Apoptosis in human myelodysplastic syndrome CD34+ cells is modulated by the upregulation of TLRs and histone H4 acetylation via a β-arrestin 1 dependent mechanism. (Q55059498)

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English	Apoptosis in human myelodysplastic syndrome CD34+ cells is modulated by the upregulation of TLRs and histone H4 acetylation via a β-arrestin 1 dependent mechanism.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Apoptosis in human myelodysplastic syndrome CD34+ cells is modulated by the upregulation of TLRs and histone H4 acetylation via a β-arrestin 1 dependent mechanism. (English)

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

toll-like receptor

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myelodysplastic syndrome

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based on heuristic

inferred from title

author name string

Qing Zeng

1

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Jing Shu

2

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Qi Hu

3

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Shao-Hong Zhou

4

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Yi-Ming Qian

5

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Ming-Hui Hu

6

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Ling-Yan Hu

7

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Yu-Guang Wang

8

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Yong-Ming Zhou

9

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Jia-Hui Lu

10

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16 June 2018

http://europepmc.org/abstract/MED/26708616

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17 December 2015

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Experimental Cell Research

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16 June 2018

http://europepmc.org/abstract/MED/26708616

340

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

1

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PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

22-31

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

Myelodysplastic syndromes: From pathogenesis and prognosis to treatment

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Recent developments in myelodysplastic syndromes.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Apoptosis and antiapoptotic mechanisms in the progression of myelodysplastic syndrome

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Excessive proliferation matched by excessive apoptosis in myelodysplastic syndromes: the cause-effect relationship

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Programmed cell death is an intrinsic feature of MDS progenitors, predominantly found in the cluster-forming cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

'Low-risk' myelodysplastic syndrome is associated with excessive apoptosis and an increased ratio of pro- versus anti-apoptotic bcl-2-related proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

The role of mitochondrial-mediated apoptosis in a myelodysplastic syndrome secondary to congenital deletion of the short arm of chromosome 4

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

NF-kappaB and FLIP in arsenic trioxide (ATO)-induced apoptosis in myelodysplastic syndromes (MDSs)

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Apoptosis in patients with myelodysplastic syndromes: differential involvement of marrow cells in 'good' versus 'poor' prognosis patients and correlation with apoptosis-related genes

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Excessive apoptosis in low risk myelodysplastic syndromes (MDS).

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Toll-like receptor-4 is up-regulated in hematopoietic progenitor cells and contributes to increased apoptosis in myelodysplastic syndromes

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Toll-like receptor alterations in myelodysplastic syndrome

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Clinical effect of point mutations in myelodysplastic syndromes

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Global H3K4me3 genome mapping reveals alterations of innate immunity signaling and overexpression of JMJD3 in human myelodysplastic syndrome CD34+ cells.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Histone acetylation in gene regulation

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

The myelodysplastic syndrome as a prototypical epigenetic disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Myelodysplastic syndromes

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

A nuclear function of beta-arrestin1 in GPCR signaling: regulation of histone acetylation and gene transcription

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

β-arrestin-1 is a nuclear transcriptional regulator of endothelin-1-induced β-catenin signaling.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Endocytosis of G protein-coupled receptors: roles of G protein-coupled receptor kinases and beta-arrestin proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Multifaceted roles of beta-arrestins in the regulation of seven-membrane-spanning receptor trafficking and signalling

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Identification of beta-arrestin2 as a G protein-coupled receptor-stimulated regulator of NF-kappaB pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

beta-Arrestin inhibits NF-kappaB activity by means of its interaction with the NF-kappaB inhibitor IkappaBalpha.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Beta-arrestins 1 and 2 differentially regulate LPS-induced signaling and pro-inflammatory gene expression.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Morphine promotes apoptosis via TLR2, and this is negatively regulated by beta-arrestin 2.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Toll-like receptors on hematopoietic progenitor cells stimulate innate immune system replenishment

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2015.12.008

7 January 2021

Identifiers

10.1016/J.YEXCR.2015.12.008

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/26708616

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