Reduced TNF-α and increased IGF-I levels in the serum of Alzheimer's disease patients treated with the neurotrophic agent cerebrolysin. (Q53302466)

13 May 2018

title

Reduced TNF-α and increased IGF-I levels in the serum of Alzheimer's disease patients treated with the neurotrophic agent cerebrolysin. (English)

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13 May 2018

0 references

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Ramon Cacabelos

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13 May 2018

X Anton Alvarez

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13 May 2018

Carolina Sampedro

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13 May 2018

Carlos Linares

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13 May 2018

Manuel Aleixandre

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13 May 2018

Manuel García-Fantini

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13 May 2018

Herbert Moessler

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13 May 2018

publication date

17 June 2009

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13 May 2018

The International Journal of Neuropsychopharmacology

published in

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13 May 2018

volume

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13 May 2018

issue

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13 May 2018

page(s)

867-872

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13 May 2018

Use of serum insulin-like growth factor-I levels to predict psychiatric non-response to donepezil in patients with Alzheimer's disease.

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A 24-week, double-blind, placebo-controlled study of three dosages of Cerebrolysin in patients with mild to moderate Alzheimer's disease.

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Serum TNF-alpha levels are increased and correlate negatively with free IGF-I in Alzheimer disease.

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7 January 2021

Cerebrolysin decreases amyloid-beta production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer's disease.

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Insulin and insulin-like growth factor expression and function deteriorate with progression of Alzheimer's disease: link to brain reductions in acetylcholine

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Cerebrolysin ameliorates performance deficits, and neuronal damage in apolipoprotein E-deficient mice.

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The Clinician Interview-Based Impression (CIBI): a clinician's global change rating scale in Alzheimer's disease.

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Rescue of TNFalpha-inhibited neuronal cells by IGF-1 involves Akt and c-Jun N-terminal kinases

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Identifiers

DOI

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13 May 2018

1 reference

13 May 2018