36825392) (← links)
- O-GlcNAcylation of protein kinase A catalytic subunits enhances its activity: a mechanism linked to learning and memory deficits in Alzheimer's disease (Q36863783) (← links)
- Cross-talk between GlcNAcylation and phosphorylation: site-specific phosphorylation dynamics in response to globally elevated O-GlcNAc (Q36893523) (← links)
- A mitotic GlcNAcylation/phosphorylation signaling complex alters the posttranslational state of the cytoskeletal protein vimentin (Q36914304) (← links)
- Interplay between Metabolism and Epigenetics: A Nuclear Adaptation to Environmental Changes (Q36969070) (← links)
- NFkappaB activation is associated with its O-GlcNAcylation state under hyperglycemic conditions (Q36970951) (← links)
- Murine platelets are not regulated by O-linked beta-N-acetylglucosamine (Q36984274) (← links)
- Glycan antagonists and inhibitors: a fount for drug discovery (Q37027396) (← links)
- Metabolic control of proteasome function (Q37029653) (← links)
- Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system. (Q37088282) (← links)
- Dynamic O-GlcNAcylation and its roles in the cellular stress response and homeostasis. (Q37099485) (← links)
- Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity (Q37114599) (← links)
- The O-linked N-acetylglucosamine modification in cellular signalling and the immune system. 'Protein modifications: beyond the usual suspects' review series (Q37212757) (← links)
- Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation (Q37241211) (← links)
- Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury (Q37267216) (← links)
- HINCUTs in cancer: hypoxia-induced noncoding ultraconserved transcripts. (Q37299459) (← links)
- Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts (Q37330282) (← links)
- O-linked N-acetylglucosamine modification on CCAAT enhancer-binding protein beta: role during adipocyte differentiation (Q37339267) (← links)
- Differential hexosamine biosynthetic pathway gene expression with type 2 diabetes. (Q37434092) (← links)
- O-GlcNAcomics--Revealing roles of O-GlcNAcylation in disease mechanisms and development of potential diagnostics (Q37445785) (← links)
- The intersections between O-GlcNAcylation and phosphorylation: implications for multiple signaling pathways (Q37479201) (← links)
- O-GlcNAcylation regulates breast cancer metastasis via SIRT1 modulation of FOXM1 pathway. (Q37538368) (← links)
- Hexosamine flux, the O-GlcNAc modification, and the development of insulin resistance in adipocytes (Q37607447) (← links)
- O-GlcNAc and the cardiovascular system. (Q37619691) (← links)
- O-linked beta-N-acetylglucosaminylation in mouse embryonic neural precursor cells (Q37641874) (← links)
- Nutrient-driven O-GlcNAc cycling - think globally but act locally (Q37732989) (← links)
- Site-specific interplay between O-GlcNAcylation and phosphorylation in cellular regulation (Q37736782) (← links)
- Protein O-linked β-N-acetylglucosamine: a novel effector of cardiomyocyte metabolism and function (Q37924015) (← links)
- The role of intracellular protein O-glycosylation in cell adhesion and disease (Q38095924) (← links)
- Hepatic glucose sensing and integrative pathways in the liver (Q38160330) (← links)
- The effect of peri-conception hyperglycaemia and the involvement of the hexosamine biosynthesis pathway in mediating oocyte and embryo developmental competence. (Q38177732) (← links)
- Interpreting the language of histone and DNA modifications (Q38196193) (← links)
- Mechanisms of CaMKII Activation in the Heart (Q38207020) (← links)
- Chemical tools to explore nutrient-driven O-GlcNAc cycling (Q38231368) (← links)
- Regulation of autophagy by protein post-translational modification (Q38264531) (← links)
- The Role of the O-GlcNAc Modification in Regulating Eukaryotic Gene Expression (Q38284902) (← links)
- The transcription factor PDX-1 is post-translationally modified by O-linked N-acetylglucosamine and this modification is correlated with its DNA binding activity and insulin secretion in min6 beta-cells. (Q38353199) (← links)
- Functional analysis of recombinant human and Yarrowia lipolytica O-GlcNAc transferases expressed in Saccharomyces cerevisiae. (Q38410278) (← links)
- The Utilities of Chemical Reactions and Molecular Tools for O-GlcNAc Proteomic Studies (Q38533861) (← links)
- New insights: A role for O-GlcNAcylation in diabetic complications (Q38710250) (← links)
- Identification and characterization of a missense mutation in the O-linked β-N-acetylglucosamine (O-GlcNAc) transferase gene that segregates with X-linked intellectual disability (Q38711921) (← links)
- Fatty acid synthase inhibits the O-GlcNAcase during oxidative stress (Q38715120) (← links)
- Fuel availability and fate in cardiac metabolism: A tale of two substrates (Q38781072) (← links)
- Role of O-linked N-acetylglucosamine modification in diabetic nephropathy. (Q38782341) (← links)
- Potential coordination role between O-GlcNAcylation and epigenetics (Q38794897) (← links)
- Protein-Specific Imaging of O-GlcNAcylation in Single Cells. (Q38826163) (← links)
- O-GlcNAcylation in Cancer Biology: Linking Metabolism and Signaling (Q38876699) (← links)
- O-GlcNAcylation and neurodegeneration (Q38920683) (← links)
- Enzymatic properties and subtle differences in the substrate specificity of phylogenetically distinct invertebrate N-glycan processing hexosaminidases. (Q38930713) (← links)
- Therapeutic Targeting of Epithelial Plasticity Programs: Focus on the Epithelial-Mesenchymal Transition. (Q38953751) (← links)
- Stress-induced O-GlcNAcylation: an adaptive process of injured cells. (Q38959360) (← links)
- SRT1720 counteracts glucosamine-induced endoplasmic reticulum stress and endothelial dysfunction (Q38999901) (← links)
- In Vitro Biochemical Assays for O-GlcNAc-Processing Enzymes (Q39285550) (← links)
- The emerging link between O-GlcNAcylation and neurological disorders. (Q39324009) (← links)
- O-Linked β-N-acetylglucosamine (O-GlcNAc) Regulates Stress-induced Heat Shock Protein Expression in a GSK-3β-dependent Manner (Q39647598) (← links)
- O-GlcNAc-glycosylation of beta-catenin regulates its nuclear localization and transcriptional activity (Q39967364) (← links)
- Mechanism, Structure, and Inhibition of O-GlcNAc Processing Enzymes (Q40025244) (← links)
- Distributive O-GlcNAcylation on the Highly Repetitive C-Terminal Domain of RNA Polymerase II. (Q40065322) (← links)
- Stabilization of plakoglobin and enhanced keratinocyte cell-cell adhesion by intracellular O-glycosylation (Q40309909) (← links)
- The Early Metazoan Trichoplax adhaerens Possesses a Functional O-GlcNAc System (Q40431483) (← links)
- O-GlcNAc modification is an endogenous inhibitor of the proteasome (Q40607491) (← links)
- The coactivator of transcription CREB-binding protein interacts preferentially with the glycosylated form of Stat5. (Q40620228) (← links)
- Down-regulation of Sp1 activity through modulation of O-glycosylation by treatment with a low glucose mimetic, 2-deoxyglucose. (Q40628182) (← links)
- Dynamic interplay between O-glycosylation and O-phosphorylation of nucleocytoplasmic proteins: alternative glycosylation/phosphorylation of THR-58, a known mutational hot spot of c-Myc in lymphomas, is regulated by mitogens (Q40743661) (← links)
- Protein O-GlcNAcylation: A critical regulator of the cellular response to stress. (Q40794510) (← links)
- O-GlcNAc regulation of autophagy and α-synuclein homeostasis; implications for Parkinson's disease. (Q41064100) (← links)
- Detecting O-GlcNAc using in vitro sulfation (Q41189292) (← links)
- O-GlcNAcylation of co-activator-associated arginine methyltransferase 1 regulates its protein substrate specificity (Q41587950) (← links)
- Structural and functional insight into human O-GlcNAcase (Q41686648) (← links)
- Structural insights into the substrate binding adaptability and specificity of human O-GlcNAcase (Q41689766) (← links)
- Mechanism of Human Nucleocytoplasmic Hexosaminidase D. (Q41816155) (← links)
- Systematic analysis of barrier-forming FG hydrogels from Xenopus nuclear pore complexes (Q41828480) (← links)
- O-Linked N-acetylglucosamine transferase 1 regulates global histone H4 acetylation via stabilization of the nonspecific lethal protein NSL3. (Q41987024) (← links)
- Difficulties encountered during glycopeptide syntheses (Q42003328) (← links)
- O-GlcNAcylation of TAB1 modulates TAK1-mediated cytokine release. (Q42007526) (← links)
- Characterization of beta-N-acetylglucosaminidase cleavage by caspase-3 during apoptosis (Q42117656) (← links)
- Detection and analysis of proteins modified by O-linked N-acetylglucosamine (Q42157350) (← links)
- O-GlcNAc Transferase Is Essential for Sensory Neuron Survival and Maintenance (Q42318087) (← links)
- In vivo modulation of O-GlcNAc levels regulates hippocampal synaptic plasticity through interplay with phosphorylation. (Q42444776) (← links)
- O-GLcNAc post-translational modifications regulate the entry of neurons into an axon branching program (Q42560281) (← links)
- Cysteine S-linked N-acetylglucosamine (S-GlcNAcylation), A New Post-translational Modification in Mammals (Q42687789) (← links)
- Quantitative time-resolved chemoproteomics reveals that stable O-GlcNAc regulates box C/D snoRNP biogenesis (Q42798614) (← links)
- Perturbations in O-linked beta-N-acetylglucosamine protein modification cause severe defects in mitotic progression and cytokinesis (Q42812161) (← links)
- Mapping O-GlcNAc modification sites on tau and generation of a site-specific O-GlcNAc tau antibody (Q43713127) (← links)
- O-GlcNAc expression in developing and ageing mouse brain (Q43719643) (← links)
- Identification of GlcNAcylation sites of peptides and alpha-crystallin using Q-TOF mass spectrometry (Q43769874) (← links)
- O-GlcNAcylation of 8-Oxoguanine DNA Glycosylase (Ogg1) Impairs Oxidative Mitochondrial DNA Lesion Repair in Diabetic Hearts (Q43873009) (← links)
- Prediction of structure and functional residues for O-GlcNAcase, a divergent homologue of acetyltransferases (Q44174039) (← links)
- High glucose and insulin promote O-GlcNAc modification of proteins, including alpha-tubulin (Q44191960) (← links)
- Characterization of the O-GlcNAc protein modification in Xenopus laevis oocyte during oogenesis and progesterone-stimulated maturation (Q44193512) (← links)
- Localization of the O-GlcNAc transferase and O-GlcNAc-modified proteins in rat cerebellar cortex (Q44346570) (← links)
- Identification of novel sites of O-N-acetylglucosamine modification of serum response factor using quadrupole time-of-flight mass spectrometry. (Q44396316) (← links)
- O-glycosylation of Sp1 and transcriptional regulation of the calmodulin gene by insulin and glucagon (Q44538187) (← links)
- Unique in vivo modifications of coagulation factor V produce a physically and functionally distinct platelet-derived cofactor: characterization of purified platelet-derived factor V/Va (Q44640434) (← links)
- Human Alzheimer's disease synaptic O-GlcNAc site mapping and iTRAQ expression proteomics with ion trap mass spectrometry (Q44833697) (← links)
- Neutrophils exhibit rapid agonist-induced increases in protein-associated O-GlcNAc (Q45025090) (← links)
- O-GlcNAcylation of SIRT1 enhances its deacetylase activity and promotes cytoprotection under stress. (Q45940207) (← links)
- Proteomic analysis and abrogated expression of O-GlcNAcylated proteins associated with primary breast cancer. (Q45951647) (← links)
- Analysis of mammalian O-glycopeptides - we have made a good start, but there is a long way to go. (Q46846034) (← links)
- Regulation of the inositol 1,4,5-trisphosphate receptor type I by O-GlcNAc glycosylation. (Q46852197) (← links)
- O-GlcNAc on PKCζ Inhibits the FGF4-PKCζ-MEK-ERK1/2 Pathway via Inhibition of PKCζ Phosphorylation in Mouse Embryonic Stem Cells (Q47241925) (← links)
- Enzymatic properties of β-N-acetylglucosaminidases. (Q47343121) (← links)
- Nutrient-driven O-GlcNAc in proteostasis and neurodegeneration (Q47628534) (← links)
- Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats (Q47705876) (← links)
- O-GlcNAc transferase regulates transcriptional activity of human Oct4. (Q47784492) (← links)
- Production of O-GlcNAc Modified Recombinant Tau in E. coli and Detection of Ser400 O-GlcNAc Tau In Vivo (Q47852182) (← links)
- A Conserved Splicing Silencer Dynamically Regulates O-GlcNAc Transferase Intron Retention and O-GlcNAc Homeostasis (Q47999442) (← links)
- Structures of human O-GlcNAcase and its complexes reveal a new substrate recognition mode. (Q48103560) (← links)
- O-GlcNAc occurs cotranslationally to stabilize nascent polypeptide chains (Q48216207) (← links)
- Thiamet G mediates neuroprotection in experimental stroke by modulating microglia/macrophage polarization and inhibiting NF-κB p65 signaling. (Q48423896) (← links)
- O-Linked β-N-acetylglucosamine (O-GlcNAc) modification: a new pathway to decode pathogenesis of diabetic retinopathy (Q49911705) (← links)
- O-GlcNAcylation: key regulator of glycolytic pathways (Q49980779) (← links)
- Characterization of tools to detect and enrich human and mouse O-GlcNAcase. (Q50953375) (← links)
- Insights into activity and inhibition from the crystal structure of human O-GlcNAcase. (Q51084028) (← links)
- High extent of O-GlcNAcylation in breast cancer cells correlates with the levels of HAS enzymes, accumulation of hyaluronan, and poor outcome. (Q51450789) (← links)
- AANL (Agrocybe aegerita lectin 2) is a new facile tool to probe for O-GlcNAcylation. (Q52644748) (← links)
- Dissecting PUGNAc-mediated inhibition of the pro-survival action of insulin. (Q53144959) (← links)
- Increasing O-GlcNAc slows neurodegeneration and stabilizes tau against aggregation. (Q53434499) (← links)
- [Protein O-GlcNAcylation and regulation of cell signalling: involvement in pathophysiology]. (Q53468556) (← links)
- Changes in Protein O-GlcNAcylation During Mouse Epididymal Sperm Maturation. (Q55266020) (← links)
- O-GlcNAc cycling in the developing, adult and geriatric brain. (Q55416370) (← links)
- Xpo7 is a broad-spectrum exportin and a nuclear import receptor. (Q55517575) (← links)
- Computational evidence for the substrate-assisted catalytic mechanism of O-GlcNAcase. A DFT investigation (Q56985730) (← links)
- AMP-Activated Protein Kinase and O-GlcNAcylation, Two Partners Tightly Connected to Regulate Key Cellular Processes (Q57110459) (← links)
- Proteomic analysis of the OGT interactome: novel links to epithelial-mesenchymal transition and metastasis of cervical cancer (Q57293808) (← links)
- -GlcNAc Modification During Pregnancy: Focus on Placental Environment (Q57300455) (← links)
- O-GlcNAc transferase associates with the MCM2-7 complex and its silencing destabilizes MCM-MCM interactions (Q57752771) (← links)
- Analysis of transition state mimicry by tight binding aminothiazoline inhibitors provides insight into catalysis by human -GlcNAcase (Q57810292) (← links)
- Thioglycosides Are Efficient Metabolic Decoys of Glycosylation that Reduce Selectin Dependent Leukocyte Adhesion (Q57815295) (← links)
- Critical observations that shaped our understanding of the function(s) of Intracellular Glycosylation (O-GlcNAc) (Q58564337) (← links)
- '-GlcNAc Code' Mediated Biological Functions of Downstream Proteins (Q58692776) (← links)
- Chemical Reporters and Their Bioorthogonal Reactions for Labeling Protein -GlcNAcylation (Q58693028) (← links)
- O-GlcNAc as an Integrator of Signaling Pathways (Q59125594) (← links)
- Too sweet to resist: Control of immune cell function by O-GlcNAcylation (Q59906659) (← links)
- Phospho-GlcNAc modulation of slow MLC2 during soleus atrophy through a multienzymatic and sarcomeric complex (Q60403346) (← links)