[go: up one dir, main page]

The role of cytokines in the pathogenesis of hepatic granulomatous disease in Schistosoma mansoni infected mice

Mem Inst Oswaldo Cruz. 1992:87 Suppl 4:81-5. doi: 10.1590/s0074-02761992000800011.

Abstract

Cytokines are important in the cell-mediated response to Schistosoma mansoni eggs. We have found that Th2 cytokine responses (e.g. IL-4 and IL-5) are augmented after egg laying begins while Th1 responses (IL-2 and IFN-gamma) are down regulated in S. mansoni infected mice. Treatment of mice with anti-IL-5 monoclonal antibodies (Mab) suppressed the eosinophil response almost completely but did not affect granuloma size and slightly increased hepatic fibrosis. Anti-IL-4 treatment abolished IgE responses in infected mice and decreased hepatic fibrosis slightly. Anti-IFN-gamma treatment had no effect on hepatic pathology. Anti-IL-2 treatment decreased granuloma size significantly and decreased hepatic fibrosis markedly. Anti-IL-2 treatment dramatically decreased IL-5 secretion by splenic cells in vitro and decreased peripheral blood and tissue eosinophilia. In contrast IL-4 secretion was unaffected and serum IgE was normal or increased. IL-2 and IFN-gamma secretion by splenic cells of treated mice were slightly but not significantly increased suggesting that anti-IL-2 treatment is affecting Th2 rather than Th1 responses.

MeSH terms

  • Animals
  • Cells, Cultured
  • Cytokines / antagonists & inhibitors
  • Cytokines / physiology*
  • Female
  • Fibrosis
  • Granuloma / parasitology*
  • Granuloma / physiopathology
  • Interferon-gamma / antagonists & inhibitors
  • Interferon-gamma / biosynthesis
  • Interleukins / antagonists & inhibitors
  • Interleukins / biosynthesis
  • Liver Diseases, Parasitic / parasitology*
  • Liver Diseases, Parasitic / pathology
  • Mice
  • Receptors, Interleukin-2 / antagonists & inhibitors
  • Schistosomiasis mansoni / physiopathology*
  • Spleen / pathology
  • T-Lymphocytes, Helper-Inducer / immunology

Substances

  • Cytokines
  • Interleukins
  • Receptors, Interleukin-2
  • Interferon-gamma