A polymorphism in the complement component C1r is not associated with sporadic Alzheimer's disease. (Q52025813)

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scientific article published in January 2003

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English	A polymorphism in the complement component C1r is not associated with sporadic Alzheimer's disease.	scientific article published in January 2003

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Europe PubMed Central

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12499050

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20 April 2018

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http://europepmc.org/abstract/MED/12499050

title

A polymorphism in the complement component C1r is not associated with sporadic Alzheimer's disease. (English)

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12499050

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Hanna Rosenmann

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1

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12499050

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Zeev Meiner

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2

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12499050

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Esther Kahana

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Zoja Aladjem

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4

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Gideon Friedman

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Arie Ben-Yehuda

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6

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Tal Grenader

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7

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Eli Wertman

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8

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12499050

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20 April 2018

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Oded Abramsky

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9

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12499050

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http://europepmc.org/abstract/MED/12499050

language of work or name

English

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publication date

1 January 2003

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12499050

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20 April 2018

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http://europepmc.org/abstract/MED/12499050

published in

Neuroscience Letters

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12499050

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20 April 2018

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http://europepmc.org/abstract/MED/12499050

volume

336

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Europe PubMed Central

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12499050

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20 April 2018

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http://europepmc.org/abstract/MED/12499050

issue

2

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12499050

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20 April 2018

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http://europepmc.org/abstract/MED/12499050

page(s)

101-104

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cites work

Candidate genes showing no evidence for association or linkage with Alzheimer's disease using family-based methodologies.

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7 January 2021

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Genetic polymorphisms in the cathespin D and interleukin-6 genes and the risk of Alzheimer's disease.

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7 January 2021

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7 January 2021

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The number of trait loci in late-onset Alzheimer disease

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7 January 2021

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Association of early-onset Alzheimer's disease with an interleukin-1? gene polymorphism

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7 January 2021

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2-amino-4H-3,1-benzoxazin-4-ones as inhibitors of C1r serine protease

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

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The importance of inflammatory mechanisms in Alzheimer disease

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7 January 2021

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Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

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Crossref

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

based on heuristic

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A common amino acid polymorphism in complement component C1R.

1 reference

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Crossref

reference URL

https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Genetic susceptibility to Alzheimer disease

1 reference

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Crossref

reference URL

https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Lack of replication of association findings in complex disease: an analysis of 15 polymorphisms in prior candidate genes for sporadic Alzheimer's disease

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Crossref

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

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Genetic association study between alpha 1-antichymotrypsin polymorphism and Alzheimer disease in Chinese Han population.

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7 January 2021

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New frontiers in Alzheimer's disease genetics

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

based on heuristic

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Neurons express proteins of the classical complement pathway in Alzheimer disease

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

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Up-regulated production and activation of the complement system in Alzheimer's disease brain

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https://api.crossref.org/works/10.1016%2FS0304-3940%2802%2901218-1

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7 January 2021

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Identifiers

DOI

10.1016/S0304-3940(02)01218-1

1 reference

stated in

Europe PubMed Central

PubMed publication ID

12499050

retrieved

20 April 2018

reference URL

http://europepmc.org/abstract/MED/12499050

PubMed publication ID

12499050

1 reference

stated in

Europe PubMed Central

PubMed publication ID

12499050

retrieved

20 April 2018

reference URL

http://europepmc.org/abstract/MED/12499050

 

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