Expression of matrix metalloproteinases MMP-2 and MMP-9 is altered during nephrogenesis in fetuses from diabetic rats (Q51986135)

19 April 2018

title

Expression of matrix metalloproteinases MMP-2 and MMP-9 is altered during nephrogenesis in fetuses from diabetic rats (English)

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19 April 2018

Patrick Bruneval

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19 April 2018

author name string

Jean-Paul Duong Van Huyen

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19 April 2018

Mélanie Viltard

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19 April 2018

Touria Nehiri

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19 April 2018

Nicole Freund

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19 April 2018

Marie-France Bélair

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19 April 2018

Cécile Martinerie

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19 April 2018

Brigitte Lelongt

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19 April 2018

Martine Lelièvre-Pégorier

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19 April 2018

publication date

14 May 2007

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19 April 2018

published in

Laboratory Investigation

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19 April 2018

volume

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19 April 2018

issue

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19 April 2018

page(s)

680-689

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19 April 2018

Gelatinase B (MMP-9) is not essential in the normal kidney and does not influence progression of renal disease in a mouse model of Alport syndrome

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Matrix metalloproteinases MMP2 and MMP9 are produced in early stages of kidney morphogenesis but only MMP9 is required for renal organogenesis in vitro

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High glucose concentration causes a decrease in mesangium degradation. A factor in the pathogenesis of diabetic nephropathy

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Connective tissue growth factor mediates high glucose effects on matrix degradation through tissue inhibitor of matrix metalloproteinase type 1: implications for diabetic nephropathy

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High glucose decreases matrix metalloproteinase-2 activity in rat mesangial cells via transforming growth factor-beta1

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Connective tissue growth factor mediates the profibrotic effects of transforming growth factor-beta produced by tubular epithelial cells in response to high glucose

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Temporal expression profile and distribution pattern indicate a role of connective tissue growth factor (CTGF/CCN-2) in diabetic nephropathy in mice

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Spatiotemporal distribution of insulin-like growth factor receptors during nephrogenesis in fetuses from normal and diabetic rats

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7 January 2021

Matrix metalloproteinase 2 (MMP2) and MMP9 are produced by kidney collecting duct principal cells but are differentially regulated by SV40 large-T, arginine vasopressin, and epidermal growth factor

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Altered nephrogenesis due to maternal diabetes is associated with increased expression of IGF-II/mannose-6-phosphate receptor in the fetal kidney

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Identifiers

DOI

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19 April 2018

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19 April 2018