Characterization of p73 functional domains necessary for transactivation and growth suppression (Q73646221)

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5 November 2019

title

Characterization of p73 functional domains necessary for transactivation and growth suppression (English)

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5 November 2019

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5 November 2019

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5 November 2019

Susan Nozell

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12853970%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

Yijun Wu

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12853970%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

Kelly McNaughton

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5 November 2019

Amy Willis

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5 November 2019

Xinbin Chen

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5 November 2019

language of work or name

English

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publication date

1 July 2003

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5 November 2019

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5 November 2019

volume

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5 November 2019

issue

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5 November 2019

page(s)

4333-4347

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Interaction of c-Abl and p73alpha and their collaboration to induce apoptosis

5 November 2019

cites work

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The proline-rich domain of p53 is required for cooperation with anti-neoplastic agents to promote apoptosis of tumor cells

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Induction of the growth inhibitor IGF-binding protein 3 by p53

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Two tandem and independent sub-activation domains in the amino terminus of p53 require the adaptor complex for activity

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7 January 2021

p73 is transcriptionally regulated by DNA damage, p53, and p73

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p73 and p63 are homotetramers capable of weak heterotypic interactions with each other but not with p53

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7 January 2021

Two new p73 splice variants, gamma and delta, with different transcriptional activity.

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7 January 2021

Comparative analysis of p73 and p53 regulation and effector functions

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Transactivation-deficient p73alpha (p73Deltaexon2) inhibits apoptosis and competes with p53.

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7 January 2021

The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

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Nuclear import and export signals in control of the p53-related protein p73.

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p73 is a simian [correction of human] p53-related protein that can induce apoptosis

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Monoallelically Expressed Gene Related to p53 at 1p36, a Region Frequently Deleted in Neuroblastoma and Other Human Cancers

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p73: Friend or foe in tumorigenesis

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Analysis of p73 in human borderline and invasive ovarian tumor

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p21B, a variant of p21(Waf1/Cip1), is induced by the p53 family

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7 January 2021

An anti-apoptotic role for the p53 family member, p73, during developmental neuron death

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7 January 2021

A proline-rich motif in p53 is required for transactivation-independent growth arrest as induced by Gas1.

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7 January 2021

The polyproline region of p53 is required to activate apoptosis but not growth arrest.

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7 January 2021

Transactivation-deficient Delta TA-p73 inhibits p53 by direct competition for DNA binding: implications for tumorigenesis.

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7 January 2021

Physical interaction with Yes-associated protein enhances p73 transcriptional activity

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7 January 2021

Definition of a p53 transactivation function-deficient mutant and characterization of two independent p53 transactivation subdomains

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7 January 2021

DN-p73 is activated after DNA damage in a p53-dependent manner to regulate p53-induced cell cycle arrest.

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7 January 2021

Identification of a novel p53 functional domain that is necessary for efficient growth suppression

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7 January 2021

P63 and P73: P53 mimics, menaces and more

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7 January 2021

p73-deficient mice have neurological, pheromonal and inflammatory defects but lack spontaneous tumours

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7 January 2021

p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage

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7 January 2021

Differential regulation of cellular target genes by p53 devoid of the PXXP motifs with impaired apoptotic activity

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7 January 2021

Definition of the p53 functional domains necessary for inducing apoptosis

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7 January 2021

Identification of a novel p53 functional domain that is necessary for mediating apoptosis

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7 January 2021