An immunological epitope selective for pathological monomer-misfolded SOD1 in ALS. (Q52578937)

3 May 2018

title

An immunological epitope selective for pathological monomer-misfolded SOD1 in ALS. (English)

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3 May 2018

amyotrophic lateral sclerosis

main subject

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Neil R Cashman

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3 May 2018

Christine Vande Velde

object named as

Christine Vande Velde

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3 May 2018

author name string

Rishi Rakhit

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3 May 2018

Janice Robertson

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3 May 2018

Patrick Horne

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3 May 2018

Deborah M Ruth

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3 May 2018

Jennifer Griffin

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3 May 2018

Don W Cleveland

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3 May 2018

Avijit Chakrabartty

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3 May 2018

language of work or name

English

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publication date

7 May 2007

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3 May 2018

published in

Nature Medicine

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3 May 2018

volume

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3 May 2018

issue

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3 May 2018

page(s)

754-759

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3 May 2018

Unraveling the mechanisms involved in motor neuron degeneration in ALS

cites work

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7 January 2021

Genetic epidemiology of amyotrophic lateral sclerosis

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7 January 2021

High threshold for induction of the stress response in motor neurons is associated with failure to activate HSF1.

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7 January 2021

Proteasomal inhibition by misfolded mutant superoxide dismutase 1 induces selective motor neuron death in familial amyotrophic lateral sclerosis

1 reference

7 January 2021

Toxicity of familial ALS-linked SOD1 mutants from selective recruitment to spinal mitochondria.

1 reference

7 January 2021

Amyotrophic lateral sclerosis-associated SOD1 mutant proteins bind and aggregate with Bcl-2 in spinal cord mitochondria

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7 January 2021

Induction of nitric oxide-dependent apoptosis in motor neurons by zinc-deficient superoxide dismutase

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7 January 2021

Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity

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7 January 2021

Protein aggregation in motor neurone disorders.

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7 January 2021

High molecular weight complexes of mutant superoxide dismutase 1: age-dependent and tissue-specific accumulation

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7 January 2021

Oxidation-induced misfolding and aggregation of superoxide dismutase and its implications for amyotrophic lateral sclerosis

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7 January 2021

Monomeric Cu,Zn-superoxide dismutase is a common misfolding intermediate in the oxidation models of sporadic and familial amyotrophic lateral sclerosis.

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7 January 2021

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

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7 January 2021

A prion protein epitope selective for the pathologically misfolded conformation

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7 January 2021

Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase

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7 January 2021

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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7 January 2021

An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

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7 January 2021

ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.

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7 January 2021

Aberrantly increased hydrophobicity shared by mutants of Cu,Zn-superoxide dismutase in familial amyotrophic lateral sclerosis.

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7 January 2021

Minute quantities of misfolded mutant superoxide dismutase-1 cause amyotrophic lateral sclerosis

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7 January 2021

Oxygen-induced maturation of SOD1: a key role for disulfide formation by the copper chaperone CCS.

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7 January 2021

Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mut

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7 January 2021

Conversion to the amyotrophic lateral sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria

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7 January 2021

ALS-associated mutant SOD1G93A causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes

1 reference

7 January 2021

Overloading of stable and exclusion of unstable human superoxide dismutase-1 variants in mitochondria of murine amyotrophic lateral sclerosis models

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7 January 2021

Spinal cord endoplasmic reticulum stress associated with a microsomal accumulation of mutant superoxide dismutase-1 in an ALS model

1 reference

7 January 2021

Induction of the unfolded protein response in familial amyotrophic lateral sclerosis and association of protein-disulfide isomerase with superoxide dismutase 1.

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7 January 2021

Onset and progression in inherited ALS determined by motor neurons and microglia

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7 January 2021

Substrate specificity of the DnaK chaperone determined by screening cellulose-bound peptide libraries

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7 January 2021

Identifiers

DOI

10.1038/NM1559

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3 May 2018

Dimensions Publication ID

1040991729

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Fatcat ID

release_5iyho2mbyjdvrl2fgthhmvkgam

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Fatcat

https://api.fatcat.wiki/v0/release/5iyho2mbyjdvrl2fgthhmvkgam

24 November 2022

mapped directly with Wikidata item

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3 May 2018