CD14 Facilitates Invasive Respiratory Tract Infection byStreptococcus pneumoniae (Q62516237)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17185649%20AND%20SRC:MED&resulttype=core&format=json

23 December 2019

title

CD14 facilitates invasive respiratory tract infection by Streptococcus pneumoniae (English)

1 reference

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respiratory tract infection

23 December 2019

main subject

Streptococcus pneumoniae

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based on heuristic

inferred from title

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23 December 2019

Mark C Dessing

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23 December 2019

Sandrine Florquin

series ordinal

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23 December 2019

Alex F de Vos

series ordinal

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23 December 2019

Tom van der Poll

series ordinal

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23 December 2019

language of work or name

English

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publication date

21 December 2006

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American Journal of Respiratory and Critical Care Medicine

23 December 2019

number of pages

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inferred from page(s)

published in

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23 December 2019

volume

175

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23 December 2019

issue

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23 December 2019

page(s)

604-611

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Analogues of platelet activating factor. 4. Some modifications of the phosphocholine moiety

23 December 2019

cites work

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Increased Circulating Soluble Cd14 Is Associated With High Mortality In Gram-Negative Septic Shock

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Streptococcus pneumoniae anchor to activated human cells by the receptor for platelet-activating factor

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Lipopolysaccharide rapidly traffics to and from the Golgi apparatus with the toll-like receptor 4-MD-2-CD14 complex in a process that is distinct from the initiation of signal transduction

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Critical Role of Lipopolysaccharide-Binding Protein and CD14 in Immune Responses against Gram-Negative Bacteria

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Treatment with an anti-CD14 monoclonal antibody delays and inhibits lipopolysaccharide-induced gene expression in humans in vivo

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CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

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Lipoteichoic acid preparations of gram-positive bacteria induce interleukin-12 through a CD14-dependent pathway.

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Transport of bacterial lipopolysaccharide to the golgi apparatus

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Double-stranded RNA-mediated TLR3 activation is enhanced by CD14.

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TNF-alpha compensates for the impaired host defense of IL-1 type I receptor-deficient mice during pneumococcal pneumonia

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Identifiers

DOI

10.1164/RCCM.200606-824OC

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17185649%20AND%20SRC:MED&resulttype=core&format=json

23 December 2019

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