Nitric oxide via an inducible isoform of nitric oxide synthase is a possible factor to eliminate inflammatory cells from the central nervous system of mice with experimental allergic encephalomyelitis (Q73128576)

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scientific article published on 01 March 1997

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English	Nitric oxide via an inducible isoform of nitric oxide synthase is a possible factor to eliminate inflammatory cells from the central nervous system of mice with experimental allergic encephalomyelitis	scientific article published on 01 March 1997

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1 November 2019

Nitric oxide via an inducible isoform of nitric oxide synthase is a possible factor to eliminate inflammatory cells from the central nervous system of mice with experimental allergic encephalomyelitis (English)

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9058766%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

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Y Okuda

1

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1 November 2019

S Sakoda

2

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1 November 2019

H Fujimura

3

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1 November 2019

T Yanagihara

4

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1 November 2019

publication date

1 March 1997

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1 November 2019

Journal of Neuroimmunology

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1 November 2019

73

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1 November 2019

1-2

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1 November 2019

107-116

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1 November 2019

Interleukin-1 beta-induced nitric oxide production activates apoptosis in pancreatic RINm5F cells

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Apoptosis and necrosis: two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/superoxide in cortical cell cultures

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Reactive nitrogen intermediates in human neuropathology: an overview

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Aminoguanidine, an inhibitor of inducible nitric oxide synthase, ameliorates experimental autoimmune encephalomyelitis in SJL mice.

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Cytokine production in the central nervous system of Lewis rats with experimental autoimmune encephalomyelitis: dynamics of mRNA expression for interleukin-10, interleukin-12, cytolysin, tumor necrosis factor α and tumor necrosis factor β

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Apoptotic cell death triggered by nitric oxide in pancreatic beta-cells

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Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with downregulation of inflammatory cytokines and differential upregulation of transforming growth factor beta, interleukin 4,

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In vivo expression of inducible nitric oxide synthase in experimentally induced neurologic diseases

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Nitric oxide localized to spinal cords of mice with experimental allergic encephalomyelitis: an electron paramagnetic resonance study

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Elevated secretion of reactive nitrogen and oxygen intermediates by inflammatory leukocytes in hyperacute experimental autoimmune encephalomyelitis: enhancement by the soluble products of encephalitogenic T cells

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Inflammatory leukocytes and cytokines in the peptide-induced disease of experimental allergic encephalomyelitis in SJL and B10.PL mice

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Nitric oxide as a potential pathological mechanism in demyelination: its differential effects on primary glial cells in vitro

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Nitric oxide induces necrotic but not apoptotic cell death in oligodendrocytes

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DNA damage and mutation in human cells exposed to nitric oxide in vitro

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7 January 2021

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Macrophage Apoptosis in the Central Nervous System in Experimental Autoimmune Encephalomyelitis

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7 January 2021

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Polyclonal antibody against an inducible form of nitric oxide synthase purified from the liver of rats treated with Propionibacterium acnes and lipopolysaccharide

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7 January 2021

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Expression of the inducible isoform of nitric oxide synthase in the central nervous system of mice correlates with the severity of actively induced experimental allergic encephalomyelitis

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Nitric oxide induces apoptosis in mouse splenic T lymphocytes

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7 January 2021

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Apoptosis in the nervous system in experimental allergic encephalomyelitis

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Nitric oxide toxicity in islet cells involves poly(ADP-ribose) polymerase activation and concomitant NAD+ depletion.

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Identifiers

10.1016/S0165-5728(96)00194-4

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9058766%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9058766%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

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