Sustained regression of tumors upon MYC inactivation requires p53 or thrombospondin-1 to reverse the angiogenic switch. (Q35133764)

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scholarly article

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English	Sustained regression of tumors upon MYC inactivation requires p53 or thrombospondin-1 to reverse the angiogenic switch.	scholarly article

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scholarly article

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21 December 2019

Sustained regression of tumors upon MYC inactivation requires p53 or thrombospondin-1 to reverse the angiogenic switch (English)

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21 December 2019

Sylvie Giuriato

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21 December 2019

Jan van Riggelen

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Jan van Riggelen

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21 December 2019

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Judah Folkman

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21 December 2019

Emmanuelle Passegué

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Emmanuelle Passegué

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21 December 2019

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Sandra Ryeom

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21 December 2019

Alice C Fan

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21 December 2019

Pavan Bachireddy

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21 December 2019

Ryan C Lynch

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21 December 2019

Matthew J Rioth

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21 December 2019

Andrew M Kopelman

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21 December 2019

Flora Tang

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21 December 2019

Dean W Felsher

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21 December 2019

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20 October 2006

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21 December 2019

Proceedings of the National Academy of Sciences of the United States of America

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21 December 2019

103

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21 December 2019

44

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21 December 2019

16266-16271

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21 December 2019

Loss of p53 impedes the antileukemic response to BCR-ABL inhibition

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Function of endogenous inhibitors of angiogenesis as endothelium-specific tumor suppressors

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Oncogene addiction: sometimes a temporary slavery

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MYC inactivation uncovers pluripotent differentiation and tumour dormancy in hepatocellular cancer

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Ras modulates Myc activity to repress thrombospondin-1 expression and increase tumor angiogenesis

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Impact of p53 loss on reversal and recurrence of conditional Wnt-induced tumorigenesis

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Role of angiogenesis in tumor growth and metastasis

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Rules for making human tumor cells

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Cancer. Addiction to oncogenes--the Achilles heal of cancer

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Thrombospondin-1 as an endogenous inhibitor of angiogenesis and tumor growth.

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c-MYC induces mammary tumorigenesis by means of a preferred pathway involving spontaneous Kras2 mutations

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Activation of the myc oncoprotein leads to increased turnover of thrombospondin-1 mRNA

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Disruption of the ARF-Mdm2-p53 tumor suppressor pathway in Myc-induced lymphomagenesis

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Reversible tumorigenesis by MYC in hematopoietic lineages

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The p53 tumor suppressor gene inhibits angiogenesis by stimulating the production of thrombospondin.

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Sustained loss of a neoplastic phenotype by brief inactivation of MYC

1 reference

https://pubmed.ncbi.nlm.nih.gov/17056717

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Angiogenesis is an early event in the generation of myc-induced lymphomas

1 reference

https://pubmed.ncbi.nlm.nih.gov/17056717

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Genomically complex lymphomas undergo sustained tumor regression upon MYC inactivation unless they acquire novel chromosomal translocations

1 reference

https://pubmed.ncbi.nlm.nih.gov/17056717

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Synergy between a human c-myc transgene and p53 null genotype in murine thymic lymphomas: contrasting effects of homozygous and heterozygous p53 loss

1 reference

https://pubmed.ncbi.nlm.nih.gov/17056717

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1073/PNAS.0608017103

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17056717%20AND%20SRC:MED&resulttype=core&format=json

21 December 2019

2006PNAS..10316266G

0 references

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1 reference

Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17056717%20AND%20SRC:MED&resulttype=core&format=json

21 December 2019

PubMed publication ID

1 reference

Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17056717%20AND%20SRC:MED&resulttype=core&format=json

21 December 2019

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