Ubiquitin-dependent degradation of SnoN and Ski is increased in renal fibrosis induced by obstructive injury (Q83177771)

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19 January 2020

title

Ubiquitin-dependent degradation of SnoN and Ski is increased in renal fibrosis induced by obstructive injury (English)

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19 January 2020

1 reference

Fukasawa H

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19 January 2020

Yamamoto T

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19 January 2020

Togawa A

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19 January 2020

Ohashi N

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19 January 2020

Fujigaki Y

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19 January 2020

Oda T

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19 January 2020

Uchida C

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19 January 2020

Kitagawa K

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19 January 2020

Hattori T

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19 January 2020

Suzuki S

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19 January 2020

Kitagawa M

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19 January 2020

Hishida A

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19 January 2020

language of work or name

English

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publication date

1 May 2006

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19 January 2020

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19 January 2020

volume

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19 January 2020

issue

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19 January 2020

page(s)

1733-1740

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Transforming growth factor-beta: recent progress and new challenges

19 January 2020

cites work

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Transforming growth factor beta in tissue fibrosis

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Regulation of Smad activity

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Increased expression of c-Ski as a co-repressor in transforming growth factor-beta signaling correlates with progression of esophageal squamous cell carcinoma

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7 January 2021

Downregulation of Smad transcriptional corepressors SnoN and Ski in the fibrotic kidney: an amplification mechanism for TGF-beta1 signaling

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7 January 2021

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Ubiquitin-mediated proteolysis: biological regulation via destruction

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TGF-beta induces assembly of a Smad2-Smurf2 ubiquitin ligase complex that targets SnoN for degradation

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7 January 2021

Down-regulation of Smad7 expression by ubiquitin-dependent degradation contributes to renal fibrosis in obstructive nephropathy in mice

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7 January 2021

Obstructive nephropathy and renal fibrosis

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Ski/Sno and TGF-beta signaling.

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Ski and SnoN: negative regulators of TGF-beta signaling

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Isolation of human cDNA clones of ski and the ski-related gene, sno

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7 January 2021

Proto-oncogene Sno expression, alternative isoforms and immediate early serum response

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Ski is a component of the histone deacetylase complex required for transcriptional repression by Mad and thyroid hormone receptor

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Negative feedback regulation of TGF-beta signaling by the SnoN oncoprotein

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The Ski oncoprotein interacts with the Smad proteins to repress TGFbeta signaling

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7 January 2021

SnoN and Ski protooncoproteins are rapidly degraded in response to transforming growth factor beta signaling

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7 January 2021

Regulation of Smad degradation and activity by Smurf2, an E3 ubiquitin ligase

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7 January 2021

Smad7 binds to Smurf2 to form an E3 ubiquitin ligase that targets the TGF beta receptor for degradation

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7 January 2021

Impaired Smad7-Smurf-mediated negative regulation of TGF-beta signaling in scleroderma fibroblasts

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7 January 2021

Transcriptional induction of Smurf2 ubiquitin ligase by TGF-beta

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7 January 2021

Transforming growth factor-beta receptors in self-limited vs. chronic progressive nephritis in rats.

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Treatment with anti-TGF-beta antibody ameliorates chronic progressive nephritis by inhibiting Smad/TGF-beta signaling

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7 January 2021

Ubiquitin-Dependent Degradation of Smad2 Is Increased in the Glomeruli of Rats with Anti-Thymocyte Serum Nephritis

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7 January 2021

An F-box protein, FWD1, mediates ubiquitin-dependent proteolysis of beta-catenin

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7 January 2021