Oncogenic potential diverge among human papillomavirus type 16 natural variants. (Q53268480)

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6 February 2020

title

Oncogenic potential diverge among human papillomavirus type 16 natural variants (English)

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6 February 2020

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22748253%20AND%20SRC:MED&resulttype=core&format=json

6 February 2020

João Simão Sobrinho

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6 February 2020

Luisa Lina Villa

series ordinal

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6 February 2020

language of work or name

English

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publication date

28 June 2012

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6 February 2020

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6 February 2020

volume

432

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6 February 2020

issue

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6 February 2020

page(s)

127-132

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Human papillomavirus 5 and 8 E6 downregulate interleukin-8 secretion in primary human keratinocytes.

6 February 2020

cites work

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Activities of human papillomavirus 16 E6 natural variants in human keratinocytes

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Asian-American variants of human papillomavirus 16 and risk for cervical cancer: a case-control study

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Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras.

7 January 2021

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Diversifying selection in human papillomavirus type 16 lineages based on complete genome analyses

7 January 2021

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Higher expression and activity of metalloproteinases in human cervical carcinoma cell lines is associated with HPV presence.

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Classification of papillomaviruses

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Mutations in CHEK2 associated with prostate cancer risk

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Human papillomavirus type 16 variants and risk of cervical cancer

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Progression of human papillomavirus type 18-immortalized human keratinocytes to a malignant phenotype

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p16INK4a Immunohistochemistry Improves Interobserver Agreement in the Diagnosis of Cervical Intraepithelial Neoplasia

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HPV16 E6 natural variants exhibit different activities in functional assays relevant to the carcinogenic potential of E6.

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Human papillomavirus genotype as a predictor of persistence and development of high-grade lesions in women with minor cervical abnormalities

7 January 2021

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RECKing MMP function: implications for cancer development

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The immortalizing and transforming ability of two common human papillomavirus 16 E6 variants with different prevalences in cervical cancer

7 January 2021

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Reduced local immunity in HPV-related VIN: expression of chemokines and involvement of immunocompetent cells

7 January 2021

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Tumour progression and metastatic behaviour in vivo correlates with integrin expression on melanocytic tumours.

7 January 2021

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The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53

7 January 2021

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High grade cervical lesions are caused preferentially by non-European variants of HPVs 16 and 18.

7 January 2021

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Epidemiological and functional implications of molecular variants of human papillomavirus.

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Pathology and epidemiology of HPV infection in females

7 January 2021

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The roles of E6-AP and MDM2 in p53 regulation in human papillomavirus-positive cervical cancer cells

7 January 2021

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Analysis of human papillomavirus type 16 E6 variants in relation to p53 codon 72 polymorphism genotypes in cervical carcinogenesis

7 January 2021

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Differences in transformation activity between HPV-18 and HPV-16 map to the viral LCR-E6-E7 region

7 January 2021

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Human papillomavirus is a necessary cause of invasive cervical cancer worldwide

7 January 2021

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A CHEK2 genetic variant contributing to a substantial fraction of familial breast cancer

7 January 2021

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Expression of integrins and Toll-like receptors in cervical cancer: effect of infectious agents

7 January 2021

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Risk for high-grade cervical intraepithelial neoplasia associated with variants of human papillomavirus types 16 and 18.

7 January 2021

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7 January 2021

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The genetic drift of human papillomavirus type 16 is a means of reconstructing prehistoric viral spread and the movement of ancient human populations

7 January 2021

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Natural variants of the human papillomavirus type 16 E6 protein differ in their abilities to alter keratinocyte differentiation and to induce p53 degradation.

7 January 2021

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inferred from DOI database lookup

7 January 2021

based on heuristic

Human papillomavirus type 16 variant lineages in United States populations characterized by nucleotide sequence analysis of the E6, L2, and L1 coding segments

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2012.06.011