Role of cGMP versus 20-HETE in the vasodilator response to nitric oxide in rat cerebral arteries (Q57219771)

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J R Falck

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H Okamoto

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D R Harder

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R J Roman

American Journal of Physiology Heart and Circulatory Physiology

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language of work or name

English

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publication date

July 2000

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published in

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volume

279

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issue

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page(s)

H339-50

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cites work

Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase

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Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle

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Recent insights into the regulation of cerebral circulation

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Coexistence of two types of Ca(2+)-activated K+ channels in rat renal arterioles.

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Differential relaxant responses of guinea-pig lung strips and bronchial rings to sodium nitroprusside: a mechanism independent of cGMP formation.

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20-HETE is an endogenous inhibitor of the large-conductance Ca(2+)-activated K+ channel in renal arterioles.

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